Federica Cuozzo

发表

Vitamin-D-Binding Protein Contributes to the Maintenance of α Cell Function and Glucagon Secretion

P. MacDonald, N. Morgan, S. Richardson, 2020, Cell reports.

The prolyl hydroxylase PHD 3 maintains β-cell glucose metabolism during fatty 3 acid excess 4 5

Ildem Akerman, D. Tennant, D. Hodson, 2020 .

The prolyl hydroxylase PHD3 maintains β-cell glucose metabolism during fatty acid excess

Ildem Akerman, D. Tennant, D. Hodson, 2020, bioRxiv.

Isoform-specific Roles of Prolyl Hydroxylases in the Regulation of Pancreatic β-Cell Function

S. Janssen, D. Hodson, G. Fong, 2021, Endocrinology.

Mature and immature β-cells both contribute to islet function and insulin release

G. Rutter, Ildem Akerman, L. Piemonti, 2020 .

Oncogenic IDH1 Mutations Promote Enhanced Proline Synthesis through PYCR1 to Support the Maintenance of Mitochondrial Redox Homeostasis

C. Ludwig, I. Tomlinson, Y. Mao, 2018, Cell reports.

PDX1LOW MAFALOW β-cells contribute to islet function and insulin release

G. Rutter, L. Piemonti, D. Hodson, 2021, Nature Communications.

Prolyl-4-hydroxylase 3 maintains β cell glucose metabolism during fatty acid excess in mice

L. Hodson, D. Tennant, D. Hodson, 2021, JCI insight.

Prolyl-4-hydroxylase 3 maintains β-cell glucose metabolism during fatty acid 1 excess in mice 2 3

Leanne, Hodson, D. Tennant, 2021 .

Proline synthesis through PYCR1 is required to support cancer cell proliferation and survival in oxygen-limiting conditions

E. Bridges, K. Eales, D. Tennant, 2021, Cell reports.

GC-globulin/vitamin D-binding Protein is Required for Pancreatic α Cell 2 Adaptation to Metabolic Stress.

D. Hodson, M. Hewison, S. Heising, 2022, Diabetes.

Author Correction: PDX1LOW MAFALOW β-cells contribute to islet function and insulin release

G. Rutter, L. Piemonti, D. Hodson, 2021, Nature Communications.

Revealing the tissue-level complexity of endogenous glucagon-like peptide-1 receptor expression and signaling

D. Owen, D. Hodson, D. Nieves, 2023, Nature Communications.

High-resolution glucose fate-mapping reveals LDHB-dependent lactate production by human pancreatic β cells

C. Ludwig, F. Pattou, C. Bonner, 2022, bioRxiv.

A WFS1 variant disrupting acceptor splice site uncovers the impact of alternative splicing on ER-stress independent β cell apoptosis in a patient with Wolfram syndrome

V. Sordi, Federica Cuozzo, G. Frontino, 2023, bioRxiv.

LDHB contributes to the regulation of lactate levels and basal insulin secretion in human pancreatic β cells

J. Kerr-Conte, Daniela Nasteska, Katrina Viloria, 2024, Cell reports.

High-resolution glucose tracing and in situ imaging reveals regulated lactate production in human pancreatic β cells

C. Ludwig, F. Pattou, C. Bonner, 2023, bioRxiv.