C. Tong

发表

New Methods in Cardiovascular Biology Neonatal Mouse – Derived Engineered Cardiac Tissue

R. Moss, J. Ralphe, A. Grimes, 2012 .

Roles of phosphorylation of myosin binding protein‐C and troponin I in mouse cardiac muscle twitch dynamics

D. Zawieja, M. Muthuchamy, C. Tong, 2004, The Journal of physiology.

Temporary Left Ventricular Assist Device Through an Axillary Access is a Promising Approach to Improve Outcomes in Refractory Cardiogenic Shock Patients

B. Sareyyupoglu, E. Góngora, K. Doersch, 2015, ASAIO journal.

Superiority of mitral valve replacement with preservation of subvalvular structure to conventional replacement in severe rheumatic mitral valve disease: a modified technique and results of one-year follow up.

Zhirong Wu, C. Tong, F. Zhong, 2000, The Journal of heart valve disease.

Cardiac Myosin Binding Protein-C Phosphorylation Mitigates Age-Related Cardiac Dysfunction

R. Solaro, C. Warren, J. Trzeciakowski, 2019, JACC. Basic to translational science.

Estrogen Protects Cardiac Function and Energy Metabolism in Dilated Cardiomyopathy Induced by Loss of Cardiac IRS1 and IRS2

Shaodong Guo, Hui Yan, C. Allred, 2022, Circulation. Heart failure.

Mechanosensing and Regulation of Cardiac Function

S. Glaser, D. M. Foster, Honey B. Golden, 2014, Journal of clinical & experimental cardiology.

ZBP 1 sequesters cGAS in the cytoplasm and sustains type I interferon responses to 1 mitochondrial DNA 2 3

M. Bosenberg, A. West, G. Shadel, 2022 .

Myosin-binding protein C stabilizes, but is not the sole determinant of SRX myosin in cardiac muscle

S. Sadayappan, D. Warshaw, Shane R. Nelson, 2022, bioRxiv.

ZBP1 sequesters cGAS in the cytoplasm and sustains type I interferon responses to mitochondrial DNA

M. Bosenberg, A. West, G. Shadel, 2022, bioRxiv.

Elevated type I interferon responses potentiate metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice

A. West, S. Bell, R. Watson, 2021, Science Advances.

Phosphoregulation of Cardiac Inotropy via Myosin Binding Protein-C During Increased Pacing Frequency or 1-Adrenergic Stimulation Tong et al: Phosphoregulation of Inotropy via Cardiac MyBP-C

P. Powers, R. Moss, S. Sadayappan, 2017 .

Phosphoregulation of Cardiac Inotropy via Myosin Binding Protein-C During Increased Pacing Frequency or &bgr;1-Adrenergic Stimulation

P. Powers, R. Moss, Xin Wu, 2015, Circulation. Heart failure.

Automated analysis of contractile force and Ca2+ transients in engineered heart tissue.

Thomas Eschenhagen, Arne Hansen, Alexandra Eder, 2014, American journal of physiology. Heart and circulatory physiology.

Charged residue alterations in the inner‐core domain and carboxy‐terminus of α‐tropomyosin differentially affect mouse cardiac muscle contractility

T. Hewett, D. Zawieja, J. Robbins, 2004, The Journal of physiology.

Type I interferon potentiates metabolic dysfunction, inflammation, and accelerated aging in mtDNA mutator mice

A. West, C. Martinez, S. Bell, 2020, bioRxiv.

Myosin binding protein-C phosphorylation is the principal mediator of protein kinase A effects on thick filament structure in myocardium.

R. Moss, T. Irving, Brett A. Colson, 2012, Journal of molecular and cellular cardiology.

Preparation and Identification of Cardiac Myofibrils from Whole Heart Samples.

C. Tong, Heidi A Creed, 2021, Methods in molecular biology.

Status of Early-Career Academic Cardiology: A Global Perspective.

D. Mann, M. B. Turner, E. Spatz, 2017, Journal of the American College of Cardiology.

Src/FAK complex phosphorylates cardiac myosin binding protein c (cMyBP-C) in vitro and in vivo

Shenyuan L. Zhang, Yixin Jin, M. Muthuchamy, 2020, bioRxiv.

Challenges facing early career academic cardiologists.

S. Houser, T. Bunch, T. Dardas, 2014, Journal of the American College of Cardiology.

Acceleration of Crossbridge Kinetics by Protein Kinase A Phosphorylation of Cardiac Myosin Binding Protein C Modulates Cardiac Function

P. Powers, R. Moss, M. Greaser, 2008, Circulation research.

Cardiac myosin-binding protein-C is a critical mediator of diastolic function

N. Nair, K. Doersch, C. Tong, 2014, Pflügers Archiv - European Journal of Physiology.

National Institutes of Health Career Development Awards for Cardiovascular Physician-Scientists: Recent Trends and Strategies for Success.

N. Desai, D. Mann, C. Balke, 2015, Journal of the American College of Cardiology.

Neonatal Mouse–Derived Engineered Cardiac Tissue: A Novel Model System for Studying Genetic Heart Disease

R. Moss, W. D. de Lange, J. Ralphe, 2011, Circulation research.

Isolation of Adult Mouse Using Perfusion Apparatus

D. Dostal, C. Tong, Yang Liu, 2021, Methods in Molecular Biology.

Deletion of Cdc42 in embryonic cardiomyocytes results in right ventricle hypoplasia

Jian Wang, Yang Liu, Shenyuan L. Zhang, 2017, Clinical and Translational Medicine.

Cardiac myosin-binding protein-C is a critical mediator of diastolic function

N. Nair, K. Doersch, C. Tong, 2014, Pflügers Archiv - European Journal of Physiology.

Cooperative sensing of mitochondrial DNA by ZBP1 and cGAS promotes cardiotoxicity

Y. Li, M. Bosenberg, A. West, 2023, Cell.

Usefulness of Released Cardiac Myosin Binding Protein-C as a Predictor of Cardiovascular Events

S. Sadayappan, J. Trzeciakowski, C. Tong, 2017, The American journal of cardiology.