I. Mihaylova

发表

DENTAL STUDENTS’ PERSPECTIVES ON THE IMPLEMENTATION AND EFFECTIVENESS OF ONLINE LEARNING DUE TO COVID-19 PANDEMIC: A DESCRIPTIVE CROSS-SECTIONAL STUDY IN BULGARIA

N. Avramova, I. Mihaylova, K. Yaneva-Ribagina, 2021, Advanced Education.

Not in the mind but in the cell: increased production of cyclo-oxygenase-2 and inducible NO synthase in chronic fatigue syndrome.

M. Maes, E. Bosmans, M. Kubera, 2007, Neuro endocrinology letters.

Increased plasma peroxides and serum oxidized low density lipoprotein antibodies in major depression: markers that further explain the higher incidence of neurodegeneration and coronary artery disease.

M. Maes, E. Bosmans, M. Kubera, 2010, Journal of affective disorders.

Increased serum IgA and IgM against LPS of enterobacteria in chronic fatigue syndrome (CFS): indication for the involvement of gram-negative enterobacteria in the etiology of CFS and for the presence of an increased gut-intestinal permeability.

M. Maes, J. Leunis, I. Mihaylova, 2007, Journal of affective disorders.

IgM-mediated autoimmune responses directed against anchorage epitopes are greater in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) than in major depression

M. Geffard, M. Kubera, Michael Maes, 2012, Metabolic Brain Disease.

IgM-mediated autoimmune responses directed against multiple neoepitopes in depression: new pathways that underpin the inflammatory and neuroprogressive pathophysiology.

M. Maes, M. Geffard, M. Kubera, 2011, Journal of affective disorders.

The immune effects of TRYCATs (tryptophan catabolites along the IDO pathway): relevance for depression - and other conditions characterized by tryptophan depletion induced by inflammation.

M. Maes, E. Bosmans, M. Kubera, 2007, Neuro endocrinology letters.

Decreased expression of CD69 in chronic fatigue syndrome in relation to inflammatory markers: evidence for a severe disorder in the early activation of T lymphocytes and natural killer cells.

E. Bosmans, M. Maes, I. Mihaylova, 2007, Neuro endocrinology letters.

Coenzyme Q10 deficiency in myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) is related to fatigue, autonomic and neurocognitive symptoms and is another risk factor explaining the early mortality in ME/CFS due to cardiovascular disorder.

M. Maes, E. Bosmans, M. Kubera, 2009, Neuro endocrinology letters.

Why fish oils may not always be adequate treatments for depression or other inflammatory illnesses: docosahexaenoic acid, an omega-3 polyunsaturated fatty acid, induces a Th-1-like immune response.

M. Maes, E. Bosmans, M. Kubera, 2007, Neuro endocrinology letters.

Increased serum IgM antibodies directed against phosphatidyl inositol (Pi) in chronic fatigue syndrome (CFS) and major depression: evidence that an IgM-mediated immune response against Pi is one factor underpinning the comorbidity between both CFS and depression.

M. Maes, J. Leunis, I. Mihaylova, 2007, Neuro endocrinology letters.

Lower serum zinc in Chronic Fatigue Syndrome (CFS): relationships to immune dysfunctions and relevance for the oxidative stress status in CFS.

M. Maes, I. Mihaylova, M. De Ruyter, 2006, Journal of affective disorders.

IgM-mediated autoimmune responses directed against anchorage epitopes are greater in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome (ME/CFS) than in major depression

M. Geffard, M. Kubera, Michael Maes, 2012, Metabolic Brain Disease.

Activation of cell-mediated immunity in depression: Association with inflammation, melancholia, clinical staging and the fatigue and somatic symptom cluster of depression

M. Kubera, Michael Maes, I. Mihaylova, 2012, Progress in Neuro-Psychopharmacology and Biological Psychiatry.

Decreased expression of CD 69 in chronic fatigue syndrome in relation to inflammatory markers : evidence for a severe disorder in the early activation of T lymphocytes and natural killer cells

E. Bosmans, M. Maes, I. Mihaylova, 2007 .

Chronic fatigue syndrome is accompanied by an IgM-related immune response directed against neopitopes formed by oxidative or nitrosative damage to lipids and proteins.

M. Maes, J. Leunis, I. Mihaylova, 2006, Neuro endocrinology letters.

Increased 8-hydroxy-deoxyguanosine, a marker of oxidative damage to DNA, in major depression and myalgic encephalomyelitis / chronic fatigue syndrome.

M. Maes, E. Bosmans, M. Kubera, 2009, Neuro endocrinology letters.

Lower whole blood glutathione peroxidase (GPX) activity in depression, but not in myalgic encephalomyelitis / chronic fatigue syndrome: another pathway that may be associated with coronary artery disease and neuroprogression in depression.

M. Maes, E. Bosmans, M. Kubera, 2011, Neuro endocrinology letters.

In chronic fatigue syndrome, the decreased levels of omega-3 poly-unsaturated fatty acids are related to lowered serum zinc and defects in T cell activation.

M. Maes, J. Leunis, I. Mihaylova, 2005, Neuro endocrinology letters.

In chronic fatigue syndrome , the decreased levels of omega-3 polyunsaturated fatty acids are related to lowered serum zinc and defects in T cell activation

M. Maes, J. Leunis, I. Mihaylova, 2005 .

Not in the mind of neurasthenic lazybones but in the cell nucleus: patients with chronic fatigue syndrome have increased production of nuclear factor kappa beta.

M. Maes, E. Bosmans, I. Mihaylova, 2007, Neuro endocrinology letters.

Lower plasma Coenzyme Q10 in depression: a marker for treatment resistance and chronic fatigue in depression and a risk factor to cardiovascular disorder in that illness.

M. Maes, E. Bosmans, M. Kubera, 2009, Neuro endocrinology letters.

Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: implications for the pathways to chronic depression and neuroprogression.

Michael Berk, Piotr Galecki, Michel Geffard, 2013, Journal of affective disorders.

Decreased dehydroepiandrosterone sulfate but normal insulin-like growth factor in chronic fatigue syndrome (CFS): relevance for the inflammatory response in CFS.

M. Maes, I. Mihaylova, M. De Ruyter, 2005, Neuro endocrinology letters.

Increased autoimmune responses against auto-epitopes modified by oxidative and nitrosative damage in depression: implications for the pathways to chronic depression and neuroprogression.

M. Maes, M. Geffard, M. Berk, 2013, Journal of affective disorders.