M. Tortarolo

发表

RNS60 exerts therapeutic effects in the SOD1 ALS mouse model through protective glia and peripheral nerve rescue

A. Kalmes, C. Bendotti, V. Bonetto, 2018, Journal of Neuroinflammation.

Specific Induction of Akt3 in Spinal Cord Motor Neurons is Neuroprotective in a Mouse Model of Familial Amyotrophic Lateral Sclerosis

R. Piva, C. Bendotti, M. Peviani, 2013, Molecular Neurobiology.

Kif1Bβ isoform is enriched in motor neurons but does not change in a mouse model of amyotrophic lateral sclerosis

C. Bendotti, M. Coleman, L. Conforti, 2003 .

Erythropoietin does not preserve motor neurons in a mouse model of familial ALS

C. Bendotti, G. Grignaschi, M. Tortarolo, 2007, Amyotrophic lateral sclerosis : official publication of the World Federation of Neurology Research Group on Motor Neuron Diseases.

Glutamate release in human cerebral cortex and its modulation by 5‐hydroxtryptamine acting at h 5‐HT1D receptors

M. Raiteri, M. Marcoli, G. Maura, 1998, British journal of pharmacology.

A Novel HGF/SF Receptor (MET) Agonist Transiently Delays the Disease Progression in an Amyotrophic Lateral Sclerosis Mouse Model by Promoting Neuronal Survival and Dampening the Immune Dysregulation

E. Gherardi, C. Bendotti, V. Bonetto, 2020, International journal of molecular sciences.

Multiple intracerebroventricular injections of human umbilical cord mesenchymal stem cells delay motor neurons loss but not disease progression of SOD1G93A mice.

C. Bendotti, Azzurra Agostini, D. Moscatelli, 2017, Stem cell research.

Targeting Extracellular Cyclophilin A Reduces Neuroinflammation and Extends Survival in a Mouse Model of Amyotrophic Lateral Sclerosis

S. Pozzi, S. Fumagalli, R. Chiesa, 2017, The Journal of Neuroscience.

Lack of TNF‐alpha receptor type 2 protects motor neurons in a cellular model of amyotrophic lateral sclerosis and in mutant SOD1 mice but does not affect disease progression

F. Fiordaliso, C. Bendotti, V. Bonetto, 2015, Journal of neurochemistry.

Altered Metabolic Profiles Associate with Toxicity in SOD1G93A Astrocyte-Neuron Co-Cultures

H. Keun, C. Bendotti, L. Cantoni, 2017, Scientific Reports.

Unraveling the complexity of amyotrophic lateral sclerosis: recent advances from the transgenic mutant SOD1 mice.

C. Bendotti, I. Caron, M. Peviani, 2010, CNS & neurological disorders drug targets.

Lack of apoptosis in mice with ALS

D. Schiffer, R. Piva, A. Migheli, 1999, Nature Medicine.

E FFECT OF ELEVATED CALCIUM LEVEL AND CALCIUM BINDING PROTEINS IN ACUTE AND CHRONIC DEGENERATION OF MOTOR NEURONS Summary of the Ph

I., C. Bendotti, J. Engelhardt, 2017 .

Expression of SOD1 G93A or wild‐type SOD1 in primary cultures of astrocytes down‐regulates the glutamate transporter GLT‐1: lack of involvement of oxidative stress

Andrew J. Crossthwaite, M. Rattray, Robert J. Williams, 2003, Journal of neurochemistry.

Transgenic SOD1 G93A mice develop reduced GLT‐1 in spinal cord without alterations in cerebrospinal fluid glutamate levels

M. Rattray, C. Bendotti, L. Carvelli, 2001, Journal of neurochemistry.

Proteomic analysis of spinal cord of presymptomatic amyotrophic lateral sclerosis G93A SOD1 mouse.

M. Salmona, C. Bendotti, E. Gianazza, 2007, Biochemical and biophysical research communications.

Activation of the p38MAPK cascade is associated with upregulation of TNF alpha receptors in the spinal motor neurons of mouse models of familial ALS

J. Julien, C. Bendotti, D. Coco, 2006, Molecular and Cellular Neuroscience.

The p97‐Nploc4 ATPase complex plays a role in muscle atrophy during cancer and amyotrophic lateral sclerosis

A. R. Re Cecconi, R. Piccirillo, C. Bendotti, 2022, Journal of cachexia, sarcopenia and muscle.

Presymptomatically applied AMPA receptor antagonist prevents calcium increase in vulnerable type of motor axon terminals of mice modeling amyotrophic lateral sclerosis.

C. Bendotti, L. Siklós, J. Engelhardt, 2017, Biochimica et biophysica acta. Molecular basis of disease.

Inter- and Intracellular Signaling in Amyotrophic Lateral Sclerosis: Role of p38 Mitogen-Activated Protein Kinase

C. Cheroni, C. Bendotti, D. Coco, 2006, Neurodegenerative Diseases.

Glial activation and TNFR-I upregulation precedes motor dysfunction in the spinal cord of mnd mice.

P. Ghezzi, W. Buurman, A. Cagnotto, 2004, Cytokine.

Biocompatible fluorescent nanoparticles for in vivo stem cell tracking

M. Morbidelli, M. Lupi, P. Ubezio, 2013, Nanotechnology.

Talampanel reduces the level of motoneuronal calcium in transgenic mutant SOD1 mice only if applied presymptomatically

C. Bendotti, L. Siklós, J. Engelhardt, 2011, Amyotrophic lateral sclerosis : official publication of the World Federation of Neurology Research Group on Motor Neuron Diseases.

A Mouse Model of Familial ALS Has Increased CNS Levels of Endogenous Ubiquinol9/10 and Does Not Benefit from Exogenous Administration of Ubiquinol10

E. Beghi, M. Gobbi, S. Pozzi, 2013, PloS one.

Mutant Copper-Zinc Superoxide Dismutase (SOD1) Induces Protein Secretion Pathway Alterations and Exosome Release in Astrocytes

S. Pozzi, F. Fiordaliso, C. Bendotti, 2013, The Journal of Biological Chemistry.

New Insights on the Mechanisms of Disease Course Variability in ALS from Mutant SOD1 Mouse Models

C. Bendotti, V. Bonetto, Mattia Freschi, 2016, Brain pathology.

Longitudinal tracking of triple labeled umbilical cord derived mesenchymal stromal cells in a mouse model of Amyotrophic Lateral Sclerosis.

M. Salmona, C. Bendotti, R. Ferrari, 2015, Stem cell research.

Activated p38MAPK Is a Novel Component of the Intracellular Inclusions Found in Human Amyotrophic Lateral Sclerosis and Mutant SOD1 Transgenic Mice

M. Strong, R. Piva, A. Migheli, 2004, Journal of neuropathology and experimental neurology.

Persistent activation of p38 mitogen-activated protein kinase in a mouse model of familial amyotrophic lateral sclerosis correlates with disease progression

C. Rossi, A. Migheli, C. Bendotti, 2003, Molecular and Cellular Neuroscience.