C. Pavoine

发表

Activation of Na+/Ca2+ exchange by adenosine in ewe heart sarcolemma is mediated by a pertussis toxin-sensitive G protein.

S. Lotersztajn, F. Pecker, V. Brechler, 1990, The Journal of biological chemistry.

beta2-Adrenergic signaling in human heart: shift from the cyclic AMP to the arachidonic acid pathway.

F. Roudot-thoraval, P. Duvaldestin, A. Pawlak, 2003, Molecular pharmacology.

Glucagon-(19-29) exerts a biphasic action on the liver plasma membrane Ca2+ pump which is mediated by G proteins.

D. Bataille, S. Lotersztajn, D. Le-Nguyen, 1990, The Journal of biological chemistry.

N-Acetylcysteine Prevents the Deleterious Effect of Tumor Necrosis Factor-&agr; on Calcium Transients and Contraction in Adult Rat Cardiomyocytes

T. Levade, F. Pecker, N. Andrieu-Abadie, 2004, Circulation.

Structural Localization and Expression of CXCL12 and CXCR4 in Rat Heart and Isolated Cardiac Myocytes

E. Deroubaix, C. Rücker-Martin, A. Lombet, 2007, The journal of histochemistry and cytochemistry : official journal of the Histochemistry Society.

Sphingomyelinases: their regulation and roles in cardiovascular pathophysiology.

F. Pecker, C. Pavoine, 2009, Cardiovascular research.

M2 kupffer cells promote hepatocyte senescence: an IL-6-dependent protective mechanism against alcoholic liver disease.

J. Wan, S. Lotersztajn, M. Benkdane, 2014, The American journal of pathology.

Cannabinoid CB2 receptors protect against alcoholic liver disease by regulating Kupffer cell polarization in mice

A. Mallat, S. Lotersztajn, A. Zimmer, 2011, Hepatology.

The cannabinoid receptor type 2 promotes cardiac myocyte and fibroblast survival and protects against ischemia/reperfusion‐induced cardiomyopathy

J. Wan, S. Lotersztajn, A. Zimmer, 2009, The FASEB Journal.

Xanthine affects [Ca2+]i and contractile responses of ventricular cardiocytes to electrical stimulation.

F. Pecker, A. Sauvadet, T. Rohn, 1997, The American journal of physiology.

Neutral sphingomyelinase inhibition participates to the benefits of N-acetylcysteine treatment in post-myocardial infarction failing heart rats.

P. Mulder, L. Hittinger, G. Candiani, 2007, Journal of molecular and cellular cardiology.

Early Protective Role of Inflammation in Cardiac Remodeling and Heart Failure: Focus on TNFα and Resident Macrophages

S. Besse, E. Balse, S. Nadaud, 2022, Cells.

Early activation of the cardiac CX3CL1/CX3CR1 axis delays β-adrenergic-induced heart failure

M. Keck, C. Combadière, E. Gautier, 2021, Scientific Reports.

[Calcium and cell death].

A. Mallat, S. Lotersztajn, F. Pecker, 1987, Gastroenterologie clinique et biologique.

Evidence for a beta2-adrenergic/arachidonic acid pathway in ventricular cardiomyocytes. Regulation by the beta1-adrenergic/camp pathway.

F. Pecker, A. Sauvadet, C. Pavoine, 1999, The Journal of biological chemistry.

Transcriptomic and Lipidomic Mapping of Macrophages in the Hub of Chronic Beta-Adrenergic-Stimulation Unravels Hypertrophy-, Proliferation-, and Lipid Metabolism-Related Genes as Novel Potential Markers of Early Hypertrophy or Heart Failure

W. Le Goff, E. Balse, S. Nadaud, 2022, Biomedicines.

Arachidonic Acid Drives Mini-glucagon Action in Cardiac Cells*

F. Pecker, A. Sauvadet, T. Rohn, 1997, The Journal of Biological Chemistry.

The Cytosolic Phospholipase A2 Pathway, a Safeguard of β2-Adrenergic Cardiac Effects in Rat*

P. Duvaldestin, G. Candiani, F. Pecker, 2005, Journal of Biological Chemistry.

TNFR1 and TNFR2 Signaling Interplay in Cardiac Myocytes*

F. Pecker, N. Defer, C. Pavoine, 2007, Journal of Biological Chemistry.

Arachidonic acid mediates dual effect of TNF-alpha on Ca2+ transients and contraction of adult rat cardiomyocytes.

F. Pecker, C. Pavoine, Aïssata Amadou, 2002, American journal of physiology. Cell physiology.

Evidence for a β2-Adrenergic/Arachidonic Acid Pathway in Ventricular Cardiomyocytes

F. Pecker, A. Sauvadet, C. Pavoine, 1999, The Journal of Biological Chemistry.

The Platelet-Derived Growth Factor Pathway in Pulmonary Arterial Hypertension: Still an Interesting Target?

F. Soubrier, S. Nadaud, C. Pavoine, 2022, Life.

Platelet‐Derived Growth Factor Receptor Type α Activation Drives Pulmonary Vascular Remodeling Via Progenitor Cell Proliferation and Induces Pulmonary Hypertension

M. Humbert, R. Harvey, F. Soubrier, 2022, Journal of the American Heart Association.

The high affinity (Ca2+-Mg2+)-ATPase in liver plasma membranes is a Ca2+ pump. Reconstitution of the purified enzyme into phospholipid vesicles.

A. Mallat, S. Lotersztajn, F. Pecker, 1987, The Journal of biological chemistry.

A glucagon fragment is responsible for the inhibition of the liver Ca2+ pump by glucagon

D. Bataille, A. Mallat, S. Lotersztajn, 1987, Nature.

Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling

M. Nonnenmacher, J. Hulot, R. Hajjar, 2016, Circulation.

Cardiac Stim 1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC 2 / Akt Signaling

M. Nonnenmacher, J. Hulot, R. Hajjar, 2016 .

P. Blache, D. Bataille, A. Mallat, 1988, Annals of the New York Academy of Sciences.

Emergence of Orai3 activity during cardiac hypertrophy.

J. Hulot, M. Keck, M. Trebak, 2015, Cardiovascular research.

Glucagon stimulates the cardiac Ca2+ current by activation of adenylyl cyclase and inhibition of phosphodiesterase

R. Fischmeister, P. Mery, F. Pecker, 1990, Nature.

Beta(2)-adrenergic receptor agonists increase intracellular free Ca(2+) concentration cycling in ventricular cardiomyocytes through p38 and p42/44 MAPK-mediated cytosolic phospholipase A(2) activation.

F. Pecker, C. Pavoine, S. Magne, 2001, The Journal of biological chemistry.

Hormonal and Non-Hormonal Regulation of Ca2+ Current and Adenylate Cyclase in Cardiac Cells

A. Shrier, R. Fischmeister, P. Mery, 1990 .

Erythro-9-(2-hydroxy-3-nonyl)adenine inhibits cyclic GMP-stimulated phosphodiesterase in isolated cardiac myocytes.

R. Fischmeister, P. Mery, F. Pecker, 1995, Molecular pharmacology.

Biological Effects of C-type Natriuretic Peptide in Human Myofibroblastic Hepatic Stellate Cells*

A. Mallat, S. Lotersztajn, J. Nhieu, 1999, The Journal of Biological Chemistry.

Hormonal Inhibition of the Liver Plasma Membrane (Ca 2+ —Mg 2+ ) ATPase is Mediated by a G s -like Protein

P. Insel, D. Stengel, A. Mallat, 1989 .

Inhibition by glucagon of the cGMP-inhibited low-Km cAMP phosphodiesterase in heart is mediated by a pertussis toxin-sensitive G-protein.

R. Fischmeister, R. Hanf, F. Pecker, 1992, The Journal of biological chemistry.

Regulation of liver plasma membrane Ca2+ pump.

A. Mallat, S. Lotersztajn, F. Pecker, 1988, Advances in experimental medicine and biology.

The cardiac β2-adrenergic signalling a new role for the cPLA2

N. Defer, C. Pavoine, 2005 .

Calcium dynamics in cardiac myocytes: a model for drugs effect description

Yskandar Hamam, Rosalba Lamanna, Marisol Delgado, 2004, Simul. Model. Pract. Theory.

Glucagon III

J. Holst, G. Paolisso, D. Bataille, 1996, Handbook of Experimental Pharmacology.

M2 Kupffer cells promote M1 Kupffer cell apoptosis: A protective mechanism against alcoholic and nonalcoholic fatty liver disease

Albert Tran, Sophie Lotersztajn, Alexandre Louvet, 2014, Hepatology.

Adult cardiac myocytes survive and remain excitable during long-term culture on synthetic supports.

S. Hatem, E. Deroubaix, T. Folliguet, 2001, The Journal of thoracic and cardiovascular surgery.

Glutathione Deficiency in Cardiac Patients Is Related to the Functional Status and Structural Cardiac Abnormalities

F. Roudot-thoraval, J. Dubois-Randé, L. Hittinger, 2009, PloS one.

Synergistic actions of glucagon and miniglucagon on Ca2+ mobilization in cardiac cells.

F. Pecker, A. Sauvadet, T. Rohn, 1996, Circulation research.

Inhibition of the sarcolemmal Ca2+ pump in embryonic chick heart cells by mini-glucagon.

F. Pecker, A. Sauvadet, C. Pavoine, 1995, Cell calcium.

Miniglucagon [glucagon-(19-29)] is a component of the positive inotropic effect of glucagon.

P. Blache, D. Bataille, D. Le-Nguyen, 1991, The American journal of physiology.

Glucagon-(19-29), a Ca2+ pump inhibitory peptide, is processed from glucagon in the rat liver plasma membrane by a thiol endopeptidase.

P. Blache, D. Bataille, S. Lotersztajn, 1990, The Journal of biological chemistry.

β2-Adrenergic Receptor Agonists Increase Intracellular Free Ca2+ Concentration Cycling in Ventricular Cardiomyocytes through p38 and p42/44 MAPK-mediated Cytosolic Phospholipase A2 Activation*

F. Pecker, C. Pavoine, S. Magne, 2001, The Journal of Biological Chemistry.

Molecular Pathogenesis of Genetic and Inherited Diseases Delayed Cardiomyopathy in Dystrophin Deficient mdx Mice Relies on Intrinsic Glutathione Resource

K. Wahbi, C. Christov, F. Pecker, 2010 .

Role of G protein beta gamma subunits in the regulation of the plasma membrane Ca2+ pump.

D. Bataille, J. Capeau, A. Mallat, 1992, The Journal of biological chemistry.

Nitric oxide regulates cardiac Ca2+ current. Involvement of cGMP-inhibited and cGMP-stimulated phosphodiesterases through guanylyl cyclase activation.

R Fischmeister, R. Fischmeister, P. Mery, 1993, The Journal of biological chemistry.

The inhibitor of liver plasma membrane (Ca2+-Mg2+)-ATPase. Purification and identification as a mediator of glucagon action.

A. Mallat, S. Lotersztajn, F. Pecker, 1985, The Journal of biological chemistry.

Cardiac inflammatory CD11b/c cells exert a protective role in hypertrophied cardiomyocyte by promoting TNFR2- and Orai3- dependent signaling

J. Hulot, M. Keck, F. Atassi, 2019, Scientific Reports.

Tumor necrosis factor alpha and glutathione interplay in chronic heart failure.

L. Hittinger, G. Candiani, M. Kirsch, 2005, Archives des maladies du coeur et des vaisseaux.

Cholera toxin blocks glucagon-mediated inhibition of the liver plasma membrane (Ca2+-Mg2+)-ATPase.

P. Insel, D. Stengel, A. Mallat, 1987, The Journal of biological chemistry.

Mode of Action of Glucagon Revisited

F. Pecker, C. Pavoine, 1996 .

The liver plasma membrane Ca2+ pump: hormonal sensitivity.

R. Epand, A. Mallat, S. Lotersztajn, 1985, Biochimie.

Protein kinase C and granule release in human platelets.

R. Brasseur, J. Ruysschaert, M. Castagna, 1985, Advances in experimental medicine and biology.

Pharmacological and molecular characterisation of beta-adrenoceptors in adult rat diaphragm muscle.

G. Atlan, F. Pecker, B. Fève, 1998, Respiration physiology.

Sphingomyelinases: their regulation and roles in cardiovascular pathophysiology.

F. Pecker, C. Pavoine, 2009, Cardiovascular Research.

Transcription of the sarcoplasmic/endoplasmic reticulum Ca2+-ATPase type 3 gene, ATP2A3, is regulated by the calcineurin/NFAT pathway in endothelial cells.

Catherine Pavoine, A. Lompré, Lahouaria Hadri, 2006, The Biochemical journal.

The cannabinoid receptor type 2 promotes cardiac myocyte and fibroblast survival and protects against ischemia/reperfusion‐induced cardiomyopathy

J. Wan, S. Lotersztajn, A. Zimmer, 2009, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Cardiac inflammatory CD11b/c cells exert a protective role in hypertrophied cardiomyocyte by promoting TNFR2- and Orai3- dependent signaling

J. Hulot, M. Keck, F. Atassi, 2019, Scientific Reports.

Emergence of Orai3 activity during cardiac hypertrophy.

J. Hulot, M. Keck, M. Trebak, 2015, Cardiovascular research.

The cardiac beta2-adrenergic signalling a new role for the cPLA2.

N. Defer, C. Pavoine, 2005, Cellular signalling.

5 – EHNA as an Inhibitor of PDE2: A Pharmacological and Biochemical Study in Cardiac Myocytes

R. Fischmeister, P. Mery, F. Pecker, 1996 .