J. F. Rosen

发表

Rickets with alopecia: an inborn error of vitamin D metabolism.

H. DeLuca, L. Finberg, A. Fleischman, 1979, The Journal of pediatrics.

Assessment of lead stores in children: validation of an 8-hour CaNa2EDTA provocative test.

J. F. Rosen, M. Markowitz, 1984, The Journal of pediatrics.

Need for the lead mobilization test in children with lead poisoning.

J. F. Rosen, M. Markowitz, 1991, The Journal of pediatrics.

A cellular defect in hereditary vitamin-D-dependent rickets type II: defective nuclear uptake of 1,25-dihydroxyvitamin D in cultured skin fibroblasts.

U. Liberman, S. Marx, J. F. Rosen, 1981, The New England journal of medicine.

The displacement of calcium from osteocalcin at submicromolar concentrations of free lead.

R. Gupta, C. Gundberg, J. F. Rosen, 1994, Biochimica et biophysica acta.

The cellular metabolism of lead and calcium: a kinetic analysis in cultured osteoclastic bone cells.

J. Pounds, J. F. Rosen, 1988, Contributions to nephrology.

Cellular metabolism of lead: A kinetic analysis in cultured osteoclastic bone cells☆

J. Pounds, J. F. Rosen, 1986 .

Studies of lead transport in bone organ culture

J. F. Rosen, Emma E. Wexler, 1977 .

Metabolic abnormalities in lead toxic children: public health implications.

J. F. Rosen, 1989, Bulletin of the New York Academy of Medicine.

Effects of CaNa2EDTA on lead and trace metal metabolism in bone organ culture.

H. Kraner, K. Jones, J. F. Rosen, 1982, Toxicology and applied pharmacology.

L-line x-ray fluorescence of cortical bone lead compared with the CaNa2EDTA test in lead-toxic children: public health implications.

P. Bijur, J. Kalef-Ezra, D. Slatkin, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Immobilization hypercalcemia after single limb fractures in children and adolescents.

L. Finberg, J. F. Rosen, D. Wolin, 1978 .

Hereditary resistance to 1,25-dihydroxyvitamin D: defective function of receptors for 1,25-dihydroxyvitamin D in cells cultured from bone.

U. Liberman, P. Holst, S. Marx, 1983, The Journal of clinical endocrinology and metabolism.

Calcitonin in metabolic disorders.

J. F. Rosen, A. Haymovits, 1972, Advances in metabolic disorders.

Association between age, blood lead concentration, and serum 1,25-dihydroxycholecalciferol levels in children.

J. T. Peeler, H. DeLuca, R. Chesney, 1982, The American journal of clinical nutrition.

1,25-Dihydroxycholecalciferol: its use in the long-term management of idiopathic hypoparathyroidism in children.

H. DeLuca, J. Eisman, L. Finberg, 1977, The Journal of clinical endocrinology and metabolism.

Circadian variations in serum zinc (Zn) concentrations: correlation with blood ionized calcium, serum total calcium and phosphate in humans.

M. Mizruchi, J. F. Rosen, M. Markowitz, 1985, The American journal of clinical nutrition.

Osteocalcin in human serum: a circadian rhythm.

M. Mizruchi, C. Gundberg, J. F. Rosen, 1985, The Journal of clinical endocrinology and metabolism.

1,25-dihydroxyvitamin D3-treated hypoparathyroidism: 35 patients years in 10 children.

H. DeLuca, J. F. Rosen, M. Markowitz, 1982, The Journal of clinical endocrinology and metabolism.

25-Hydroxycholecalciferol for early neonatal hypocalcemia. Occurrence in premature newborns.

A. Fleischman, J. F. Rosen, G. Nathenson, 1978, American journal of diseases of children.

25-Hydroxyvitamin D. Plasma levels in mothers and their premature infants with neonatal hypocalcemia.

L. Finberg, J. F. Rosen, M. Roginsky, 1974, American journal of diseases of children.

Serum 1,25-dihydroxyvitamin D levels in normal children and in vitamin D disorders.

H. DeLuca, R. Chesney, J. F. Rosen, 1980, American journal of diseases of children.

Maternal and fetal serum 1,25-dihydroxyvitamin D levels at term.

H. DeLuca, A. Fleischman, J. F. Rosen, 1980, The Journal of pediatrics.

Adverse health effects of lead at low exposure levels: trends in the management of childhood lead poisoning.

J. F. Rosen, 1995, Toxicology.

Health effects of lead at low exposure levels. Expert consensus and rationale for lowering the definition of childhood lead poisoning.

J. F. Rosen, 1992, American journal of diseases of children.

Effects of growth hormone therapy on circadian osteocalcin rhythms in idiopathic short stature.

P. Saenger, J. Dimartino-Nardi, J. F. Rosen, 1989, The Journal of clinical endocrinology and metabolism.

25-hydroxyvitamin D. Serum levels and oral administration of calcifediol in neonates.

A. Fleischman, J. F. Rosen, G. Nathenson, 1978, Archives of internal medicine.

The metabolism of lead in isolated bone cell populations: interactions between lead and calcium.

J. F. Rosen, 1983, Toxicology and applied pharmacology.

The effect of Pb(2+) on the structure and hydroxyapatite binding properties of osteocalcin.

L. Mints, C. Gundberg, J. F. Rosen, 2001, Biochimica et biophysica acta.

Cellular Ca2+ homeostasis and Ca2+-mediated cell processes as critical targets for toxicant action: conceptual and methodological pitfalls.

J. Pounds, J. F. Rosen, John F. Rosen, 1988, Toxicology and applied pharmacology.

25-Hydroxycholecalciferol inthe management of rickets associated with extrahepatic biliary atresia.

L. Finberg, Michael I. Cohen, J. F. Rosen, 1976, The Journal of pediatrics.

Ionized calcium: serum levels during symptomatic hypocalcemia.

J. F. Rosen, M. Sorell, John F. Rosen, 1975, The Journal of pediatrics.

Development of 19F NMR for measurement of [Ca2+]i and [Pb2+]i in cultured osteoblastic bone cells.

R. Gupta, J. F. Rosen, F. Schanne, 1990, Environmental health perspectives.

Basal Ganglia Calcification (BGC) in Down's Syndrome (DS)—Another Manifestation of Premature Aging *

H. Wiśniewski, K. Wisniewski, M. Tenner, 1982, Annals of the New York Academy of Sciences.

Lead inhibits 1,25-dihydroxyvitamin D-3 regulation of calcium metabolism in osteoblastic osteosarcoma cells (ROS 17/2.8).

R. Gupta, J. F. Rosen, F. Schanne, 1992, Biochimica et biophysica acta.

Liver lysosomes in congenital osteopetrosis. A study of lysosomal function, calcitonin, parathyroid hormone, and 3',5'-adenosine monophosphate.

J. F. Rosen, A. Haymovits, 1972, The Journal of pediatrics.

Interactions of lead, calcium, vitamin D, and nutrition in lead-burdened children.

J. F. Rosen, M. Sorrell, M. Roginsky, 1977, Archives of environmental health.

THE SKELETAL METABOLISM OF LEAD: STUDIES OF LEAD TRANSPORT IN BONE ORGAN CULTURE

J. F. Rosen, E. Trinidad, 1974, Pediatric Research.

Sequential measurements of bone lead content by L X-ray fluorescence in CaNa2EDTA-treated lead-toxic children.

P. Bijur, J. Kalef-Ezra, D. Slatkin, 1991, Environmental health perspectives.

Circulating calcitriol concentrations in health and disease

R. Chesney, J. F. Rosen, J. F. Rosen, 1983 .

Relationships among blood lead levels, iron deficiency, and cognitive development in two-year-old children.

P. Bijur, H. Ruff, J. F. Rosen, 1996, Environmental health perspectives.

Cellular and molecular toxicity of lead in bone.

J. Pounds, J. F. Rosen, G. J. Long, 1991, Environmental health perspectives.

Lead impairs the production of osteocalcin by rat osteosarcoma (ROS 17/2.8) cells.

J. Pounds, J. F. Rosen, G. J. Long, 1990, Toxicology and applied pharmacology.

Effect of lead on parathyroid hormone-induced responses in rat osteoblastic osteosarcoma cells (ROS 17/2.8) using 19F-NMR.

R. Gupta, J. F. Rosen, F. Schanne, 1990, Biochimica et biophysica acta.

Cellular lead toxicity and metabolism in primary and clonal osteoblastic bone cells.

J. Pounds, J. F. Rosen, G. J. Long, 1990, Toxicology and applied pharmacology.

Effect of dietary calcium and lead status on intestinal calcium absorption.

C. Fullmer, J. F. Rosen, 1990, Environmental research.

Lead increases free Ca2+ concentration in cultured osteoblastic bone cells: simultaneous detection of intracellular free Pb2+ by 19F NMR.

R. Gupta, J. F. Rosen, F. Schanne, 1989, Proceedings of the National Academy of Sciences of the United States of America.

Reduction in 1,25-dihydroxyvitamin D in children with increased lead absorption.

K R Mahaffey, H. DeLuca, R. Chesney, 1980, The New England journal of medicine.

Calcitonin: its nature and role in man.

J. F. Rosen, A. Haymovits, 1970, Pediatrics.

Depressed Excretion of 6²-Hydroxycortisol in Lead-Toxic Children*

P. Saenger, J. F. Rosen, M. Markowitz, 1984 .

Management of childhood lead poisoning.

S. Piomelli, J. Graef, J. F. Rosen, 1984, The Journal of pediatrics.

31P NMR and saturation transfer studies of the effect of Pb2+ on cultured osteoblastic bone cells.

R. Gupta, J. F. Rosen, T. Dowd, 1990, The Journal of biological chemistry.

Lead perturbs epidermal growth factor (EGF) modulation of intracellular calcium metabolism and collagen synthesis in clonal rat osteoblastic (ROS 17/2.8) cells.

J. F. Rosen, G. J. Long, 1992, Toxicology and applied pharmacology.

Parathyroid adenoma, parathyroid crisis, and acute pancreatitis in an adolescent.

S. Boley, J. F. Rosen, F. Daum, 1973, The Journal of pediatrics.

Childhood lead poisoning in China.

J F Rosen, X Shen, S Wu, 1996, The Science of the total environment.

Significance of Plasma Lead Levels in Normal and Lead-Intoxicated Children*

J. F. Rosen, E. Trinidad, 1974, Environmental health perspectives.

Serial measurements of aminolevulinic acid dehydratase in children with lead toxicity.

J. F. Rosen, M. Markowitz, V. Morris, 1988, The Journal of pediatrics.

Vitamin D-Dependent Rickets: Actions of Parathyroid Hormone and 25-Hydroxycholecalciferol

L. Finberg, John F Rosen, Laurence Finberg, 1972, Pediatric Research.

Temporal interrelationships between the circadian rhythms of serum parathyroid hormone and calcium concentrations.

M. Thorpy, S. Arnaud, J. F. Rosen, 1988, The Journal of clinical endocrinology and metabolism.

Pseudohypoparathyroidism type III (PHP III), a new form of PHP: Normal renal responsiveness to exogenous parathyroid hormone (PTH)

J. F. Rosen, M. Markowitz, J. Schessel, 1980 .

Lead contamination in milks fed to infants: 1972-1973.

S. Lamm, J. F. Rosen, 1974, Pediatrics.

Effects of iron deficiency on lead excretion in children with moderate lead intoxication.

P. Bijur, J. F. Rosen, M. Markowitz, 1990, The Journal of pediatrics.

The three-dimensional structure of bovine calcium ion-bound osteocalcin using 1H NMR spectroscopy.

C. Gundberg, J. F. Rosen, T. Dowd, 2003, Biochemistry.

Low-level lead-induced neurotoxicity in children: an update on central nervous system effects

John F Rosen, Yoram Finkelstein, Morri E Markowitz, 1998, Brain Research Reviews.

Feasibility of noninvasive analysis of lead in the human tibia by soft x-ray fluorescence.

K J Ellis, L Wielopolski, D N Slatkin, 1983, Medical physics.

The Toxicological Importance of Lead in Bone: The Evolution and Potential Uses of Bone Lead Measurements by X-Ray Fluorescence to Evaluate Treatment Outcomes in Moderately Lead Toxic Children

J. F. Rosen, 1988 .

Diagnostic significance of edetate disodium calcium testing in children with increased lead absorption.

P. Saenger, J. F. Rosen, M. Markowitz, 1982, American journal of diseases of children.

Neurodevelopmental E¤ects of Childhood Exposure to Heavy Metals: Lessons from Pediatric Lead Poisoning

J. Schneider, T. Lidsky, J. F. Rosen, 2006 .

Children with moderately elevated blood lead levels: a role for other diagnostic tests?

J. F. Rosen, M. Markowitz, I. Clemente, 1997, Environmental health perspectives.

Quantitative interactions between Pb2+ and Ca2+ homeostasis in cultured osteoclastic bone cells.

J. Pounds, J. F. Rosen, 1989, Toxicology and applied pharmacology.

Plasma lead levels in normal and lead-intoxicated children.

J. F. Rosen, E. Trinidad, C. Zarate-Salvador, 1974, The Journal of pediatrics.

Moderate lead poisoning: trends in blood lead levels in unchelated children.

P. Bijur, H. Ruff, J. F. Rosen, 1996, Environmental health perspectives.

Primary prevention of childhood lead poisoning--the only solution.

J. F. Rosen, P. Mushak, 2001, The New England journal of medicine.

Declining blood lead levels and cognitive changes in moderately lead-poisoned children.

P. Bijur, H. Ruff, J. F. Rosen, 1993, JAMA.

A randomized trial of calcium supplementation for childhood lead poisoning.

M. Sinnett, J. F. Rosen, M. Markowitz, 2004, Pediatrics.

The circadian rhythm of serum osteocalcin concentrations: Effects of 1,25 dihydroxyvitamin D administration

C. Gundberg, J. F. Rosen, M. Markowitz, 1987, Calcified Tissue International.

Lead perturbs 1,25 dihydroxyvitamin D3 modulation of intracellular calcium metabolism in clonal rat osteoblastic (ROS 17/2.8) cells.

J. F. Rosen, G. J. Long, 1994, Life sciences.

Lead induced rise in intracellular free calcium is mediated through activation of protein kinase C in osteoblastic bone cells.

J. F. Rosen, F. Schanne, G. J. Long, 1997, Biochimica et biophysica acta.

Seasonal fluctuations in serum concentrations of vitamin D metabolites in normal subjects.

R. Chesney, J. Birkenhager, J. F. Rosen, 1981, British medical journal.

Absence of seasonal variation in serum concentrations of 1,25-dihydroxyvitamin D despite a rise in 25-hydroxyvitamin D in summer.

H. DeLuca, R. Chesney, K. Mahaffey, 1981, The Journal of clinical endocrinology and metabolism.

The effects of succimer on the absorption of lead in adults determined by using the stable isotope 204Pb.

A. Flegal, D. Smith, J. F. Rosen, 1994, Environmental Research.

Blood collection and analytical considerations in blood lead screening in children.

D. Schonfeld, J. Deutsch, K. Verebey, 1995, Clinical chemistry.

In vivo measurement of cortical bone lead using polarized x rays.

L Wielopolski, D N Slatkin, J. Kalef-Ezra, 1989, Medical physics.

Lead activation of protein kinase C from rat brain. Determination of free calcium, lead, and zinc by 19F NMR.

J F Rosen, G J Long, F A Schanne, 1994, The Journal of biological chemistry.

25-Hydroxycholecalciferol inthe management of rickets associated with extrahepatic biliary atresia.

M. Cohen, L. Finberg, J. F. Rosen, 1976, The Journal of pediatrics.

Lead intoxication alters basal and parathyroid hormone-regulated cellular calcium homeostasis in rat osteosarcoma (ROS 17/2.8) cells

J. Pounds, J. F. Rosen, G. J. Long, 1992, Calcified Tissue International.

The microdetermination of blood lead in children by flameless atomic absorption: the carbon rod atomizer.

J. F. Rosen, 1972, The Journal of laboratory and clinical medicine.

"Severe chronic lead insult that maintains body burdens of lead related to those in the skeleton": observations by Dr. Clair Patterson conclusively demonstrated.

J. Pounds, J. F. Rosen, 1998, Environmental research.

Sequential measurements of bone lead content by L X-ray fluorescence in CaNa2EDTA-treated lead-toxic children.

P. Bijur, J. Kalef-Ezra, D. Slatkin, 1991, Environmental health perspectives.

Circadian rhythms of blood minerals in humans.

L. Rotkin, J. F. Rosen, M. Markowitz, 1981, Science.

A critical evaluation of public health programs at the Bunker Hill Superfund site.

J. F. Rosen, 2003, The Science of the total environment.

Effects of 1,25 dihydroxyvitamin D3 administration on circadian mineral rhythms in humans

M. Mizruchi, J. F. Rosen, M. Markowitz, 1985, Calcified Tissue International.

Circadian Rhythms of Blood Minerals during Adolescence

S. Laxminarayan, M. Mizruchi, J. F. Rosen, 1984, Pediatric Research.

Ionized calcium: hypocalcemia Serum levels during symptomatic

J. F. Rosen, M. Sorell, 1975 .

Penicillamine : Its Actions on Lead Transport in Bone Organ Culture

J. F. Rosen, M. Markowitz, 2007 .

338 A NEW TYPE OF RICKETS: UNRESPONSIVENESS OF BONE AND INTESTINE TO HIGH LEVELS OF ENDOGENOUSLY SYNTHESIZED 1,25-DIHYDROXYVITAMIN D3 [1,25(0H)2D3]

H. DeLuca, L. Finberg, A. Fleischman, 1978, Pediatric Research.

Effects of low levels of lead exposure.

J. F. Rosen, 1992, Science.

Rachel ' s Environment & Health

J. Schwartz, J. F. Rosen, 2002 .

25-hydroxyvitamin D. Serum levels and oral administration of calcifediol in neonates.

A. Fleischman, J. F. Rosen, G. Nathenson, 1978, Archives of internal medicine.

Clinical Applications of L-Line X-Ray Fluorescence To Estimate Bone Lead Values in Lead-Poisoned Young Children and in Children, Teenagers, and Adults From Lead-Exposed and Non-Lead-Exposed Suburban Communities in the United States

J. F. Rosen, 1997, Toxicology and industrial health.

THE TOXICOLOGICAL IMPORTANCE OF LEAD IN BONE: THE EVOLUTION AND POTENTIAL USES OF BONE LEAD MEASUREMENTS BY X-RAY FLUORESCENCE TO EVALUATE TREATMENT OUTCOMES IN MODERATELY LEAD TOXIC CHILDREN

J. F. Rosen, 2004 .