J. Caldwell

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Mass‐spectrometric evaluation of HLA‐A*0201‐associated peptides identifies dominant naturally processed forms of CTL epitopes from MART‐1 and gp100

J. Shabanowitz, D. Hunt, V. Engelhard, 1999 .

oncoprotein proteasome-mediated cleavage of the lymphoid blast crisis (Lbc) The minor histocompatibility antigen HA-3 arises from differential

J. Shabanowitz, D. Hunt, V. Engelhard, 2013 .

Set2 Is a Nucleosomal Histone H3-Selective Methyltransferase That Mediates Transcriptional Repression

P. Grant, C. Allis, J. Shabanowitz, 2002, Molecular and Cellular Biology.

Preventing the Spontaneous Modification of an HLA-A2-Restricted Peptide at an N-Terminal Glutamine or an Internal Cysteine Residue Enhances Peptide Antigenicity

J. Shabanowitz, D. Hunt, V. Engelhard, 2004, Journal of immunotherapy.

The Immunogenicity of a New Human Minor Histocompatibility Antigen Results from Differential Antigen Processing

J. Shabanowitz, D. Hunt, V. Engelhard, 2001, The Journal of experimental medicine.

Melanomas with concordant loss of multiple melanocytic differentiation proteins: immune escape that may be overcome by targeting unique or undefined antigens

D. Hunt, V. Engelhard, C. Slingluff, 2000, Cancer Immunology, Immunotherapy.

Cutting edge: the HLA-A*0101-restricted HY minor histocompatibility antigen originates from DFFRY and contains a cysteinylated cysteine residue as identified by a novel mass spectrometric technique.

J. Shabanowitz, D. Hunt, V. Engelhard, 1999, Journal of immunology.

Binding of anti-GRP78 autoantibodies to cell surface GRP78 increases tissue factor procoagulant activity via the release of calcium from endoplasmic reticulum stores.

S. Pizzo, M. Gonzalez-Gronow, A. Kapoor, 2015, The Journal of Biological Chemistry.

Analysis of a natural immune response against tumor antigens in a melanoma survivor: lessons applicable to clinical trial evaluations.

D. Hunt, C. Slingluff, G. Yamshchikov, 2001, Clinical cancer research : an official journal of the American Association for Cancer Research.

Mass-spectrometric evaluation of HLA-A*0201-associated peptides identifies dominant naturally processed forms of CTL epitopes from MART-1 and gp100.

J. Shabanowitz, D. Hunt, V. Engelhard, 1999, International journal of cancer.

Active Nercc1 protein kinase concentrates at centrosomes early in mitosis and is necessary for proper spindle assembly.

A. Groen, J. Avruch, J. Roig, 2005, Molecular biology of the cell.

Postvasectomy alterations in protein synthesis and secretion in the rat caput epididymidis are not repaired after vasovasostomy.

D. Hunt, T. Turner, C. Flickinger, 2000, Journal of andrology.

A Mitotic Cascade of NIMA Family Kinases Nercc1/Nek9 ACTIVATES THE Nek6 AND Nek7 KINASES*

J. Avruch, B. Kemp, J. Roig, 2003 .

Set2 Is a Nucleosomal Histone H3-Selective Methyltransferase That Mediates Transcriptional Repression

P. Grant, C. Allis, J. Shabanowitz, 2002, Molecular and Cellular Biology.

Binding of Anti-GRP78 Autoantibodies to Cell Surface GRP78 Increases Tissue Factor Procoagulant Activity via the Release of Calcium from Endoplasmic Reticulum Stores*

S. Pizzo, M. Gonzalez-Gronow, A. Kapoor, 2010, The Journal of Biological Chemistry.

Mitotic Phosphorylation of Histone H3 Is Governed by Ipl1/aurora Kinase and Glc7/PP1 Phosphatase in Budding Yeast and Nematodes

Zu-Wen Sun, Cynthia J Brame, R. Lin, 2000, Cell.

Identification of the β cell antigen targeted by a prevalent population of pathogenic CD8+ T cells in autoimmune diabetes

J. Shabanowitz, D. Hunt, Bingye Han, 2003, Proceedings of the National Academy of Sciences of the United States of America.

Methylation of histone H4 at arginine 3 occurs in vivo and is mediated by the nuclear receptor coactivator PRMT1

Brian D. Strahl, Cynthia J. Brame, Michael R. Stallcup, 2001, Current Biology.

A Mitotic Cascade of NIMA Family Kinases

J. Avruch, B. Kemp, J. Roig, 2003, Journal of Biological Chemistry.

Gene silencing: Trans-histone regulatory pathway in chromatin

Brian D. Strahl, Zu-Wen Sun, C. Allis, 2002, Nature.

The minor Histocompatibility antigen HA-3 arises from differential proteasome-mediated cleavage of the lymphoid blast crisis (Lbc) oncoprotein

J. Shabanowitz, D. Hunt, V. Engelhard, 2003 .

Development of a continuous assay for the measurement of tissue factor procoagulant activity on intact cells

R. Austin, J. Dickhout, Ali A. Al-Hashimi, 2010, Laboratory Investigation.

The minor histocompatibility antigen HA-3 arises from differential proteasome-mediated cleavage of the lymphoid blast crisis (Lbc) oncoprotein.

J. Shabanowitz, D. Hunt, V. Engelhard, 2003, Blood.

The Immunogenicity of a New Human Minor Histocompatibility Antigen Results from Differential Antigen Processing

J. Shabanowitz, D. Hunt, V. Engelhard, 2001, The Journal of experimental medicine.

The minor histocompatibility antigen HA-3 arises from differential proteasome-mediated cleavage of the lymphoid blast crisis (Lbc) oncoprotein.

J. Shabanowitz, D. Hunt, V. Engelhard, 2003, Blood.