H. Dam

发表

UBE2O negatively regulates TRAF6-mediated NF-κB activation by inhibiting TRAF6 polyubiquitination

P. Dijke, Long Zhang, Xiaofei Zhang, 2013, Cell Research.

A novel function of the transforming domain of E1a: Repression of AP-1 activity

R. Offringa, P. Herrlich, S. Gebel, 1990, Cell.

The N-terminal transactivation domain of ATF2 is a target for the co-operative activation of the c-jun promoter by p300 and 12S E1A

M. Verlaan, A. Zantema, A. Eb, 1999, Oncogene.

YAP antagonizes innate antiviral immunity and is targeted for lysosomal degradation through IKKɛ-mediated phosphorylation

Bing Yang, Junling Jia, Fangfang Zhou, 2017, Nature Immunology.

The role of JNK, p38 and ERK MAP-kinases in insulin-induced Thr69 and Thr71-phosphorylation of transcription factor ATF2

D. Ouwens, J. Maassen, G. C. Zon, 2009 .

Transcription Factor 2 Kinase in Insulin-Induced Thr69 and Thr71 Phosphorylation of Activating The Role of c-Jun N-Terminal Kinase, p38, and Extracellular Signal-Regulated

D. Ouwens, J. Maassen, G. C. Zon, 2006 .

ATF‐2 is preferentially activated by stress‐activated protein kinases to mediate c‐jun induction in response to genotoxic agents.

I. Herr, P. Herrlich, P. Angel, 1995, The EMBO journal.

ULTRAVIOLET‐RADIATION INDUCED c‐jun GENE TRANSCRIPTION: TWO AP‐1 LIKE BINDING SITES MEDIATE THE RESPONSE

P. Herrlich, A. Eb, P. Angel, 1992, Photochemistry and photobiology.

14-3-3ζ turns TGF-β to the dark side.

P. Dijke, P. ten Dijke, H. Dam, 2015, Cancer cell.

Heterodimer formation of cJun and ATF‐2 is responsible for induction of c‐jun by the 243 amino acid adenovirus E1A protein.

P. Herrlich, A. Zantema, A. Eb, 1993, The EMBO journal.

Ionizing radiation-induced activation of ATF-2 phosphorylation and atf3 expression is dependent on ATM and NBS kinases

A. Eb, C. Terleth, J. Kool, 2003 .

Soluble endoglin regulates signaling by bone morphogenetic protein 9

M. V. Dinther, P. Dijke, M. Thorikay, 2016 .

Distinct roles of Jun : Fos and Jun : ATF dimers in oncogenesis

Hans van Dam, M. Castellazzi, Marc Castellazzi, 2001, Oncogene.

Fefana Publication Fefana Publication

R. Kok, H. Dam, Corbion Purac, 2015 .

The TGF-β/Smad pathway induces breast cancer cell invasion through the up-regulation of matrix metalloproteinase 2 and 9 in a spheroid invasion model system

P. Dijke, G. Pluijm, E. Pardali, 2011, Breast Cancer Research and Treatment.

DNA damage in transcribed genes induces apoptosis via the JNK pathway and the JNK-phosphatase MKP-1

A. Yasui, Mohamed Hamdi, F. Carlotti, 2005, Oncogene.

Induction of ATF3 by ionizing radiation is mediated via a signaling pathway that includes ATM, Nibrin1, stress-induced MAPkinases and ATF-2

Mohamed Hamdi, A. Eb, C. Terleth, 2003, Oncogene.

Specific interactions between Smad proteins and AP-1 components determine TGFβ-induced breast cancer cell invasion

U. Landegren, K. Miyazono, C. Heldin, 2013, Oncogene.

Control of endothelial sprouting by a Tel–CtBP complex

Kazuki Kobayashi, J. Peterson-Maduro, S. Schulte-Merker, 2010, Nature Cell Biology.

Transcription factor ATF3 partially transforms chick embryo fibroblasts by promoting growth factor-independent proliferation

Sandrine Perez, M. Castellazzi, E. Vial, 2001, Oncogene.

Chapter Aberrant Polycystin-1 Expression results in Modifi cation of AP-1 Activity , whereas Wnt Signaling remains unaffected 2

H. Clevers, M. Wetering, J. Calvet, 2006 .

Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation.

P. Herrlich, K. Kooistra, H. van Dam, 1998, Genes & development.

Fas-associated factor 1 antagonizes Wnt signaling by promoting β-catenin degradation.

P. Dijke, T. Laar, Fangfang Zhou, 2011 .

Both ATF-2 and c-Jun are phosphorylated by stress-activated protein kinases in response to UV irradiation.

I. Herr, P. Herrlich, H. van Dam, 1995, Immunobiology.