M. Davidson

发表

PPARβ/δ activation blocks lipid-induced inflammatory pathways in mouse heart and human cardiac cells.

W. Wahli, L. Michalik, X. Palomer, 2011, Biochimica et biophysica acta.

Mitochondrial damage mediates genotoxicity of arsenic in mammalian cells.

Xiuwei Tang, M. Athar, T. Hei, 2005, Cancer research.

Defects of mtDNA Replication Impaired Mitochondrial Biogenesis During Trypanosoma cruzi Infection in Human Cardiomyocytes and Chagasic Patients: The Role of Nrf1/2 and Antioxidant Response

N. Garg, Shivali Gupta, M. Davidson, 2012, Journal of the American Heart Association.

Hypoxia induces B-type natriuretic peptide release in cell lines derived from human cardiomyocytes.

W. Jiménez, M. Morales-Ruiz, J. Ros, 2009, American journal of physiology. Heart and circulatory physiology.

Evidence of ROS generation by mitochondria in cells with impaired electron transport chain and mitochondrial DNA damage.

H. Majima, H. Yen, Y. Koga, 2007, Mitochondrion.

A calcium signaling defect in the pathogenesis of a mitochondrial DNA inherited oxidative phosphorylation deficiency

E. Schon, M. King, P. Pinton, 1999, Nature Medicine.

Trypanosoma cruzi Induces the Reactive Oxygen Species-PARP-1-RelA Pathway for Up-regulation of Cytokine Expression in Cardiomyocytes*

N. Garg, Shivali Gupta, M. Davidson, 2010, The Journal of Biological Chemistry.

17β-Estradiol-induced interaction of estrogen receptor α and human atrial essential myosin light chain modulates cardiac contractile function

V. Regitz-Zagrosek, E. Dworatzek, S. Mahmoodzadeh, 2016, Basic Research in Cardiology.

The Interplay between NF-kappaB and E2F1 Coordinately Regulates Inflammation and Metabolism in Human Cardiac Cells

T. Chan, A. Feldman, X. Palomer, 2011, PloS one.

The p65 subunit of NF-kappaB binds to PGC-1alpha, linking inflammation and metabolic disturbances in cardiac cells.

T. Chan, A. Feldman, X. Palomer, 2010, Cardiovascular research.

The p65 subunit of NF-kB binds to PGC-1a, linking inflammation and metabolic disturbances in cardiac cells

T. Chan, A. Feldman, X. Palomer, 2010 .

TNF-alpha reduces PGC-1alpha expression through NF-kappaB and p38 MAPK leading to increased glucose oxidation in a human cardiac cell model.

T. Chan, A. Feldman, X. Palomer, 2009, Cardiovascular research.

A functionally dominant mitochondrial DNA mutation.

S. Dimauro, E. Schon, E. Trevisson, 2008, Human molecular genetics.

Evidence for nuclear modifier gene in mitochondrial cardiomyopathy.

C. Nesti, M. Davidson, W. F. Walker, 2009, Journal of molecular and cellular cardiology.

Neutral lipid storage disease with subclinical myopathy due to a retrotransposal insertion in the PNPLA2 gene

S. Dimauro, R. Haller, H. Akman, 2010, Neuromuscular Disorders.

Fatal infantile form of muscle phosphofructokinase deficiency

S. Dimauro, S. Servidei, M. Kornfeld, 1986, Neurology.

TNF- a reduces PGC-1 a expression through NF- k B and p38 MAPK leading to increased glucose oxidation in a human cardiac cell model

T. Chan, A. Feldman, X. Palomer, 2009 .

Pathogenesis of the deafness-associated A1555G mitochondrial DNA mutation.

S. Dimauro, E. Schon, F. Santorelli, 2002, Biochemical and biophysical research communications.

Fatal infantile cardioencephalomyopathy with COX deficiency and mutations in SCO2, a COX assembly gene

P. Kaplan, S. Dimauro, E. Schon, 1999, Nature Genetics.

Mutation Screening in Patients With Isolated Cytochrome c Oxidase Deficiency

S. Dimauro, C. Sue, D. Vivo, 2003, Pediatric Research.

A Pathogenic 15-Base Pair Deletion in Mitochondrial DNA-encoded Cytochrome c Oxidase Subunit III Results in the Absence of Functional Cytochrome c Oxidase*

K. Hoffbuhr, M. King, E. Davidson, 2000, The Journal of Biological Chemistry.

SIRT3-mediated inhibition of FOS through histone H3 deacetylation prevents cardiac fibrosis and inflammation

F. Villarroya, I. Miguel-Escalada, F. Crispi, 2020, Signal Transduction and Targeted Therapy.

SIRT3-mediated inhibition of FOS through histone H3 deacetylation prevents cardiac fibrosis and inflammation

F. Villarroya, I. Miguel-Escalada, F. Crispi, 2020, Signal Transduction and Targeted Therapy.

miR-146a targets Fos expression in human cardiac cells

T. Chan, A. Feldman, E. Barroso, 2015, Disease Models & Mechanisms.

Targeted cytoplasmic irradiation and autophagy.

T. Hei, M. Davidson, Jinhua Wu, 2017, Mutation research.

Copper supplementation restores cytochrome c oxidase activity in cultured cells from patients with SCO2 mutations.

S. Dimauro, E. Schon, L. Salviati, 2002, The Biochemical journal.

Arsenic induced mitochondrial DNA damage and altered mitochondrial oxidative function: implications for genotoxic mechanisms in mammalian cells.

T. Hei, S. X. Huang, M. Davidson, 2007, Cancer research.

Ketogenic treatment reduces deleted mitochondrial DNAs in cultured human cells

E. Schon, R. Gilkerson, M. Davidson, 2004, Annals of neurology.

Tetramethylpyrazine (TMP) protects against sodium arsenite-induced nephrotoxicity by suppressing ROS production, mitochondrial dysfunction, pro-inflammatory signaling pathways and programed cell death

T. Hei, V. Ivanov, M. Davidson, 2014, Archives of Toxicology.

Sex differences in exercise-induced physiological myocardial hypertrophy are modulated by oestrogen receptor beta.

V. Regitz-Zagrosek, J. Gustafsson, A. Kusch, 2014, Cardiovascular research.

Sex-specific pathways in early cardiac response to pressure overload in mice

V. Regitz-Zagrosek, C. Schubert, C. Grohé, 2008, Journal of Molecular Medicine.

PPARβ/δ attenuates palmitate-induced endoplasmic reticulum stress and induces autophagic markers in human cardiac cells.

W. Wahli, L. Michalik, E. Barroso, 2014, International journal of cardiology.

Novel cell lines derived from adult human ventricular cardiomyocytes.

M. Hirano, C. Nesti, M. Davidson, 2005, Journal of molecular and cellular cardiology.

Mitochondrial DNA background modifies the bioenergetics of NARP/MILS ATP6 mutant cells.

G. Manfredi, C. Vives-Bauza, M. Davidson, 2010, Human molecular genetics.

Biochemical analysis of respiratory function in cybrid cell lines harbouring mitochondrial DNA mutations.

S. Dimauro, E. Schon, A. Baracca, 2004, The Biochemical journal.

Mitochondria and the heart

S. Dimauro, M. Hirano, M. Davidson, 2001, Current opinion in cardiology.

Cellular and molecular studies in muscle and cultures from patients with multiple mitochondrial DNA deletions

R. Carrozzo, M. Hirano, A. Miranda, 1999, Journal of the Neurological Sciences.

Cytoplasmic irradiation results in mitochondrial dysfunction and DRP1-dependent mitochondrial fission.

T. Hei, Chunxin Wang, M. Davidson, 2013, Cancer research.

Differences in nuclear gene expression between cells containing monomer and dimer mitochondrial genomes.

L. Papadopoulou, T. Brown, M. Davidson, 2002, Gene.

Mitochondria regulate DNA damage and genomic instability induced by high LET radiation.

T. Hei, M. Davidson, Bo Zhang, 2014, Life sciences in space research.

The complete nucleotide sequence of Chinese hamster (Cricetulus griseus) mitochondrial DNA

T. Hei, M. Davidson, M. Partridge, 2007, DNA sequence : the journal of DNA sequencing and mapping.

Altered Gene Expression by Low-Dose Arsenic Exposure in Humans and Cultured Cardiomyocytes: Assessment by Real-Time PCR Arrays

D. DeMarini, J. Mo, T. Wade, 2011, International journal of environmental research and public health.

Mitochondrial function and nuclear factor-kappaB-mediated signaling in radiation-induced bystander effects.

T. Hei, V. Ivanov, M. Davidson, 2008, Cancer research.

Cardiomyocyte Specific Deficiency of Serine Palmitoyltransferase Subunit 2 Reduces Ceramide but Leads to Cardiac Dysfunction*

Yong-Moon Lee, I. Goldberg, I. Park, 2012, The Journal of Biological Chemistry.

Mitochondrial nucleoids maintain genetic autonomy but allow for functional complementation

E. Schon, R. Gilkerson, M. Davidson, 2008, The Journal of cell biology.

Analysis of cybrids harboring MELAS mutations in the mitochondrial tRNALeu(UUR) gene

E. Schon, M. King, Y. Koga, 1995, Muscle & nerve. Supplement.

Studies of COX16, COX19, and PET191 in human cytochrome-c oxidase deficiency.

S. Dimauro, E. Schon, M. Mancuso, 2004, Archives of neurology.

Resveratrol induces nuclear factor-κB activity in human cardiac cells.

F. Villarroya, M. Pallàs, A. Camins, 2013, International journal of cardiology.

A homoplasmic mtDNA variant can influence the phenotype of the pathogenic m.7472Cins MTTS1 mutation: are two mutations better than one?

Robert W. Taylor, M. Elstner, D. Turnbull, 2008, European Journal of Human Genetics.

Signaling in Radiation-Induced Bystander Effects Mediated − B κ Mitochondrial Function and Nuclear Factor-Updated

T. Hei, V. Ivanov, M. Davidson, 2008 .

The m.3243A>G mtDNA mutation is pathogenic in an in vitro model of the human blood brain barrier.

M. Davidson, W. F. Walker, Evelyn Hernandez-Rosa, 2009, Mitochondrion.

Cytochrome c oxidase deficiency due to a novel SCO2 mutation mimics Werdnig-Hoffmann disease.

S. Dimauro, E. Schon, P. Canoll, 2002, Archives of neurology.

A homoplasmic mitochondrial transfer ribonucleic acid mutation as a cause of maternally inherited hypertrophic cardiomyopathy.

Robert W. Taylor, S. Dimauro, D. Turnbull, 2003, Journal of the American College of Cardiology.

Molecular and Cellular Pathobiology Cytoplasmic Irradiation Results inMitochondrial Dysfunction and DRP 1-Dependent Mitochondrial Fission

T. Hei, Chunxin Wang, M. Davidson, 2013 .

T. Hei, V. Ivanov, M. Davidson, 2015, Archives of Toxicology.

Mitochondria-Derived Reactive Intermediate Species Mediate Asbestos-Induced Genotoxicity and Oxidative Stress–Responsive Signaling Pathways

S. Amundson, T. Hei, S. Ghandhi, 2012, Environmental health perspectives.

17β-Estradiol-induced interaction of ERα with NPPA regulates gene expression in cardiomyocytes.

V. Regitz-Zagrosek, E. Dworatzek, S. Mahmoodzadeh, 2012, Cardiovascular research.

Environmental Mutagens Induced Transversions but not Transitions in Regulatory Region of Mitochondrial DNA

M. Kibriya, H. Ahsan, T. Hei, 2009, Journal of toxicology and environmental health. Part A.

Defects in mitochondrial protein synthesis and respiratory chain activity segregate with the tRNA(Leu(UUR)) mutation associated with mitochondrial myopathy, encephalopathy, lactic acidosis, and strokelike episodes.

E. Schon, M. King, Y. Koga, 1992, Molecular and cellular biology.

Nuclear Factor-κB Regulates Estrogen Receptor-α Transcription in the Human Heart*

V. Regitz-Zagrosek, E. Dworatzek, E. Becher, 2009, The Journal of Biological Chemistry.

Hypoxia induces B-type natriuretic peptide release in cell lines derived from human cardiomyocytes.

W. Jiménez, M. Morales-Ruiz, J. Ros, 2009, American journal of physiology. Heart and circulatory physiology.

In vitro analysis of mutations causing myoclonus epilepsy with ragged-red fibers in the mitochondrial tRNA(Lys)gene: two genotypes produce similar phenotypes

E. Schon, M. King, Y. Koga, 1995, Molecular and cellular biology.

Cytoplasmic irradiation results in mitochondrial dysfunction and DRP1-dependent mitochondrial fission.

T. Hei, Chunxin Wang, M. Davidson, 2013, Cancer research.

A functionally dominant mitochondrial DNA mutation.

S. Dimauro, E. Schon, E. Trevisson, 2008, Human molecular genetics.

Heterogeneity of the molecular lesions in inherited phosphofructokinase deficiency.

S. Dimauro, E. Frenkel, N. Noble, 1983, The Journal of clinical investigation.

Recurrent myoglobinuria due to a nonsense mutation in the COX I gene of mitochondrial DNA

S. Dimauro, R. Haller, T. Taivassalo, 2000, Neurology.

17β-Estradiol-induced interaction of ERα with NPPA regulates gene expression in cardiomyocytes.

V. Regitz-Zagrosek, E. Dworatzek, S. Mahmoodzadeh, 2012, Cardiovascular research.