N. Roeser

发表

Alleviation of fatty acid and hypoxia-reoxygenation-induced proximal tubule deenergization by ADP/ATP carrier inhibition and glutamate.

A. Kribben, J. Weinberg, T. Feldkamp, 2007, American journal of physiology. Renal physiology.

Quantitative assessment of neuroprotection against NMDA-induced brain injury

M. Johnston, J. Mcdonald, F. Silverstein, 1989, Experimental Neurology.

Substrate Modulation of Fatty Acid Effects on Energization and Respiration of Kidney Proximal Tubules during Hypoxia/Reoxygenation

A. Kribben, J. Weinberg, T. Feldkamp, 2014, PloS one.

Mitochondrial dysfunction during hypoxia/reoxygenation and its correction by anaerobic metabolism of citric acid cycle intermediates.

J. Weinberg, M. Venkatachalam, I. Nissim, 2000, Proceedings of the National Academy of Sciences of the United States of America.

proximal tubules hypoxia-reoxygenation-induced mitochondrial injury in Preservation of complex I function during

A. Kribben, J. Weinberg, T. Feldkamp, 2016 .

Preservation of complex I function during hypoxia-reoxygenation-induced mitochondrial injury in proximal tubules.

A. Kribben, J. Weinberg, T. Feldkamp, 2004, American journal of physiology. Renal physiology.

Glycine-protected, hypoxic, proximal tubules develop severely compromised energetic function.

J. Weinberg, M. Venkatachalam, J. Davis, 1997, Kidney international.

Cyclophilin D and the mitochondrial permeability transition in kidney proximal tubules after hypoxic and ischemic injury.

J. Weinberg, T. Feldkamp, N. Roeser, 2011, American journal of physiology. Renal physiology.

Regulation of the mitochondrial permeability transition in kidney proximal tubules and its alteration during hypoxia-reoxygenation.

J. Weinberg, T. Feldkamp, M. Venkatachalam, 2009, American journal of physiology. Renal physiology.

Mitochondrial dysfunction induced by pancreatic and crotalic (Crotalus durissus terrificus) phospholipases A2 on rabbit proximal tubules suspensions.

J. Weinberg, H. Monteiro, N. Roeser, 2008, Toxicon : official journal of the International Society on Toxinology.

Accumulation of nonesterified fatty acids causes the sustained energetic deficit in kidney proximal tubules after hypoxia-reoxygenation.

A. Kribben, J. Weinberg, T. Feldkamp, 2006, American journal of physiology. Renal physiology.

Energetic determinants of tyrosine phosphorylation of focal adhesion proteins during hypoxia/reoxygenation of kidney proximal tubules.

J. Weinberg, M. Venkatachalam, I. Nissim, 2001, The American journal of pathology.

Relationships between intracellular amino acid levels and protection against injury to isolated proximal tubules.

J. Weinberg, I. Nissim, S. Schultz, 1991, The American journal of physiology.

Differential cytoprotection by glycine against oxidant damage to proximal tubule cells.

J. Weinberg, M. Venkatachalam, N. Roeser, 1996, Kidney international.

Calcium dependence of integrity of the actin cytoskeleton of proximal tubule cell microvilli.

J. Weinberg, S. Nurko, M. Venkatachalam, 1996, The American journal of physiology.

Contribution of actin cytoskeletal alterations to ATP depletion and calcium-induced proximal tubule cell injury.

B. Athey, D. Hinshaw, J. Weinberg, 1996, The American journal of physiology.

Modulation by Gly, Ca, and acidosis of injury-associated unesterified fatty acid accumulation in proximal tubule cells.

J. Weinberg, H. Goldberg, M. Venkatachalam, 1995, The American journal of physiology.

Role of intracellular pH during cytoprotection of proximal tubule cells by glycine or acidosis.

J. Weinberg, M. Venkatachalam, J. Davis, 1994, Journal of the American Society of Nephrology : JASN.

Protection of human umbilical vein endothelial cells by glycine and structurally similar amino acids against calcium and hydrogen peroxide-induced lethal cell injury.

J. Varani, J. Weinberg, K. Johnson, 1992, The American journal of pathology.

Amino acid protection of cultured kidney tubule cells against calcium ionophore-induced lethal cell injury.

J. Varani, J. Weinberg, K. Johnson, 1991, Laboratory investigation; a journal of technical methods and pathology.

Role of increased cytosolic free calcium in the pathogenesis of rabbit proximal tubule cell injury and protection by glycine or acidosis.

J. Weinberg, M. Venkatachalam, J. Davis, 1991, The Journal of clinical investigation.

Structural requirements for protection by small amino acids against hypoxic injury in kidney proximal tubules 1

J. Weinberg, M. Venkatachalam, J. Davis, 1990, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Acetyl Co-A Carboxylase Inhibition Halts Hyperglycemia Induced Upregulation of De Novo Lipogenesis in Podocytes and Proximal Tubular Cells

Pradeep P. Kayampilly, T. Rajendiran, S. Pennathur, 2022, Metabolites.

Anaerobic and aerobic pathways for salvage of proximal tubules from hypoxia-induced mitochondrial injury.

J. Weinberg, M. Venkatachalam, I. Nissim, 2000, American journal of physiology. Renal physiology.

Cyclophilin D and the mitochondrial permeability transition in kidney proximal tubules after hypoxic and ischemic injury.

J. Weinberg, T. Feldkamp, N. Roeser, 2011, American journal of physiology. Renal physiology.