E. Dirkx

发表

Paracrine effect of regulatory T cells promotes cardiomyocyte proliferation during pregnancy and after myocardial infarction

V. Martinelli, C. Long, L. Zentilin, 2018, Nature Communications.

Phosphatidylinositol 4-kinase IIIβ mediates contraction-induced GLUT4 translocation and shows its anti-diabetic action in cardiomyocytes

D. Neumann, J. Glatz, J. Luiken, 2020, Cellular and Molecular Life Sciences.

A new leptin-mediated mechanism for stimulating fatty acid oxidation: a pivotal role for sarcolemmal FAT/CD36.

A. Bonen, I. Momken, A. Chabowski, 2017, The Biochemical journal.

Requirement for distinct vesicle-associated membrane proteins in insulin- and AMP-activated protein kinase (AMPK)-induced translocation of GLUT4 and CD36 in cultured cardiomyocytes

R. Schwenk, A. Klip, A. Bonen, 2010, Diabetologia.

Overexpression of Vesicle-associated Membrane Protein (VAMP) 3, but Not VAMP2, Protects Glucose Transporter (GLUT) 4 Protein Translocation in an in Vitro Model of Cardiac Insulin Resistance*

R. Schwenk, A. Bonen, J. Broers, 2012, The Journal of Biological Chemistry.

Endoplasmic reticulum-associated degradation of the mouse PC1/3-N222D hypomorph and human PCSK1 mutations contributes to obesity

F. Schuit, Lieven Thorrez, J. Creemers, 2016, International Journal of Obesity.

Protein kinase-D1 overexpression prevents lipid-induced cardiac insulin resistance.

R. Schwenk, D. Neumann, B. Janssen, 2014, Journal of molecular and cellular cardiology.

Regulation of fetal gene expression in heart failure.

L. D. de Windt, P. A. da Costa Martins, Ellen Dirkx, 2013, Biochimica et biophysica acta.

Cardiac regenerative medicine: At the crossroad of microRNA function and biotechnology

E. Dirkx, Andrea Raso, 2017, Non-coding RNA research.

Non-coding RNA in control of gene regulatory programs in cardiac development and disease.

L. D. de Windt, E. Dirkx, L. Philippen, 2015, Journal of molecular and cellular cardiology.

The hypoxia-inducible microRNA cluster miR-199a∼214 targets myocardial PPARδ and impairs mitochondrial fatty acid oxidation.

Elizabeth A. McClellan, M. Mayr, Y. Pinto, 2013, Cell metabolism.

Nfat and miR-25 cooperate to reactivate the transcription factor Hand2 in heart failure

S. Heymans, M. Gladka, P. Volders, 2013, Nature Cell Biology.

Common Regulatory Pathways Mediate Activity of MicroRNAs Inducing Cardiomyocyte Proliferation

M. Marsili, L. Zentilin, M. Giacca, 2019, Cell reports.

Cardiovascular extracellular microRNAs: emerging diagnostic markers and mechanisms of cell-to-cell RNA communication

L. Windt, Ellen Dirkx, Virginie Kinet, 2013, Front. Genet..

Antisense MicroRNA Therapeutics in Cardiovascular Disease: Quo Vadis?

L. D. de Windt, P. A. da Costa Martins, E. Dirkx, 2015, Molecular therapy : the journal of the American Society of Gene Therapy.

High fat diet induced diabetic cardiomyopathy.

R. Schwenk, G. V. van Eys, J. Glatz, 2011, Prostaglandins, leukotrienes, and essential fatty acids.

Detection of cardiac myosin binding protein-C (cMyBP-C) by a phospho-specific PKD antibody in contracting rat cardiomyocytes

E. Mariman, D. Vertommen, F. Bouwman, 2013 .

Protein kinase D increases maximal Ca2+-activated tension of cardiomyocyte contraction by phosphorylation of cMyBP-C-Ser315.

R. Schwenk, G. V. van Eys, J. Van Lint, 2012, American journal of physiology. Heart and circulatory physiology.

Protein kinase D increases maximal Ca 2 (cid:1) -activated tension of cardiomyocyte contraction by phosphorylation of cMyBP-C-Ser 315

R. Schwenk, S. Sadayappan, L. Carrier, 2012 .

Protein Kinase D1 Is Essential for Contraction-induced Glucose Uptake but Is Not Involved in Fatty Acid Uptake into Cardiomyocytes*

B. Viollet, R. Schwenk, A. Bonen, 2011, The Journal of Biological Chemistry.

MicroRNA 214 Is a Potential Regulator of Thyroid Hormone Levels in the Mouse Heart Following Myocardial Infarction, by Targeting the Thyroid-Hormone-Inactivating Enzyme Deiodinase Type III

W. Paulus, Rob Janssen, C. Oudejans, 2016, Front. Endocrinol..

Author Correction: A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration

M. Giacca, M. Huibers, L. D. de Windt, 2022, Nature Communications.

Common Regulatory Pathw ays Mediate Activity of MicroRNAs Inducing Cardiomyocyte Proliferation Graphical

M. Marsili, L. Zentilin, M. Giacca, 2019 .

Therapeutic Delivery of miR-148a Suppresses Ventricular Dilation in Heart Failure

Yolan J. Reckman, L. Zentilin, M. Giacca, 2018, Molecular therapy : the journal of the American Society of Gene Therapy.

A microRNA program regulates the balance between cardiomyocyte hyperplasia and hypertrophy and stimulates cardiac regeneration

M. Giacca, M. Huibers, L. D. de Windt, 2021, Nature Communications.

Overexpression of AMP-activated protein kinase or protein kinase D prevents lipid-induced insulin resistance in cardiomyocytes.

B. Viollet, L. Bertrand, M. Foretz, 2013, Journal of molecular and cellular cardiology.

Antisense MicroRNA Therapeutics in Cardiovascular Disease: Quo Vadis?

L. D. de Windt, P. A. da Costa Martins, E. Dirkx, 2015, Molecular therapy : the journal of the American Society of Gene Therapy.

A microRNA program controls the transition of cardiomyocyte hyperplasia to hypertrophy and stimulates mammalian cardiac regeneration

M. Giacca, M. Huibers, L. D. de Windt, 2021, bioRxiv.