T. Nachman

The Mechanism of Action of β-d-2′-Deoxy-2′-Fluoro-2′-C-Methylcytidine Involves a Second Metabolic Pathway Leading to β-d-2′-Deoxy-2′-Fluoro-2′-C-Methyluridine 5′-Triphosphate, a Potent Inhibitor of the Hepatitis C Virus RNA-Dependent RNA Polymerase

M. Otto, Haiying Bao, E. Murakami, 2007, Antimicrobial Agents and Chemotherapy.

Inhibition of Hepatitis C Replicon RNA Synthesis by β-D-2′-deoxy-2′-fluoro-2′-C-Methylcytidine: A Specific Inhibitor of Hepatitis C Virus Replication

M. Otto, L. Stuyver, R. Schinazi, 2006, Antiviral chemistry & chemotherapy.

Cutting edge: IL-4 production by mast cells does not require c-maf.

M. Sherman, M. Brown, T. Nachman, 1999, Journal of immunology.

Does Not Require c-Cutting Edge : IL-4 Production by Mast Cells

Brown, M. Sherman, T. Nachman, 1999 .

N 4-Hydroxycytosine Dioxolane Nucleosides and Their Activity Against Hepatitis B Virus

M. Otto, L. Stuyver, R. Schinazi, 2005, Nucleosides, nucleotides & nucleic acids.

The mechanism of action of beta-D-2'-deoxy-2'-fluoro-2'-C-methylcytidine involves a second metabolic pathway leading to beta-D-2'-deoxy-2'-fluoro-2'-C-methyluridine 5'-triphosphate, a potent inhibitor of the hepatitis C virus RNA-dependent RNA polymerase.

M. Otto, Haiying Bao, E. Murakami, 2008, Antimicrobial agents and chemotherapy.

An intron GATA‐binding site regulates chromatin accessibility and is essential for IL‐4 gene expression in mast cells

Mildred Kwan, Melissa A Brown, T. Nachman, 2005, European journal of immunology.