I. Eichler

发表

Shear stress-induced up-regulation of the intermediate-conductance Ca(2+)-activated K(+) channel in human endothelium.

I. Grgic, R. Köhler, J. Hoyer, 2003, Cardiovascular research.

Blockade of the Intermediate-Conductance Calcium-Activated Potassium Channel as a New Therapeutic Strategy for Restenosis

K. Chandy, H. Orzechowski, I. Grgic, 2003, Circulation.

Selective blockade of endothelial Ca2+‐activated small‐ and intermediate‐conductance K+‐channels suppresses EDHF‐mediated vasodilation

A. Pries, I. Grgic, R. Köhler, 2003, British journal of pharmacology.

Selective Blockade of the Intermediate-Conductance Ca2+-Activated K+ Channel Suppresses Proliferation of Microvascular and Macrovascular Endothelial Cells and Angiogenesis In Vivo

I. Grgic, R. Köhler, J. Hoyer, 2005, Arteriosclerosis, thrombosis, and vascular biology.

Impaired EDHF-mediated vasodilation and function of endothelial Ca-activated K channels in uremic rats.

I. Grgic, R. Köhler, J. Hoyer, 2005, Kidney international.

Modulation of Ca2+-activated K+ channel in renal artery endothelium in situ by nitric oxide and reactive oxygen species.

R. Köhler, J. Hoyer, S. Brakemeier, 2003, Kidney international.

Blockade of T-lymphocyte KCa3.1 and Kv1.3 channels as novel immunosuppression strategy to prevent kidney allograft rejection.

I. Grgic, H. Wulff, R. Köhler, 2009, Transplantation proceedings.

Endothelial calcium‐activated K+‐channels and EDHF signalling

R. Köhler, I. Eichler, 2003 .

Up-regulation of endothelial stretch-activated cation channels by fluid shear stress.

R. Köhler, J. Hoyer, S. Brakemeier, 2002, Cardiovascular research.