T. Bilousova

发表

Transgenic mice with enhanced neuronal major histocompatibility complex class I expression recover locomotor function better after spinal cord injury

V. Edgerton, H. Zhong, S. Zdunowski, 2011, Journal of neuroscience research.

Levels of Soluble Apolipoprotein E/Amyloid-β (Aβ) Complex Are Reduced and Oligomeric Aβ Increased with APOE4 and Alzheimer Disease in a Transgenic Mouse Model and Human Samples*♦

H. Vinters, D. Fardo, S. Estus, 2013, The Journal of Biological Chemistry.

Neuronal pentraxin 1: A synaptic-derived plasma biomarker in Alzheimer's disease

E. Teng, L. Apostolova, B. Teter, 2018, Neurobiology of Disease.

Evaluation of an Allosteric BACE Inhibitor Peptide to Identify Mimetics that Can Interact with the Loop F Region of the Enzyme and Prevent APP Cleavage.

K. Vadivel, V. John, T. Bilousova, 2018, Journal of molecular biology.

Integrated Multiomics Analysis of Salivary Exosomes to Identify Biomarkers Associated with Changes in Mood States and Fatigue

B. Teter, G. Cole, D. Elashoff, 2022, International journal of molecular sciences.

Minocycline promotes dendritic spine maturation and improves behavioural performance in the fragile X mouse model

Michelle N. Ngo, D. Ethell, I. Ethell, 2008, Journal of Medical Genetics.

A Small Molecule Mimetic of the Humanin Peptide as a Candidate for Modulating NMDA-Induced Neurotoxicity.

K. Vadivel, D. Evseenko, V. John, 2017, ACS chemical neuroscience.

Pharmacological Rescue of Long-Term Potentiation in Alzheimer Diseased Synapses

G. Lynch, C. Cotman, N. Berchtold, 2017, The Journal of Neuroscience.

Parallel age-associated changes in brain and plasma neuronal pentraxin receptor levels in a transgenic APP/PS1 rat model of Alzheimer's disease

E. Teng, G. Cole, S. Frautschy, 2015, Neurobiology of Disease.

Multi-Omics Analysis of Microglial Extracellular Vesicles From Human Alzheimer’s Disease Brain Tissue Reveals Disease-Associated Signatures

B. Teter, Whitaker Cohn, Chunni Zhu, 2021, Frontiers in Pharmacology.

A small molecule ApoE4-targeted therapeutic candidate that normalizes sirtuin 1 levels and improves cognition in an Alzheimer’s disease mouse model

G. Cole, D. Bredesen, Johnny D. Pham, 2018, Scientific Reports.

A rose by any other name? the potential consequences of microglial heterogeneity during CNS health and disease

B. Melchior, Jonathan M. Doose, M. Carson, 2007, Neurotherapeutics.

Dual Neutral Sphingomyelinase-2/Acetylcholinesterase Inhibitors for the treatment of Alzheimer's disease.

K. Vadivel, V. John, Whitaker Cohn, 2020, ACS chemical biology.

Major histocompatibility complex class I molecules modulate embryonic neuritogenesis and neuronal polarization

Yi-Ping Jin, Blake Middleton, Jide Tian, 2012, Journal of Neuroimmunology.

Synaptic Amyloid-β Oligomers Precede p-Tau and Differentiate High Pathology Control Cases.

E. Teng, H. Vinters, C. Kawas, 2016, The American journal of pathology.

Apolipoprotein E level and cholesterol are associated with reduced synaptic amyloid beta in Alzheimer’s disease and apoE TR mouse cortex

E. Teng, H. Vinters, G. Cole, 2011, Acta Neuropathologica.

AD synapses contain abundant Aβ monomer and multiple soluble oligomers, including a 56-kDa assembly

H. Vinters, G. Cole, C. Miller, 2012, Neurobiology of Aging.

Pre‐synaptic C‐terminal truncated tau is released from cortical synapses in Alzheimer's disease

H. Vinters, C. Miller, W. Poon, 2015, Journal of neurochemistry.

Exosomal tau with seeding activity is released from Alzheimer’s disease synapses, and seeding potential is associated with amyloid beta

H. Vinters, C. Kawas, M. Corrada, 2021, Laboratory Investigation.

Dietary DHA supplementation in an APP/PS1 transgenic rat model of AD reduces behavioral and Aβ pathology and modulates Aβ oligomerization

E. Teng, G. Cole, S. Frautschy, 2015, Neurobiology of Disease.

Neurons Preferentially Respond to Self-MHC Class I Allele Products Regardless of Peptide Presented

C. Evans, Y. Yoshimura, Yuxin Lu, 2009, The Journal of Immunology.

Nanoscale Extracellular Vesicle Analysis in Alzheimer's Disease Diagnosis and Therapy

P. Heinzelman, V. John, T. Bilousova, 2016, International journal of Alzheimer's disease.

Flow Cytometry Analysis and Quantitative Characterization of Tau in Synaptosomes from Alzheimer's Disease Brains.

K. Gylys, T. Bilousova, Tina Bilousova, 2017, Methods in molecular biology.

Suppression of tau propagation using an inhibitor that targets the DK-switch of nSMase2.

K. Vadivel, Emily Miyoshi, V. John, 2018, Biochemical and biophysical research communications.

PresentedClass I Allele Products Regardless of Peptide Neurons Preferentially Respond to Self-MHC

C. Evans, Y. Yoshimura, Blake Middleton, 2009 .

Apolipoprotein E/Amyloid-β Complex Accumulates in Alzheimer Disease Cortical Synapses via Apolipoprotein E Receptors and Is Enhanced by APOE4.

H. Vinters, C. Kawas, M. Corrada, 2019, The American journal of pathology.

Pharmacological inhibition of nSMase2 reduces brain exosome release and α-synuclein pathology in a Parkinson’s disease model

A. Hatami, V. John, Whitaker Cohn, 2021, Molecular Brain.

Loss of endophilin-B1 exacerbates Alzheimer's disease pathology.

E. Cudaback, C. Keene, S. Jayadev, 2015, Brain : a journal of neurology.

Matrix metalloproteinase‐7 disrupts dendritic spines in hippocampal neurons through NMDA receptor activation

D. Rusakov, D. Ethell, I. Ethell, 2006, Journal of neurochemistry.

Major histocompatibility complex class I-mediated inhibition of neurite outgrowth from peripheral nerves.

Jide Tian, D. Kaufman, H. Dang, 2011, Immunology letters.

Enhanced neuronal expression of major histocompatibility complex class I leads to aberrations in neurodevelopment and neurorepair

Jide Tian, D. Kaufman, H. Dang, 2011, Journal of Neuroimmunology.