C. Neusch

发表

Cyclin-dependent kinase 5 (CDK5) and neuronal cell death

M. Bähr, Jochen H Weishaupt, C. Neusch, 2003, Cell and Tissue Research.

Influence of Methylene Blue on Microglia-Induced Inflammation and Motor Neuron Degeneration in the SOD1G93A Model for ALS

E. Schomburg, F. Kirchhoff, Jochen H Weishaupt, 2012, PloS one.

NO mediates microglial response to acute spinal cord injury under ATP control in vivo

E. Schomburg, F. Kirchhoff, H. Steffens, 2010, Glia.

Long‐lasting post‐mortem activity of spinal microglia in situ in mice

E. Schomburg, F. Kirchhoff, H. Steffens, 2010, Journal of neuroscience research.

In vivo imaging reveals rapid morphological reactions of astrocytes towards focal lesions in an ALS mouse model

E. Schomburg, F. Kirchhoff, H. Steffens, 2011, Neuroscience Letters.

Selegiline induces dopamine release through ATP-sensitive potassium channels in the rat caudate-putamen in vitro

A. Moser, C. Neusch, Senta Schnierle, 1997, Neurochemistry International.

Kir4.1 Channel Expression Is Essential for Parietal Cell Control of Acid Secretion*

M. Manns, P. Song, U. Seidler, 2011, The Journal of Biological Chemistry.

Progressive loss of a glial potassium channel (KCNJ10) in the spinal cord of the SOD1 (G93A) transgenic mouse model of amyotrophic lateral sclerosis

F. Kirchhoff, W. Schulz-Schaeffer, B. Holtmann, 2006, Journal of neurochemistry.

Mitofusin 2 gene mutation (R94Q) causing severe early‐onset axonal polyneuropathy (CMT2A)

T. Eggermann, M. Bähr, J. Senderek, 2007, European journal of neurology.

Hereditary motor and sensory neuropathy caused by a novel mutation in LITAF

J. Epplen, W. M. Gerding, C. Neusch, 2009, Neuromuscular Disorders.

Calcium influx mediated by the inwardly rectifying K+ channel Kir4.1 (KCNJ10) at low external K+ concentration.

J. Deitmer, S. Hülsmann, C. Neusch, 2007, Cell calcium.

Monoamine Oxidase: Interaction with Isoquinoline Derivatives

A. Moser, C. Neusch, 1998 .

Late-onset myopathy of the posterior calf muscles mimicking Miyoshi myopathy unrelated to dysferlin mutation: a case report

T. Kuhlmann, W. Kress, C. Schneider-Gold, 2012, Journal of Medical Case Reports.

Kir4.1 Potassium Channel Subunit Is Crucial for Oligodendrocyte Development and In Vivo Myelination

H. Lester, R. Jacobs, P. Kofuji, 2001, The Journal of Neuroscience.

Kir channels in the CNS: emerging new roles and implications for neurological diseases

M. Bähr, Jochen H Weishaupt, J. H. Weishaupt, 2003, Cell and Tissue Research.

Severe demyelinating hypertrophic polyneuropathy caused by a de novo frameshift mutation within the intracellular domain of myelin protein zero (MPZ/P 0 )

S. Pilgram, W. M. Gerding, P. Dibaj, 2009, Journal of the Neurological Sciences.

Glia cells in amyotrophic lateral sclerosis: New clues to understanding an old disease?

Christiane Schneider-Gold, Mathias Bähr, M. Bähr, 2007, Muscle & nerve.

Multiple neuroprotective mechanisms of minocycline in autoimmune CNS inflammation

M. Bähr, D. Merkler, R. Diem, 2007, Neurobiology of Disease.

Analysis of Astroglial K+ Channel Expression in the Developing Hippocampus Reveals a Predominant Role of the Kir4.1 Subunit

Devin K. Binder, Christian Steinhäuser, Christian Hartmann, 2009, The Journal of Neuroscience.

Time course of inner ear degeneration and deafness in mice lacking the Kir4.1 potassium channel subunit

H. Lester, C. Chiu, P. Kofuji, 2003, Hearing Research.

Heterologous expression of a glial Kir channel (KCNJ10) in a neuroblastoma spinal cord (NSC-34) cell line.

S. Hülsmann, P. Dibaj, S. Schütze, 2013, Physiological research.

Possible roles of the weakly inward rectifying k+ channel Kir4.1 (KCNJ10) in the pre-Bötzinger complex.

S. Hülsmann, N. Papadopoulos, C. Neusch, 2008, Advances in experimental medicine and biology.

Lack of the Kir4.1 channel subunit abolishes K+ buffering properties of astrocytes in the ventral respiratory group: impact on extracellular K+ regulation.

Frank Kirchhoff, Mathias Bähr, Swen Hülsmann, 2006, Journal of neurophysiology.

In Vivo Imaging Reveals Distinct Inflammatory Activity of CNS Microglia versus PNS Macrophages in a Mouse Model for ALS

E. Schomburg, F. Kirchhoff, H. Steffens, 2011, PloS one.

Flupirtine as neuroprotective add-on therapy in autoimmune optic neuritis.

M. Bähr, M. Otto, R. Diem, 2008, The American journal of pathology.

Transient decrease in F‐actin may be necessary for translocation of proteins into dendritic spines

Maryann E Martone, Mark H Ellisman, Francisco Capani, 2005, The European journal of neuroscience.

G Proteins Modulate D2 Receptor-Coupled K(ATP) Channels in Rat Dopaminergic Terminals

A. Moser, C. Neusch, Doris Runde, 2000, Neurochemical Research.

The natural history of primary progressive multiple sclerosis: insights from the German NeuroTransData registry

R. Fleischer, C. Hofer, H. Peters, 2023, BMC Neurology.

Expression of GIRK (Kir3.1/Kir3.4) channels in mouse fibroblast cells with and without β1 integrins

H. Lester, D. Mosher, C. Labarca, 2000, FEBS letters.

SonoGraphic monitoring of severe focal B‐cell myositis of the anterior calf muscle responsive to rituximab

R. Gold, M. Hasselblatt, C. Krogias, 2015, Muscle & nerve.

Kir4.1 channels regulate swelling of astroglial processes in experimental spinal cord edema

F. Kirchhoff, J. Hirrlinger, P. Dibaj, 2007, Journal of neurochemistry.

The dopamine D2 receptor agonist alpha-dihydroergocryptine modulates voltage-gated sodium channels in the rat caudate-putamen

C. Neusch, M. Althaus, V. Böhme, 2000, Journal of Neural Transmission.

Kir4.1 channels regulate swelling of astroglial processes in experimental spinal cord edema

F. Kirchhoff, J. Hirrlinger, P. Dibaj, 2007, Journal of neurochemistry.

Ventilator induzierter Zwerchfellschaden

C. Neusch, S. Rosseau, Abteilung, 2007, Pneumologie.