Dan Song

发表

Controlling load-dependent contractility of the heart at the single molecule level

J. Spudich, K. Ruppel, M. Kawana, 2018, bioRxiv.

Controlling load-dependent kinetics of β-cardiac myosin at the single molecule level

J. Spudich, K. Ruppel, M. Kawana, 2018, Nature Structural & Molecular Biology.

Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super relaxed state

J. Spudich, K. Campbell, B. Pruitt, 2020, Proceedings of the National Academy of Sciences.

A small-molecule myosin inhibitor as a targeted multi-stage antimalarial

Paul V. Ruijgrok, E. Winzeler, J. Spudich, 2022, bioRxiv.

β-Cardiac myosin hypertrophic cardiomyopathy mutations release sequestered heads and increase enzymatic activity

J. Spudich, D. Bernstein, A. Adhikari, 2019, Nature Communications.

Hypertrophic cardiomyopathy mutations at the folded-back sequestered β-cardiac myosin S1-S2 and S1-S1 interfaces release sequestered heads and increase myosin enzymatic activity

J. Spudich, D. Bernstein, A. Adhikari, 2019, bioRxiv.

SETD3 is an Actin Histidine Methyltransferase that Prevents Primary Dystocia

Joshua E. Elias, Xiaodong Cheng, J. Spudich, 2018, Nature.

β-Cardiac myosin hypertrophic cardiomyopathy mutations release sequestered heads and increase enzymatic activity

J. Spudich, D. Bernstein, A. Adhikari, 2019, Nature Communications.

Controlling load-dependent kinetics of β-cardiac myosin at the single molecule level

J. Spudich, K. Ruppel, M. Kawana, 2018, Nature Structural & Molecular Biology.

SETD3 is an Actin Histidine Methyltransferase that Prevents Primary Dystocia

Joshua E. Elias, J. Spudich, J. Carette, 2018, Nature.

Main Manuscript for Hypertrophic cardiomyopathy ß-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super-relaxed state

J. Spudich, G. Pardon, Gwanghyun Jung, 2022 .