Michelle S. Parvatiyar

发表

Cardiac Troponin Mutations and Restrictive Cardiomyopathy

J. Potter, J. Pinto, Michelle S. Parvatiyar, 2010, Journal of biomedicine & biotechnology.

Cardiovascular Injury Due to SARS-CoV-2

J. Bugert, Michelle S. Parvatiyar, I. Valera, 2021, Current Clinical Microbiology Reports.

Clinical and Functional Characterization of TNNT2 Mutations Identified in Patients With Dilated Cardiomyopathy

J. Potter, R. Hershberger, Duanxiang Li, 2009, Circulation. Cardiovascular genetics.

Stabilization of the cardiac sarcolemma by sarcospan rescues DMD-associated cardiomyopathy.

Jamie L. Marshall, J. Chamberlain, A. Beedle, 2019, JCI insight.

Sarcospan Regulates Cardiac Isoproterenol Response and Prevents Duchenne Muscular Dystrophy–Associated Cardiomyopathy

Jamie L. Marshall, Michelle S. Parvatiyar, R. H. Crosbie-Watson, 2015, Journal of the American Heart Association.

Functional Characterization of TNNC1 Rare Variants Identified in Dilated Cardiomyopathy*

N. Norton, J. Potter, R. Hershberger, 2011, The Journal of Biological Chemistry.

A Troponin T Mutation That Causes Infantile Restrictive Cardiomyopathy Increases Ca2+ Sensitivity of Force Development and Impairs the Inhibitory Properties of Troponin*

J. Potter, Michelle A Jones, Jingsheng Liang, 2008, Journal of Biological Chemistry.

In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 Is a Hypertrophic Cardiomyopathy Susceptibility Gene.

M. Ackerman, M. Vukmirovic, J. Bos, 2015, Circulation. Cardiovascular genetics.

In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 is a Hypertrophic Cardiomyopathy Susceptibility Gene

M. Vukmirovic, D. Zorio, M. Sanchez-Gonzalez, 2014 .

Fetal Cardiac Troponin Isoforms Rescue the Increased Ca2+ Sensitivity Produced by a Novel Double Deletion in Cardiac Troponin T Linked to Restrictive Cardiomyopathy

J. Potter, G. Andelfinger, Marc-Phillip Hitz, 2011, The Journal of Biological Chemistry.

Transgenic mice expressing Na+-K+-ATPase in smooth muscle decreases blood pressure.

Tracy J. Pritchard, J. Lorenz, R. Paul, 2007, American journal of physiology. Heart and circulatory physiology.

On 'The content of troponin, tropomyosin, actin, and myosin in rabbit skeletal muscle myofibrils' by James D. Potter.

J. Pinto, Michelle S. Parvatiyar, 2022, Archives of biochemistry and biophysics.

In Vivo Analysis of Troponin C Knock-In (A8V) MiceCLINICAL PERSPECTIVE

M. Ackerman, M. Vukmirovic, J. Bos, 2015 .

Autoimmunity as a sequela to obesity and systemic inflammation

Michelle S. Parvatiyar, I. Valera, K. Parvatiyar, 2022, Frontiers in Physiology.

Editorial: Skeletal muscle in age-related diseases: From molecular pathogenesis to potential interventions

R. Qaisar, Michelle S. Parvatiyar, 2022, Frontiers in Physiology.

The Functional Properties of Human Slow Skeletal Troponin T Isoforms in Cardiac Muscle Regulation*

A. Gomes, J. Potter, T. Miller, 2012, The Journal of Biological Chemistry.

Hypertrophic Cardiomyopathy Cardiac Troponin C Mutations Differentially Affect Slow Skeletal and Cardiac Muscle Regulation

P. Chase, A. Landstrom, Maicon Landim-Vieira, 2017, Front. Physiol..

Pathogenesis associated with a restrictive cardiomyopathy mutant in cardiac troponin T is due to reduced protein stability and greatly increased myofilament Ca2+ sensitivity.

J. Pinto, Michelle S. Parvatiyar, 2015, Biochimica et biophysica acta.

A Mutation in TNNC1-encoded Cardiac Troponin C, TNNC1-A31S, Predisposes to Hypertrophic Cardiomyopathy and Ventricular Fibrillation*

J. Potter, M. Ackerman, A. Landstrom, 2012, The Journal of Biological Chemistry.

Predicting Cardiomyopathic Phenotypes by Altering Ca2+ Affinity of Cardiac Troponin C*

J. Potter, Jose Renato Pinto, Michelle S Parvatiyar, 2010, The Journal of Biological Chemistry.

A Functional and Structural Study of Troponin C Mutations Related to Hypertrophic Cardiomyopathy*

Michael J Ackerman, J. Potter, M. Ackerman, 2009, The Journal of Biological Chemistry.

Post-translational modification patterns on β-myosin heavy chain are altered in ischemic and nonischemic human hearts

M. Regnier, P. Chase, M. Childers, 2022, eLife.

Meta-analysis of cardiomyopathy-associated variants in troponin genes identifies loci and intragenic hotspots that are associated with worse clinical outcomes.

S. Datta, P. Chase, Jeffrey J. Kim, 2020, Journal of molecular and cellular cardiology.

The missing links within troponin.

Wei Yang, Guilherme A. P. de Oliveira, M. A. Marques, 2019, Archives of biochemistry and biophysics.

Mutations in Troponin that cause HCM, DCM AND RCM: what can we learn about thin filament function?

A. Gomes, J. Potter, Audrey N. Chang, 2010, Journal of molecular and cellular cardiology.

Anomalous structural dynamics of minimally frustrated residues in cardiac troponin C triggers hypertrophic cardiomyopathy†

P. Chase, Jerson L. Silva, E. Cino, 2021, Chemical science.

A comprehensive guide to genetic variants and post-translational modifications of cardiac troponin C

P. Chase, A. Landstrom, Maicon Landim-Vieira, 2020, Journal of Muscle Research and Cell Motility.

Molecular and functional characterization of novel hypertrophic cardiomyopathy susceptibility mutations in TNNC1-encoded troponin C.

S. Ommen, J. Potter, D. Tester, 2007, Journal of molecular and cellular cardiology.

Troponin and cardiomyopathy.

J. Potter, Audrey N. Chang, Michelle S. Parvatiyar, 2008, Biochemical and biophysical research communications.

Essential roles of the dystrophin-glycoprotein complex in different cardiac pathologies.

H. Hwang, Christina Holmes, O. Laitano, 2020, Advances in medical sciences.

Sexual dimorphism in cardiac transcriptome associated with a troponin C murine model of hypertrophic cardiomyopathy

P. Chase, J. Pinto, Michelle S. Parvatiyar, 2020, Physiological reports.

Strong Cross-bridges Potentiate the Ca2+ Affinity Changes Produced by Hypertrophic Cardiomyopathy Cardiac Troponin C Mutants in Myofilaments

J. Potter, Jingsheng Liang, J. Pinto, 2010, The Journal of Biological Chemistry.

Post-translational modification patterns on β-myosin heavy chain are altered in ischemic and non-ischemic human hearts

M. Regnier, P. Chase, M. Childers, 2021, bioRxiv.

Strong Cross-bridges Potentiate the Ca 2 (cid:1) Affinity Changes Produced by Hypertrophic Cardiomyopathy Cardiac Troponin C Mutants in Myofilaments A FAST KINETIC APPROACH *

J. Potter, Jingsheng Liang, J. Pinto, 2010 .

A comprehensive guide to genetic variants and post-translational modifications of cardiac troponin C

P. Chase, A. Landstrom, Maicon Landim-Vieira, 2020, Journal of Muscle Research and Cell Motility.

Quantitative analysis of chromatin proteomes in disease.

S. Franklin, T. Vondriska, Emma Monte, 2012, Journal of visualized experiments : JoVE.

Sarcospan Deficiency Increases Oxidative Stress and Arrhythmias in Hearts after Acute Ischemia-Reperfusion Injury

H. Hwang, Michelle S. Parvatiyar, I. Valera, 2023, International journal of molecular sciences.

Stabilization of the cardiac sarcolemma by sarcospan rescues DMD-associated cardiomyopathy.

Aaron M. Beedle, Jamie L. Marshall, J. Chamberlain, 2019, JCI insight.

Abstract 539: Molecular and Functional Characterization of Novel Hypertrophic Cardiomyopathy Susceptibility Mutations in TNNC1-Encoded Troponin C

S. Ommen, J. Potter, M. L. Marquardt, 2007 .

Functional Characterization of TNNC1 Rare Variants Identified in Dilated Cardiomyopathy*

N. Norton, J. Potter, R. Hershberger, 2011, The Journal of Biological Chemistry.

In Vivo Analysis of Troponin C Knock-In (A8V) Mice: Evidence that TNNC1 Is a Hypertrophic Cardiomyopathy Susceptibility Gene.

M. Vukmirovic, J. Jin, D. Zorio, 2015, Circulation. Cardiovascular genetics.

Structural considerations for chromatin state models with transcription as a functional readout

S. Franklin, T. Vondriska, Emma Monte, 2012, FEBS letters.