A. Doverspike

Catecholamine stimulation is associated with impaired myocardial O(2) utilization in heart failure.

R. Shannon, You-tang Shen, L. Nikolaidis, 2002, Cardiovascular research.

Angiotensin-Converting Enzyme Inhibitors Improve Coronary Flow Reserve in Dilated Cardiomyopathy by a Bradykinin-Mediated, Nitric Oxide-Dependent Mechanism

R. Shannon, L. Nikolaidis, T. Hentosz, 2002, Circulation.

Recombinant Glucagon-Like Peptide-1 Increases Myocardial Glucose Uptake and Improves Left Ventricular Performance in Conscious Dogs With Pacing-Induced Dilated Cardiomyopathy

R. Shannon, D. Elahi, You-tang Shen, 2004, Circulation.

Glucagon-Like Peptide-1 Limits Myocardial Stunning following Brief Coronary Occlusion and Reperfusion in Conscious Canines

R. Shannon, D. Elahi, You-tang Shen, 2005, Journal of Pharmacology and Experimental Therapeutics.

Mechanisms whereby rapid RV pacing causes LV dysfunction: perfusion-contraction matching and NO.

Y. Shen, R. Shannon, L. Nikolaidis, 2001, American journal of physiology. Heart and circulatory physiology.

Catecholamines restore myocardial contractility in dilated cardiomyopathy at the expense of increased coronary blood flow and myocardial oxygen consumption (MvO2 cost of catecholamines in heart failure)

R. Shannon, M. Mathier, You-tang Shen, 2004, European journal of heart failure.

Coronary blood flow responses are impaired independent of NO and endothelial function in conscious dogs with dilated cardiomyopathy.

R. Shannon, M. Mathier, L. Nikolaidis, 2005, Journal of cardiac failure.

Left Ventricular Performance in Conscious Dogs With Pacing-Induced Dilated Recombinant Glucagon-Like Peptide-1 Increases Myocardial Glucose Uptake and

R. Shannon, D. Elahi, L. Nikolaidis, 2004 .