B. L. Roman

Responsiveness of the adult male rat reproductive tract to 2,3,7,8-tetrachlorodibenzo-p-dioxin exposure: Ah receptor and ARNT expression, CYP1A1 induction, and Ah receptor down-regulation.

R. Peterson, R. Pollenz, B. L. Roman, 1998, Toxicology and applied pharmacology.

In utero and lactational exposure of the male rat to 2,3,7,8-tetrachlorodibenzo-p-dioxin impairs prostate development. 1. Effects on gene expression.

R. Peterson, B. L. Roman, 1998, Toxicology and applied pharmacology.

2,3,7,8-tetrachlorodibenzo-p-dioxin inhibits luminal cell differentiation and androgen responsiveness of the ventral prostate without inhibiting prostatic 5alpha-dihydrotestosterone formation or testicular androgen production in rat offspring.

R. Peterson, B. L. Roman, H. Theobald, 2000, Toxicological sciences : an official journal of the Society of Toxicology.

In Uteroand Lactational Exposure of the Male Rat to 2,3,7,8-Tetrachlorodibenzo-p-dioxin Impairs Prostate Development☆

R. Peterson, B. L. Roman, 1998 .

In utero and lactational exposure of the male rat to 2,3,7,8-tetrachlorodibenzo-p-dioxin impairs prostate development. 2. Effects on growth and cytodifferentiation.

G. Prins, R. Peterson, B. Timms, 1998, Toxicology and applied pharmacology.

In utero and lactational exposure of the male rat to 2,3,7,8-tetrachlorodibenzo-p-dioxin: impaired prostate growth and development without inhibited androgen production.

R. Peterson, B. L. Roman, R. Sommer, 1995, Toxicology and applied pharmacology.