A. M. Bodles

发表

APP-BP1 inhibits Aβ42 levels by interacting with Presenilin-1

R. Neve, R. Mrak, W. Griffin, 2007, Molecular Neurodegeneration.

The N‐terminal region of non‐Aβ component of Alzheimer's Disease amyloid is responsible for its tendency to assume β‐sheet and aggregate to form fibrils

G. Irvine, P. Harriott, A. M. Bodles, 1998 .

Retinol binding protein 4 expression in humans: relationship to insulin resistance, inflammation, and response to pioglitazone.

Bounleut Phanavanh, S. Fried, P. Kern, 2007, The Journal of clinical endocrinology and metabolism.

Inhibition of fibril formation and toxicity of a fragment of α-synuclein by an N-methylated peptide analogue

G. Irvine, B. Greer, A. M. Bodles, 2004, Neuroscience Letters.

Thrombospondin-1 Is an Adipokine Associated With Obesity, Adipose Inflammation, and Insulin Resistance

R. Nagarajan, C. Peterson, Bounleut Phanavanh, 2008, Diabetes.

Calcium is involved in formation of high molecular weight adiponectin.

P. Kern, G. Ranganathan, N. Rasouli, 2008, Metabolic syndrome and related disorders.

Pioglitazone increases secretion of high-molecular-weight adiponectin from adipocytes.

P. Kern, N. Rasouli, A. M. Bodles, 2006, American journal of physiology. Endocrinology and metabolism.

Pioglitazone induces apoptosis of macrophages in human adipose tissue Published, JLR Papers in Press, June 23, 2006.

M. Wabitsch, C. Peterson, Bounleut Phanavanh, 2006, Journal of Lipid Research.

Stearoyl-coenzyme A desaturase 1 gene expression increases after pioglitazone treatment and is associated with peroxisomal proliferator-activated receptor-gamma responsiveness.

Bounleut Phanavanh, P. Kern, G. Ranganathan, 2008, The Journal of clinical endocrinology and metabolism.

Human visfatin expression: relationship to insulin sensitivity, intramyocellular lipids, and inflammation.

Bounleut Phanavanh, S. Fried, P. Kern, 2007, The Journal of clinical endocrinology and metabolism.

Thrombospondin-1 (TSP1) is an adipokine associated with obesity, adipose inflammation and insulin resistance

C. Peterson, S. Fried, P. Kern, 2007 .

Induction of serine racemase expression and D-serine release from microglia by amyloid β-peptide

S. Barger, W. Griffin, Shengzhou or sheng-zhou wu, 2004, Journal of Neuroinflammation.

Amyloid precursor protein modulates β-catenin degradation

Yuzhi Chen, A. M. Bodles, 2007, Journal of Neuroinflammation.

Cytokines and the aging brain – what we don't know might help us

S. Barger, A. M. Bodles, 2004, Trends in Neurosciences.

Toxicity of non-abeta component of Alzheimer's disease amyloid, and N-terminal fragments thereof, correlates to formation of beta-sheet structure and fibrils.

G. Irvine, P. Harriott, P Harriott, 2000, European journal of biochemistry.

Secreted β-amyloid precursor protein activates microglia via JNK and p38-MAPK

S. Barger, A. M. Bodles, 2005, Neurobiology of Aging.

Identification of the region of non‐Aβ component (NAC) of Alzheimer's disease amyloid responsible for its aggregation and toxicity

G. Irvine, B. Greer, A M Bodles, 2001, Journal of neurochemistry.

Secreted beta-amyloid precursor protein activates microglia via JNK and p38-MAPK.

S. Barger, Angela M Bodles, Steven W Barger, 2005, Neurobiology of aging.

Alpha-synuclein aggregation.

G. Irvine, Angela M Bodles, G Brent Irvine, 2004, Protein and peptide letters.