J. W. Ho

发表

Human neuronal uncoupling proteins 4 and 5 (UCP4 and UCP5): structural properties, regulation, and physiological role in protection against oxidative stress and mitochondrial dysfunction

K. Chan, S. Ho, P. Ho, 2012, Brain and behavior.

Mitochondrial UCP4 attenuates MPP+ - and dopamine-induced oxidative stress, mitochondrial depolarization, and ATP deficiency in neurons and is interlinked with UCP2 expression.

K. Chan, S. Ho, P. Ho, 2009, Free radical biology & medicine.

Title Uncoupling protein-4 ( UCP 4 ) increases ATP supply byinteracting with mitochondrial complex II in neuroblastoma cells

K. Chan, S. Ho, P. Ho, 2012 .

Uncoupling Protein-4 (UCP4) Increases ATP Supply by Interacting with Mitochondrial Complex II in Neuroblastoma Cells

K. Chan, S. Ho, P. Ho, 2012, PloS one.

Title Mitochondrial uncoupling protein-2 ( UCP 2 ) mediates leptinprotection against MPP + toxicity in neuronal cells

K. Chan, S. Ho, P. Ho, 2010 .

Mitochondrial Uncoupling Protein-2 (UCP2) Mediates Leptin Protection Against MPP+ Toxicity in Neuronal Cells

K. Chan, S. Ho, P. Ho, 2009, Neurotoxicity Research.

Transcriptional regulation of UCP4 by NF-kappaB and its role in mediating protection against MPP+ toxicity.

K. Chan, S. Ho, P. Ho, 2010, Free radical biology & medicine.

Methyl‐4‐phenylpyridinium ion modulates expression of mitochondrial uncoupling proteins 2, 4, and 5 in catecholaminergic (SK‐N‐SH) cells

S. Ho, P. Ho, D. Ramsden, 2005, Journal of neuroscience research.

LRRK2 R1441G mice are more liable to dopamine depletion and locomotor inactivity

S. Ho, Zhongjun Zhou, P. Ho, 2014, Annals of clinical and translational neurology.

UCP4 is a target effector of the NF-κB c-Rel prosurvival pathway against oxidative stress.

K. Chan, S. Ho, P. Ho, 2012, Free radical biology & medicine.

Assessment of Cellular Estrogenic Activity Based on Estrogen Receptor-Mediated Reduction of Soluble-Form Catechol-O-Methyltransferase (COMT) Expression in an ELISA-Based System

S. Ho, P. Ho, Song Lu, 2013, PloS one.

Mitochondrial UCP5 is neuroprotective by preserving mitochondrial membrane potential, ATP levels, and reducing oxidative stress in MPP+ and dopamine toxicity.

K. Chan, S. Ho, P. Ho, 2010, Free radical biology & medicine.

Plasma amyloid-β oligomers level is a biomarker for Alzheimer's disease diagnosis.

K. Lam, O. Cheng, J. Kwan, 2012, Biochemical and biophysical research communications.

Adiponectin is Protective against Oxidative Stress Induced Cytotoxicity in Amyloid-Beta Neurotoxicity

K. Chan, Sookja K. Chung, K. Lam, 2012, PloS one.

Estrogenic phenol and catechol metabolites of PCBs modulate catechol-O-methyltransferase expression via the estrogen receptor: potential contribution to cancer risk.

S. Ho, D. Ramsden, C. Garner, 2008, Current drug metabolism.

Clinical outcome of relapsing remitting multiple sclerosis among Hong Kong Chinese

K. Chan, S. Ho, P. Ho, 2011, Clinical Neurology and Neurosurgery.

Aquaporin-4 autoantibodies cause asymptomatic aquaporin-4 loss and activate astrocytes in mouse

J. Kwan, R. Zhang, J. W. Ho, 2012, Journal of Neuroimmunology.

Brain involvement in neuromyelitis optica spectrum disorders.

K. Chan, J. Kwan, P. Ho, 2011, Archives of neurology.

Aquaporin-4 water channel expression by thymoma of patients with and without myasthenia gravis

S. Ho, W. Chui, J. Kwan, 2010, Journal of Neuroimmunology.

Mitochondrial Uncoupling Protein-2 (UCP2) Mediates Leptin Protection Against MPP+ Toxicity in Neuronal Cells

K. Chan, S. Ho, P. Ho, 2009, Neurotoxicity Research.