David L. Hoffman

发表

Chapter 10 The Interaction of Mitochondrial Membranes with Reactive Oxygen and Nitrogen Species

A. Wojtovich, S. Nadtochiy, P. Brookes, 2008 .

The permeability transition pore controls cardiac mitochondrial maturation and myocyte differentiation.

K. L. de Mesy Bentley, G. Porter, J. Molkentin, 2011, Developmental cell.

Aging and Cardiac Ischemia—Mitochondria and Free Radical Considerations

P. Brookes, David L. Hoffman, 2008 .

HUMMR, a hypoxia- and HIF-1α–inducible protein, alters mitochondrial distribution and transport

H. Federoff, P. Aspenstrom, D. Rempe, 2009, The Journal of cell biology.

The permeability transition pore controls cardiac mitochondrial maturation and myocyte differentiation.

K. L. de Mesy Bentley, G. Porter, J. Molkentin, 2011, Developmental cell.

Mitochondrial Dysfunction in Cancer Cells Due to Aberrant Mitochondrial Replication*

K. L. de Mesy Bentley, David L. Hoffman, R. Eliseev, 2011, The Journal of Biological Chemistry.

Oxygen Sensitivity of Mitochondrial Reactive Oxygen Species Generation Depends on Metabolic Conditions

P. Brookes, David L Hoffman, Paul S Brookes, 2009, The Journal of Biological Chemistry.

Bioenergetics, mitochondria, and cardiac myocyte differentiation.

K. L. de Mesy Bentley, G. Porter, S. Sheu, 2011, Progress in pediatric cardiology.

Response of mitochondrial reactive oxygen species generation to steady-state oxygen tension: implications for hypoxic cell signaling.

P. Brookes, David L. Hoffman, J. Salter, 2007, American journal of physiology. Heart and circulatory physiology.

HUMMR, a hypoxia- and HIF-1α–inducible protein, alters mitochondrial distribution and transport

H. Federoff, P. Aspenstrom, D. Rempe, 2009, The Journal of cell biology.