U. Rueckschloss

发表

The role of CEA-related cell adhesion molecule-1 (CEACAM1) in vascular homeostasis

S. Ergün, S. Kuerten, U. Rueckschloss, 2016, Histochemistry and Cell Biology.

Direct inhibition, but indirect sensitization of pacemaker activity to sympathetic tone by the interaction of endotoxin with HCN‐channels

J. Stieber, K. Werdan, M. Gekle, 2015, Clinical and experimental pharmacology & physiology.

Mechanical deformation of ventricular myocytes modulates both TRPC6 and Kir2.3 channels.

G. Isenberg, B. Husse, U. Rueckschloss, 2009, Cell Calcium.

Regulation of the endothelin system by shear stress in human endothelial cells

R. Talanow, J. Holtz, D. Darmer, 2000, The Journal of physiology.

Induction of NAD(P)H Oxidase by Oxidized Low-Density Lipoprotein in Human Endothelial Cells: Antioxidative Potential of Hydroxymethylglutaryl Coenzyme A Reductase Inhibitor Therapy

J. Holtz, J. Galle, H. Zerkowski, 2001, Circulation.

Cardiac overexpression of the human 5-HT4 receptor in mice.

P. Kirchhof, S. Hauptmann, A. Böckler, 2010, American journal of physiology. Heart and circulatory physiology.

Dose-Dependent Regulation of NAD(P)H Oxidase Expression by Angiotensin II in Human Endothelial Cells: Protective Effect of Angiotensin II Type 1 Receptor Blockade in Patients With Coronary Artery Disease

J. Holtz, H. Morawietz, M. Quinn, 2002, Arteriosclerosis, thrombosis, and vascular biology.

Endothelial barrier function is differentially regulated by CEACAM1‐mediated signaling

S. Ergün, S. Hübner, Verena Pfeiffer, 2018, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Aging aggravates heterogeneities in cell-size and stress-intolerance of cardiac ventricular myocytes

G. Isenberg, U. Rueckschloss, V. Dyachenko, 2006, Experimental Gerontology.

Differential reduction of HCN channel activity by various types of lipopolysaccharide.

K. Werdan, M. Gekle, H. Loppnow, 2011, Journal of molecular and cellular cardiology.

Increased cardiac endothelial nitric oxide synthase expression in patients taking angiotensin‐converting enzyme inhibitor therapy

G. Kojda, S. Rohrbach, J. Holtz, 2006, European journal of clinical investigation.

NOS1 induces NADPH oxidases and impairs contraction kinetics in aged murine ventricular myocytes

M. Gekle, U. Klöckner, C. Heymes, 2015, Basic Research in Cardiology.

NADPH oxidase-derived superoxide impairs calcium transients and contraction in aged murine ventricular myocytes

U. Klöckner, U. Rueckschloss, Marten Villmow, 2010, Experimental Gerontology.

Protein Phosphatase 2A Improves Cardiac Functional Response to Ischemia and Sepsis

C. Grossmann, I. Buchwalow, U. Gergs, 2022, International journal of molecular sciences.

Ca2+ transients of cardiomyocytes from senescent mice peak late and decay slowly.

G. Isenberg, U. Rueckschloss, B. Borschke, 2003, Cell calcium.

Angiotensin II induces LOX-1, the human endothelial receptor for oxidized low-density lipoprotein.

J. Holtz, T. Sawamura, B. Niemann, 1999, Circulation.

Mechanisms underlying enhanced cardiac excitation contraction coupling observed in the senescent sheep myocardium.

G. Isenberg, A. Trafford, D. Eisner, 2004, Journal of molecular and cellular cardiology.

Modulation of cardiac mechanosensitive ion channels involves superoxide, nitric oxide and peroxynitrite.

G. Isenberg, U. Rueckschloss, V. Dyachenko, 2009, Cell calcium.

The Small G-protein Rac Mediates Depolarization-induced Superoxide Formation in Human Endothelial Cells*

T. Gloe, U. Pohl, H. Sohn, 2000, The Journal of Biological Chemistry.

NADPH oxidase in endothelial cells: impact on atherosclerosis.

H. Morawietz, N. Duerrschmidt, U. Rueckschloss, 2003, Antioxidants & redox signaling.

Bone marrow-independent adventitial macrophage progenitor cells contribute to angiogenesis

S. Ergün, B. Braunger, Jochen Bauer, 2022, Cell Death & Disease.

S. Ergün, Verena Pfeiffer, S. Karnati, 2019, Aging cell.

T He Epidermal Growth Factor Receptor (egfr) Family Consists of 4 Related Tyrosine Kinase Receptors, Egfr (erbb1), Erbb2, Erbb3, and Erbb4. Loss of Epidermal Growth Factor Receptor in Vascular Smooth Muscle Cells and Cardiomyocytes Causes Arterial Hypotension and Cardiac Hypertrophy

M. Gekle, B. Schreier, C. Grossmann, 2022 .

Upregulation of vascular NAD(P)H oxidase subunit gp91phox and impairment of the nitric oxide signal transduction pathway in hypertension.

G. Kojda, N. Lauer, H. Morawietz, 2001, Biochemical and biophysical research communications.

Inhibition of cardiac pacemaker channel hHCN2 depends on intercalation of lipopolysaccharide into channel‐containing membrane microdomains

K. Werdan, M. Gekle, H. Loppnow, 2014, The Journal of physiology.

Cytochalasin D reduces Ca2+ currents via cofilin‐activated depolymerization of F‐actin in guinea‐pig cardiomyocytes

G. Isenberg, G Isenberg, U Rueckschloss, 2001, The Journal of physiology.

Henning Reductase Inhibitor Therapy Endothelial Cells : Antioxidative Potential of Hydroxymethylglutaryl Coenzyme A Induction of NAD ( P ) H Oxidase by Oxidized Low-Density Lipoprotein in Human

J. Holtz, J. Galle, H. Zerkowski, 2001 .

Low-Density Lipoprotein Angiotensin II Induces LOX-1 , the Human Endothelial Receptor for Oxidized

J. Holtz, T. Sawamura, B. Niemann, 1999 .

Differential reduction of HCN channel activity by various types of lipopolysaccharide.

K. Werdan, M. Gekle, H. Loppnow, 2011, Journal of molecular and cellular cardiology.

Contraction augments L‐type Ca2+ currents in adherent guinea‐pig cardiomyocytes

G. Isenberg, U. Rueckschloss, 2004, The Journal of physiology.

M. Gekle, B. Schreier, C. Grossmann, 2022 .