S. Yamachika

发表

Vibrio parahaemolyticus Chromosomal qnr Homologue VPA0095: Demonstration by Transformation with a Mutated Gene of Its Potential To Reduce Quinolone Susceptibility in Escherichia coli

M. Kaku, Y. Onodera, Kenichi Sato, 2005, Antimicrobial Agents and Chemotherapy.

Transfer of human serum IgG to nonobese diabetic Igmu null mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren's syndrome.

R. Jonsson, C. Stewart, J. Brayer, 1998, Proceedings of the National Academy of Sciences of the United States of America.

Anti-Pseudomonas aeruginosa compound, 1,2,3,4-tetrahydro-1,3,5-triazine derivative, exerts its action by primarily targeting MreB.

M. Yamashita, Yasunori Muramatsu, S. Yamachika, 2012, Biological & pharmaceutical bulletin.

NRF2 Activates Growth Factor Genes and Downstream AKT Signaling to Induce Mouse and Human Hepatomegaly.

M. Karin, G. Hatzivassiliou, Beicheng Sun, 2020, Journal of hepatology.

ER Stress Drives Lipogenesis and Steatohepatitis via Caspase-2 Activation of S1P

M. Karin, R. Kaufman, A. Saltiel, 2018, Cell.

A novel NOD-derived murine model of primary Sjögren's syndrome.

R. Jonsson, R. Holmdahl, J. Brayer, 1998, Arthritis and rheumatism.

An Alternative Perspective to the Immune Response in Autoimmune Exocrinopathy: Induction of Functional Quiescence Rather Than Destructive Autoaggression

R. Jonsson, J. Brayer, M. Humphreys-Beher, 1999, Scandinavian journal of immunology.

Aberrant proteolytic digestion of biglycan and decorin by saliva and exocrine gland lysates from the NOD mouse model for autoimmune exocrinopathy.

G. E. Oxford, J. Brayer, M. Humphreys-Beher, 2000, Clinical and experimental rheumatology.

Correlation between penicillin-binding protein 2 mutations and carbapenem resistance in Escherichia coli.

M. Yamashita, S. Yamachika, Y. Kamai, 2013, Journal of medical microbiology.

Alleles from chromosomes 1 and 3 of NOD mice combine to influence Sjögren's syndrome-like autoimmune exocrinopathy.

J. Brayer, M. Humphreys-Beher, S. Cha, 2000, The Journal of rheumatology.

Excessive synthesis of matrix metalloproteinases in exocrine tissues of NOD mouse models for Sjögren's syndrome.

G. E. Oxford, J. Brayer, M. Humphreys-Beher, 1998, The Journal of rheumatology.

Elevated levels of cysteine protease activity in saliva and salivary glands of the nonobese diabetic (NOD) mouse model for Sjögren syndrome.

M. Humphreys-Beher, A. Peck, S. Yamachika, 1997, Proceedings of the National Academy of Sciences of the United States of America.

Salivary gland changes in the NOD mouse model for Sjögren's syndrome: is there a non-immune genetic trigger?

Y. Nakagawa, N. Maeda, M. Humphreys-Beher, 1998, European journal of morphology.

p62, Upregulated during Preneoplasia, Induces Hepatocellular Carcinogenesis by Maintaining Survival of Stressed HCC-Initiating Cells.

S. Subramaniam, M. Karin, M. Valasek, 2016, Cancer cell.

p62/SQSTM1—Dr. Jekyll and Mr. Hyde that prevents oxidative stress but promotes liver cancer

M. Karin, K. Taniguchi, S. Yamachika, 2016, FEBS letters.

Plasmid integration method: a new tool for analysis of the essentiality and function of genes in S. aureus.

K. Hiramatsu, Y. Onodera, S. Yamachika, 2012, Journal of microbiological methods.

In vitro and in vivo antibacterial activity of sitafloxacin

K. Hoshino, M. Chiba, S. Yamachika, 2008 .

Cloning and functional characterization of chicken p160 coactivator family members.

J. Nishikawa, S. Arai, S. Yamachika, 2001, Biochimica et biophysica acta.