R. J. Rogers

Tumor Necrosis Factor-α Selectively InducesMnSOD Expression via Mitochondria-to-Nucleus Signaling, whereas Interleukin-1β Utilizes an Alternative Pathway*

H. Nick, J. Monnier, R. J. Rogers, 2001, The Journal of Biological Chemistry.

Multiplexed detection of ions and mRNA expression in single living cells.

Colin D. Medley, W. Tan, Hui Lin, 2007, The Analyst.

PIN*POINT analysis on the endogenous MnSOD promoter: specific demonstration of Sp1 binding in vivo.

H. Nick, R. J. Rogers, Ann L. Chokas, 2003, American journal of physiology. Cell physiology.

A TEAD1/p65 complex regulates the eutherian-conserved MnSOD intronic enhancer, eRNA transcription and the innate immune response.

H. Nick, Xiaolei Qiu, R. J. Rogers, 2014, Biochimica et biophysica acta.

In Vivo Architecture of the Manganese Superoxide Dismutase Promoter*

S. Chesrown, R. J. Rogers, J. K. Mellott, 1999, The Journal of Biological Chemistry.

Simultaneous monitoring of the expression of multiple genes inside of single breast carcinoma cells.

Weihong Tan, Timothy J. Drake, Colin D. Medley, 2005, Analytical chemistry.

Tumor necrosis factor-alpha selectively induces MnSOD expression via mitochondria-to-nucleus signaling, whereas interleukin-1beta utilizes an alternative pathway.

H. Nick, J. Monnier, R. J. Rogers, 2001, The Journal of biological chemistry.

Cytokine-inducible enhancer with promoter activity in both the rat and human manganese-superoxide dismutase genes.

H. Nick, S. Chesrown, J. Monnier, 2000, The Biochemical journal.