Jinjun Liu

发表

Pyridostigmine Ameliorates Cardiac Remodeling Induced by Myocardial Infarction via Inhibition of the Transforming Growth Factor-&bgr;1/TGF-&bgr;1–Activated Kinase Pathway

W. Zang, Xiao-Jiang Yu, Jun Zhou, 2014, Journal of cardiovascular pharmacology.

Exercise improves the dilatation function of mesenteric arteries in postmyocardial infarction rats via a PI3K/Akt/eNOS pathway-mediated mechanism.

W. Wier, Xiao-Jiang Yu, Jing Yang, 2010, American journal of physiology. Heart and circulatory physiology.

Acetylcholine prevents angiotensin II-induced oxidative stress and apoptosis in H9c2 cells

You Wang, Juan Zhou, W. Zang, 2010, Apoptosis.

Pyridostigmine protects against cardiomyopathy associated with adipose tissue browning and improvement of vagal activity in high-fat diet rats.

W. Zang, Xiao-Jiang Yu, Yi Lu, 2018, Biochimica et biophysica acta. Molecular basis of disease.

PVN Blockade of p44/42 MAPK Pathway Attenuates Salt-induced Hypertension through Modulating Neurotransmitters and Attenuating Oxidative Stress

Yu-Ming Kang, Xiao-Jing Yu, Kai-Li Liu, 2017, Scientific Reports.

Improving vagal activity ameliorates cardiac fibrosis induced by angiotensin II: in vivo and in vitro

W. Zang, Xiao-Jiang Yu, Mei Zhao, 2015, Scientific Reports.

Paraventricular Nucleus Infusion of Epigallocatechin-3-O-Gallate Improves Renovascular Hypertension

Yu-Ming Kang, Q. Su, Xiao-Jing Yu, 2016, Cardiovascular Toxicology.

Acetylcholine Mediates AMPK-Dependent Autophagic Cytoprotection in H9c2 Cells During Hypoxia/Reoxygenation Injury

Hao Wang, Jun Ren, W. Zang, 2013, Cellular Physiology and Biochemistry.

Vagal nerve modulation: A promising new therapeutic approach for cardiovascular diseases

Hao Wang, W. Zang, Lei Sun, 2012, Clinical and experimental pharmacology & physiology.

Specific α7 nicotinic acetylcholine receptor agonist ameliorates isoproterenol‐induced cardiac remodelling in mice through TGF‐β1/Smad3 pathway

W. Zang, Xiao-Jiang Yu, Lei Sun, 2017, Clinical and experimental pharmacology & physiology.

ANGIOTENSIN II RECEPTORS SUBTYPES MEDIATE DIVERSE GENE EXPRESSION PROFILE IN ADULT HYPERTROPHIC CARDIOMYOCYTES

Juan Zhou, Yuan-xi Lin, Jinjun Liu, 2007, Clinical and experimental pharmacology & physiology.

Optimizing the Parameters of Vagus Nerve Stimulation by Uniform Design in Rats with Acute Myocardial Infarction

Bing Wang, Ming Zhao, Yu-Ming Kang, 2012, PloS one.

Na+/K+-ATPase Alpha 2 Isoform Elicits Rac1-Dependent Oxidative Stress and TLR4-Induced Inflammation in the Hypothalamic Paraventricular Nucleus in High Salt-Induced Hypertension

Yu-Ming Kang, Q. Su, Xiao-Jing Yu, 2022, Antioxidants.

TLR4/MyD88/NF-κB signaling and PPAR-γ within the paraventricular nucleus are involved in the effects of telmisartan in hypertension.

Yu-Ming Kang, Q. Su, Hong‐Bao Li, 2016, Toxicology and applied pharmacology.

Exercise training attenuates renovascular hypertension partly via RAS- ROS- glutamate pathway in the hypothalamic paraventricular nucleus

Yu-Ming Kang, Kai-Li Liu, Wensheng Chen, 2016, Scientific Reports.

NF-κB Blockade in Hypothalamic Paraventricular Nucleus Inhibits High-Salt-Induced Hypertension Through NLRP3 and Caspase-1

Yu-Ming Kang, Q. Yi, Jie Qi, 2016, Cardiovascular Toxicology.

Protection against Ischemia-Induced Oxidative Stress Conferred by Vagal Stimulation in the Rat Heart: Involvement of the AMPK-PKC Pathway

W. Zang, Xiao-Jiang Yu, Yi Lu, 2012, International journal of molecular sciences.

Irisin lowers blood pressure by activating the Nrf2 signaling pathway in the hypothalamic paraventricular nucleus of spontaneously hypertensive rats.

Yu-Ming Kang, Q. Su, Kai-Li Liu, 2020, Toxicology and applied pharmacology.

Acetylcholine Attenuates Hypoxia/ Reoxygenation-Induced Mitochondrial and Cytosolic ROS Formation in H9c2 Cells via M2 Acetylcholine Receptor

Xiao-Jiang Yu, Lei Sun, Mei Zhao, 2013, Cellular Physiology and Biochemistry.

Alpha lipoic acid supplementation attenuates reactive oxygen species in hypothalamic paraventricular nucleus and sympathoexcitation in high salt-induced hypertension.

Yu-Ming Kang, Q. Su, Qing Yang, 2016, Toxicology letters.

Salusin β Within the Nucleus Tractus Solitarii Suppresses Blood Pressure Via Inhibiting the Activities of Presympathetic Neurons in the Rostral Ventrolateral Medulla in Spontaneously Hypertensive Rats

Q. Su, G. Zhu, Jie Qi, 2015, Cardiovascular Toxicology.

Low‐dose celecoxib improves coronary function after acute myocardial ischaemia in rabbits

W. Zang, Xiao-Jiang Yu, Mei Zhao, 2012, Clinical and experimental pharmacology & physiology.

Acetylcholine Inhibits LPS-Induced MMP-9 Production and Cell Migration via the a7 nAChR-JAK2/STAT3 Pathway in RAW264.7 Cells

W. Zang, Xiao-Jiang Yu, Lei Sun, 2015, Cellular Physiology and Biochemistry.

Central Blockade of E-Prostanoid 3 Receptor Ameliorated Hypertension Partially by Attenuating Oxidative Stress and Inflammation in the Hypothalamic Paraventricular Nucleus of Spontaneously Hypertensive Rats

Yu-Ming Kang, Q. Su, Kai-Li Liu, 2020, Cardiovascular Toxicology.

Oral CoQ10 attenuates high salt-induced hypertension by restoring neurotransmitters and cytokines in the hypothalamic paraventricular nucleus

J. Mu, Yu-Ming Kang, Kai-Li Liu, 2016, Scientific Reports.

Tert-butylhydroquinone attenuates oxidative stress and inflammation in hypothalamic paraventricular nucleus in high salt-induced hypertension.

Yu-Ming Kang, Kai-Li Liu, Wensheng Chen, 2017, Toxicology letters.

Renin-angiotensin system acting on reactive oxygen species in paraventricular nucleus induces sympathetic activation via AT1R/PKCγ/Rac1 pathway in salt-induced hypertension

Yu-Ming Kang, Q. Su, Qing Yang, 2017, Scientific Reports.

The effect of chronic tBHQ treatment on high salt-induced hypertension is reported • Chronic tBHQ supplementation attenuates hypertension and sympathoexcitation • Chronic tBHQ supplementation attenuates PVN oxidative stress and inflammation

Yu-Ming Kang, Kai-Li Liu, Wensheng Chen, 2017 .

Chronic Infusion of Astaxanthin Into Hypothalamic Paraventricular Nucleus Modulates Cytokines and Attenuates the Renin-Angiotensin System in Spontaneously Hypertensive Rats.

Yu-Ming Kang, Xiao-Min Wang, Kai-Li Liu, 2021, Journal of cardiovascular pharmacology.

NF-κB Blockade in Hypothalamic Paraventricular Nucleus Inhibits High-Salt-Induced Hypertension Through NLRP3 and Caspase-1

Q. Yi, Jie Qi, Jinjun Liu, 2015, Cardiovascular Toxicology.

Pyridostigmine prevents peripheral vascular endothelial dysfunction in rats with myocardial infarction

Xiao-Jiang Yu, Xi He, Yi Lu, 2014, Clinical and experimental pharmacology & physiology.

Tumour necrosis factor‐α and its receptors in the beneficial effects of vagal stimulation after myocardial infarction in rats

W. Zang, Xiao-Jiang Yu, Yi Lu, 2011, Clinical and experimental pharmacology & physiology.

Acetylcholine inhibits hypoxia‐induced tumor necrosis factor‐α production via regulation of MAPKs phosphorylation in cardiomyocytes

Jun Ren, W. Zang, Xiao-Jiang Yu, 2011, Journal of cellular physiology.

Apocynin attenuates pressure overload-induced cardiac hypertrophy in rats by reducing levels of reactive oxygen species.

Juan Zhou, Yuan-xi Lin, Jinjun Liu, 2010, Canadian journal of physiology and pharmacology.

Yu-Ming Kang, Q. Su, G. Zhu, 2016, Cardiovascular Toxicology.

Paraventricular Nucleus Infusion of Epigallocatechin-3-O-Gallate Improves Renovascular Hypertension

Yu-Ming Kang, Q. Su, Q. Yi, 2016, Cardiovascular Toxicology.

Acetylcholine Inhibits Tumor Necrosis Factor α Activated Endoplasmic Reticulum Apoptotic Pathway via EGFR‐PI3K Signaling in Cardiomyocytes

W. Zang, Xiao-Jiang Yu, Mei Zhao, 2015, Journal of cellular physiology.

Pyridostigmine attenuates hypertension by inhibiting activation of the renin-angiotensin system in the hypothalamic paraventricular nucleus.

Jinjun Liu, Yi Lu, Yi-Dong Wang, 2024, Naunyn-Schmiedeberg's archives of pharmacology.

Long‐term administration of pyridostigmine attenuates pressure overload‐induced cardiac hypertrophy by inhibiting calcineurin signalling

W. Zang, Xiao-Jiang Yu, Lei Sun, 2017, Journal of cellular and molecular medicine.