A. Schlicksupp

发表

Human BACE Forms Dimers and Colocalizes with APP*

T. Bayer, G. Multhaup, R. Pipkorn, 2004, Journal of Biological Chemistry.

Copper-binding amyloid precursor protein undergoes a site-specific fragmentation in the reduction of hydrogen peroxide.

C. Masters, G. Multhaup, K. Beyreuther, 1998, Biochemistry.

Amyloid-β Fosters p35/CDK5 Signaling Contributing to Changes of Inhibitory Synapses in Early Stages of Cerebral Amyloidosis.

É. Kiss, J. Kirsch, S. Kins, 2020, Journal of Alzheimer's disease : JAD.

Cyclin-Dependent Kinase 5 Is Involved in the Phosphorylation of Gephyrin and Clustering of GABAA Receptors at Inhibitory Synapses of Hippocampal Neurons

J. Kirsch, A. Schlicksupp, J. Kuhse, 2014, PloS one.

The Amyloid Precursor Protein of Alzheimer's Disease in the Reduction of Copper(II) to Copper(I)

C. Masters, G. Multhaup, K. Beyreuther, 1996, Science.

Artesunate restores the levels of inhibitory synapse proteins and reduces amyloid-β and C-terminal fragments (CTFs) of the amyloid precursor protein in an AD-mouse model

É. Kiss, J. Kirsch, S. Kins, 2021, Molecular and Cellular Neuroscience.

Components of the Translational Machinery Are Associated with Juvenile Glycine Receptors and Are Redistributed to the Cytoskeleton upon Aging and Synaptic Activity*

M. Karas, J. Kirsch, A. Schlicksupp, 2007, Journal of Biological Chemistry.

Reactive oxygen species and Alzheimer's disease.

C. Masters, G. Multhaup, K. Beyreuther, 1997, Biochemical pharmacology.

Artemisinin-treatment in pre-symptomatic APP-PS1 mice increases gephyrin phosphorylation at Ser270: a modification regulating postsynaptic GABAAR density

É. Kiss, J. Kirsch, S. Kins, 2021, Biological chemistry.

Biphasic Alteration of the Inhibitory Synapse Scaffold Protein Gephyrin in Early and Late Stages of an Alzheimer Disease Model.

É. Kiss, J. Kirsch, S. Kins, 2016, The American journal of pathology.

Phosphorylation of Gephyrin in Hippocampal Neurons by Cyclin-dependent Kinase CDK5 at Ser-270 Is Dependent on Collybistin

J. Kirsch, R. Nawrotzki, Susanne Mükusch, 2012, The Journal of Biological Chemistry.

Identification of a β-Secretase Activity, Which Truncates Amyloid β-Peptide after Its Presenilin-dependent Generation*

G. Westmeyer, G. Multhaup, W. Bode, 2003, The Journal of Biological Chemistry.

Synaptic targeting of N-methyl-d-aspartate receptor splice variants is regulated differentially by receptor activity

A. Schlicksupp, T. Pauly, J. Kuhse, 2005, Neuroscience.

Activity-Dependent Shedding of the NMDA Receptor Glycine Binding Site by Matrix Metalloproteinase 3: A PUTATIVE Mechanism of Postsynaptic Plasticity

J. Kirsch, A. Schlicksupp, T. Pauly, 2008, PloS one.

Kinetic Analysis of the Interaction of the Copper Chaperone Atox1 with the Metal Binding Sites of the Menkes Protein*

G. Multhaup, R. Pipkorn, Gerd Multhaup, 2003, Journal of Biological Chemistry.

Possible mechanisms of APP-mediated oxidative stress in Alzheimer's disease.

T. Bayer, G. Multhaup, R. Cappai, 2002, Free radical biology & medicine.

Evidence for a copper-binding superfamily of the amyloid precursor protein.

Carsten Schmidt, Colin L Masters, Roberto Cappai, 2002, Biochemistry.

KCC2 knockdown impairs glycinergic synapse maturation in cultured spinal cord neurons

A. Draguhn, C. Bruehl, J. Kirsch, 2016, Histochemistry and Cell Biology.

619 Reduction of copper(II) to copper(I) by the Amyloid Precursor Protein (APP) of Alzheimer's disease

G. Multhaup, K. Beyreuther, D. Beher, 1996, Neurobiology of Aging.

Effects of distinct collybistin isoforms on the formation of GABAergic synapses in hippocampal neurons

T. Kuner, C. Körber, J. Kirsch, 2012, Molecular and Cellular Neuroscience.