A. Pisano

发表

Neuromuscular magnetic stimulation counteracts muscle decline in ALS patients: results of a randomized, double-blind, controlled study

A. Musarò, M. Inghilleri, E. Palma, 2019, Scientific Reports.

Notch Signal Mediates the Cross-Interaction between M2 Muscarinic Acetylcholine Receptor and Neuregulin/ErbB Pathway: Effects on Schwann Cell Proliferation

C. Talora, A. Tata, S. Loreti, 2022, Biomolecules.

Nonischemic left ventricular scar and cardiac sudden death in the young.

V. Palmieri, G. d’Amati, C. D. di Gioia, 2016, Human pathology.

Mitochondrial Energetics and Ca2+-Activated ATPase in Obstructive Hypertrophic Cardiomyopathy

O. Alfieri, O. Rimoldi, F. Girolami, 2020, Journal of clinical medicine.

Can IDO activity predict primary resistance to anti-PD-1 treatment in NSCLC?

P. Marchetti, M. Simmaco, I. Zizzari, 2018, Journal of Translational Medicine.

Myosins and MyomiR Network in Patients with Obstructive Hypertrophic Cardiomyopathy

O. Alfieri, O. Rimoldi, F. Girolami, 2022, Biomedicines.

Isoleucyl-tRNA synthetase levels modulate the penetrance of a homoplasmic m.4277T>C mitochondrial tRNA(Ile) mutation causing hypertrophic cardiomyopathy.

Robert W. Taylor, A. F. Campese, L. Frontali, 2012, Human molecular genetics.

M2 muscarinic receptor activation regulates schwann cell differentiation and myelin organization

B. Avallone, C. Talora, V. Magnaghi, 2014, Developmental neurobiology.

RNA‐seq profiling reveals different pathways between remodeled vessels and myocardium in hypertrophic cardiomyopathy

O. Rimoldi, I. Olivotto, P. Camici, 2022, Microcirculation.

Efficient mitochondrial biogenesis drives incomplete penetrance in Leber’s hereditary optic neuropathy

M. Zeviani, P. Barboni, P. Chinnery, 2013, Brain : a journal of neurology.

Anti-aminoacyl-tRNA synthetase-related myositis and dermatomyositis: clues for differential diagnosis on muscle biopsy

R. Priori, G. Valesini, G. d’Amati, 2018, Virchows Archiv.

Novel compound mutations in the mitochondrial translation elongation factor (TSFM) gene cause severe cardiomyopathy with myocardial fibro-adipose replacement

Robert W. Taylor, P. Bonnen, V. Morea, 2019, Scientific Reports.

Short peptides from leucyl-tRNA synthetase rescue disease-causing mitochondrial tRNA point mutations

Robert W. Taylor, A. Fiorillo, V. Morea, 2015, Human molecular genetics.

The isolated carboxy-terminal domain of human mitochondrial leucyl-tRNA synthetase rescues the pathological phenotype of mitochondrial tRNA mutations in human cells

Robert W. Taylor, L. Frontali, M. Zeviani, 2014, EMBO molecular medicine.

Potential role for the VDR agonist elocalcitol in metabolic control: Evidences in human skeletal muscle cells

A. Lenzi, C. Crescioli, G. Vannelli, 2017, The Journal of Steroid Biochemistry and Molecular Biology.

Cardiomyopathies due to homoplasmic mitochondrial tRNA mutations: morphologic and molecular features.

Robert W. Taylor, C. Autore, V. Petrozza, 2013, Human pathology.

Impaired mitochondrial biogenesis is a common feature to myocardial hypertrophy and end-stage ischemic heart failure

Robert W. Taylor, C. Nediani, V. Petrozza, 2016, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.

CD73 expression and pathologic response to neoadjuvant chemotherapy in triple negative breast cancer

P. Ascierto, P. Marchetti, A. De Luca, 2019, Virchows Archiv.

Cerebral Mitochondrial Microangiopathy Leads to Leukoencephalopathy in Mitochondrial Neurogastrointestinal Encephalopathy

G. Plazzi, D. Manners, R. Lodi, 2018, American Journal of Neuroradiology.

Myocardial fibrosis: morphologic patterns and role of imaging in diagnosis and prognostication.

M. Francone, G. d’Amati, C. Giordano, 2021, Cardiovascular pathology : the official journal of the Society for Cardiovascular Pathology.

Breast cancer subtypes affect the nodal response after neoadjuvant chemotherapy in locally advanced breast cancer: Are we ready to endorse axillary conservation?

F. Frusone, G. d’Amati, A. Botticelli, 2019, The breast journal.

BRAIN Efﬁcient mitochondrial biogenesis drives incomplete penetrance in Leber’s hereditary optic neuropathy

M. Zeviani, P. Barboni, P. Chinnery, 2014 .

New Insights and Evidence on “Food Intolerances”: Non-Celiac Gluten Sensitivity and Nickel Allergic Contact Mucositis

A. Picarelli, C. Giordano, A. Pisano, 2023, Nutrients.

Can IDO activity predict primary resistance to anti-PD-1 treatment in NSCLC?

P. Marchetti, M. Simmaco, I. Zizzari, 2018, Journal of Translational Medicine.

Targeting estrogen receptor β as preventive therapeutic strategy for Leber's hereditary optic neuropathy.

M. Montopoli, A. Ghelli, V. Carelli, 2015, Human molecular genetics.

Neuromuscular magnetic stimulation counteracts muscle decline in ALS patients: results of a randomized, double-blind, controlled study

A. Musarò, E. Palma, M. Ceccanti, 2019, Scientific Reports.

Homozygous NOTCH3 null mutation and impaired NOTCH3 signaling in recessive early-onset arteriopathy and cavitating leukoencephalopathy

R. Lodi, T. Pippucci, C. Tonon, 2015, EMBO molecular medicine.

Exogenous peptides are able to penetrate human cell and mitochondrial membranes, stabilize mitochondrial tRNA structures, and rescue severe mitochondrial defects

A. Ghelli, V. Morea, V. Turris, 2020, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Novel compound mutations in the mitochondrial translation elongation factor (TSFM) gene cause severe cardiomyopathy with myocardial fibro-adipose replacement

P. Bonnen, V. Morea, F. Re, 2019, Scientific Reports.

PD-L1 testing in metastatic triple negative breast cancer: Results of an Italian survey

C. Della Rocca, P. Marchetti, A. Botticelli, 2023, Tumori.

Overexpression in metastatic breast cancer supports Syndecan-1 as a marker of invasiveness and poor prognosis

M. Allegretti, G. d’Amati, A. Botticelli, 2022, Clinical and Experimental Medicine.