B. Alvarez-Moya

发表

Oncogenic K-ras segregates at spatially distinct plasma membrane signaling platforms according to its phosphorylation status

I. Prior, N. Agell, F. Tebar, 2013, Journal of Cell Science.

Tumor Growth Phosphorylation at Ser-181 of Oncogenic KRAS Is Required for

G. Capellá, N. Agell, F. Vilardell, 2014 .

Phosphorylation at Ser-181 of oncogenic KRAS is required for tumor growth.

G. Capellá, N. Agell, F. Vilardell, 2014, Cancer research.

Continuous sensing of IFNα by hepatic endothelial cells shapes a vascular antimetastatic barrier

G. Sitia, A. Esposito, L. Guidotti, 2022, bioRxiv.

CaM interaction and Ser181 phosphorylation as new K-Ras signaling modulators

N. Agell, F. Tebar, C. Barceló, 2011, Small GTPases.

K-Ras4B phosphorylation at Ser181 is inhibited by calmodulin and modulates K-Ras activity and function

M. Drosten, O. Bachs, N. Agell, 2010, Oncogene.

A clathrin-dependent pathway leads to KRas signaling on late endosomes en route to lysosomes

M. Matsuda, O. Bachs, N. Agell, 2009, The Journal of cell biology.

Identification of Essential Interacting Elements in K-Ras/Calmodulin Binding and Its Role in K-Ras Localization*

O. Bachs, P. Villalonga, N. Agell, 2008, Journal of Biological Chemistry.