A. Molojavyi

发表

Direct comparison of magnetic resonance imaging and conductance microcatheter in the evaluation of left ventricular function in mice

U. Flögel, C. Jacoby, J. Schrader, 2005, Basic Research in Cardiology.

A hemodynamic response to intravenous adenovirus vector particles is caused by systemic Kupffer cell-mediated activation of endothelial cells.

S. Kochanek, W. Bloch, N. Van Rooijen, 2003, Human gene therapy.

β-Adrenergic signaling and response to pressure overload in transgenic mice with cardiac-specific overexpression of inducible NO synthase.

M. Reinartz, T. Hohlfeld, A. Gödecke, 2011, Nitric oxide : biology and chemistry.

Dexamethasone lacks effect on blood pressure in mice with a disrupted endothelial NO synthase gene.

U. Förstermann, J. Schrader, A. Gödecke, 2004, Nitric oxide : biology and chemistry.

Myoglobin Protects the Heart from Inducible Nitric-oxide Synthase (iNOS)-mediated Nitrosative Stress*

U. Flögel, C. Jacoby, J. Schrader, 2003, Journal of Biological Chemistry.

Effects of Ketamine and Its Isomers on Ischemic Preconditioning in the Isolated Rat Heart

W. Schlack, B. Preckel, V. Thämer, 2001, Anesthesiology.

Adaptation of the myoglobin knockout mouse to hypoxic stress.

U. Flögel, C. Jacoby, J. Schrader, 2004, American journal of physiology. Regulatory, integrative and comparative physiology.

Targeted Disruption of cd73/Ecto-5′-Nucleotidase Alters Thromboregulation and Augments Vascular Inflammatory Response

K. Ley, Michael L. Smith, A. Zernecke, 2004, Circulation research.

Cardiac-Specific Overexpression of Inducible Nitric Oxide Synthase Does Not Result in Severe Cardiac Dysfunction

U. Flögel, J. Schrader, A. Gödecke, 2002, Circulation research.

Severe Cardiac Dysfunction Cardiac-Specific Overexpression of Inducible Nitric Oxide Synthase Does Not Result in

U. Flögel, J. Schrader, A. Gödecke, 2001 .

Targeted Disruption of cd73 /Ecto-5 (cid:1) -Nucleotidase Alters Thromboregulation and Augments Vascular Inflammatory Response Integrative Physiology

K. Ley, Michael L. Smith, A. Zernecke, 2004 .

Myoglobin-deficient mice activate a distinct cardiac gene expression program in response to isoproterenol-induced hypertrophy.

K. Köhrer, A. Gödecke, A. Raupach, 2010, Physiological genomics.