S. Frank

发表

Keratinocyte-derived chemotactic cytokines: expressional modulation by nitric oxide in vitro and during cutaneous wound repair in vivo.

J. Pfeilschifter, C. Wetzler, H. Kämpfer, 2000, Biochemical and biophysical research communications.

Targeting Connexin43 Expression Accelerates the Rate of Wound Repair

C. Green, Paul Martin, L. Law, 2003, Current Biology.

Lipoteichoic acid induces delayed protection in the rat heart: A comparison with endotoxin.

S. Cuzzocrea, C. Thiemermann, J. Pfeilschifter, 2000, Arteriosclerosis, thrombosis, and vascular biology.

Nitric oxide drives skin repair: novel functions of an established mediator.

J. Pfeilschifter, C. Wetzler, H. Kämpfer, 2002, Kidney international.

Expression and Release of IL-18 Binding Protein in Response to IFN-γ1

H. Mühl, J. Pfeilschifter, S. Zeuzem, 2001, The Journal of Immunology.

Differential regulation of vascular endothelial growth factor and its receptor fms-like-tyrosine kinase is mediated by nitric oxide in rat renal mesangial cells.

J. Pfeilschifter, B. Stallmeyer, H. Kämpfer, 1999, The Biochemical journal.

Inhibition of proteolysis protects hippocampal neurons from ischemia.

G. Lynch, A. Arai, P. Vanderklish, 1991, Proceedings of the National Academy of Sciences of the United States of America.

Expression of interleukin-8, heme oxygenase-1 and vascular endothelial growth factor in DLD-1 colon carcinoma cells exposed to pyrrolidine dithiocarbamate.

H. Mühl, J. Pfeilschifter, M. Nold, 2002, Carcinogenesis.

Systemic anti-TNFalpha treatment restores diabetes-impaired skin repair in ob/ob mice by inactivation of macrophages.

H. Mühl, J. Pfeilschifter, U. Christen, 2007, The Journal of investigative dermatology.

Severely impaired insulin signaling in chronic wounds of diabetic ob/ob mice: a potential role of tumor necrosis factor-alpha.

J. Pfeilschifter, I. Goren, S. Frank, 2006, The American journal of pathology.

The suppressor of cytokine signaling-3 is upregulated in impaired skin repair: implications for keratinocyte proliferation.

J. Pfeilschifter, I. Goren, S. Frank, 2006, The Journal of investigative dermatology.

A novel type of glutathione peroxidase: expression and regulation during wound repair.

S. Werner, B. Munz, S. Frank, 1997, The Biochemical journal.

The human homologue of a bovine non-selenium glutathione peroxidase is a novel keratinocyte growth factor-regulated gene

S. Werner, B. Munz, S. Frank, 1997, Oncogene.

Transforming growth factors beta1, beta2, and beta3 and their receptors are differentially regulated during normal and impaired wound healing.

S. Werner, S. Frank, M. Madlener, 1996, The Journal of biological chemistry.

Nitric oxide triggers enhanced induction of vascular endothelial growth factor expression in cultured keratinocytes (HaCaT) and during cutaneous wound repair

J. Pfeilschifter, B. Stallmeyer, H. Kämpfer, 1999, FASEB journal : official publication of the Federation of American Societies for Experimental Biology.

Expression and activity of arginase isoenzymes during normal and diabetes-impaired skin repair.

J. Pfeilschifter, H. Kämpfer, S. Frank, 2003, The Journal of investigative dermatology.

A novel keratinocyte mitogen: regulation of leptin and its functional receptor in skin repair.

J. Pfeilschifter, R. Kaufmann, B. Stallmeyer, 2001, The Journal of investigative dermatology.

Counterregulation of interleukin-18 mRNA and protein expression during cutaneous wound repair in mice.

H. Mühl, J. Pfeilschifter, U. Kalina, 1999, The Journal of investigative dermatology.

REVIEW INDUCIBLE NO SYNTHASE: ROLE IN CELLULAR SIGNALLING

H. Mühl, J. Pfeilschifter, A. Huwiler, 1999 .

Interferon-gamma mediates gene expression of IL-18 binding protein in nonleukocytic cells.

H. Mühl, J. Pfeilschifter, H. Radeke, 2000, Biochemical and biophysical research communications.

Expression and release of chemokines associated with apoptotic cell death in human promonocytic U937 cells and peripheral blood mononuclear cells

H. Mühl, J. Pfeilschifter, W. Eberhardt, 1999, European journal of immunology.

The Human Homologue of the Yeast CHL1 Gene Is a Novel Keratinocyte Growth Factor-regulated Gene*

S. Werner, S. Frank, 1996, The Journal of Biological Chemistry.

[Function of keratinocyte growth factor in wound healing].

S. Werner, M. Brauchle, H. Smola, 1996, Zentralblatt fur Chirurgie.

Lack of Interferon-γ Production Despite the Presence of Interleukin-18 During Cutaneous Wound Healing

H. Mühl, J. Pfeilschifter, J. Paulukat, 2000, Molecular medicine.

Cyclooxygenase-1-coupled prostaglandin biosynthesis constitutes an essential prerequisite for skin repair.

J. Pfeilschifter, G. Geisslinger, H. Kämpfer, 2003, The Journal of investigative dermatology.

Determination of leptin signaling pathways in human and murine keratinocytes.

J. Pfeilschifter, I. Goren, S. Frank, 2003, Biochemical and biophysical research communications.

Identification of copper/zinc superoxide dismutase as a nitric oxide-regulated gene in human (HaCaT) keratinocytes: implications for keratinocyte proliferation.

J. Pfeilschifter, R. Kaufmann, M. Podda, 2000, The Biochemical journal.

Decline in the expression of copper/zinc superoxide dismutase in the kidney of rats with endotoxic shock: Effects of the superoxide anion radical scavenger, tempol, on organ injury

C. Thiemermann, J. Pfeilschifter, A. Olbrich, 1998, British journal of pharmacology.

Macrophage-Derived Heme-Oxygenase-1: Expression, Regulation, and Possible Functions in Skin Repair

J. Pfeilschifter, S Frank, J Pfeilschifter, 2001, Molecular medicine.

Large induction of the chemotactic cytokine RANTES during cutaneous wound repair: a regulatory role for nitric oxide in keratinocyte-derived RANTES expression.

J. Pfeilschifter, B. Stallmeyer, C. Wetzler, 2000, The Biochemical journal.

A transgenic mouse model of inducible macrophage depletion: effects of diphtheria toxin-driven lysozyme M-specific cell lineage ablation on wound inflammatory, angiogenic, and contractive processes.

A. Waisman, J. Pfeilschifter, Nir Yogev, 2009, The American journal of pathology.

Increase of cystathionine-γ-lyase (CSE) during late wound repair: Hydrogen sulfide triggers cytokeratin 10 expression in keratinocytes.

J. Pfeilschifter, K. Beck, I. Goren, 2019, Nitric oxide : biology and chemistry.

A heterogeneous Ly-6B2+ leukocyte population consists of yet undescribed iNOS-expressing cell types in murine skin wounds.

J. Pfeilschifter, U. Christen, I. Goren, 2018, Nitric oxide : biology and chemistry.

The neuronal nitric oxide synthase is upregulated in mouse skin repair and in response to epidermal growth factor in human HaCaT keratinocytes.

U. Förstermann, M. Bros, U. Gödtel-Armbrust, 2004, The Journal of investigative dermatology.

Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair.

J. Pfeilschifter, B. Stallmeyer, S Frank, 2000, The Journal of investigative dermatology.

Leptin enhances wound re-epithelialization and constitutes a direct function of leptin in skin repair.

J. Pfeilschifter, B. Stallmeyer, S Frank, 2000, The Journal of clinical investigation.

Anti-Inflammatory Effects of Rosiglitazone in Obesity-Impaired Wound Healing Depend on Adipocyte Differentiation

J. Pfeilschifter, I. Goren, S. Frank, 2016, PloS one.

The function of nitric oxide in wound repair: inhibition of inducible nitric oxide-synthase severely impairs wound reepithelialization.

J. Pfeilschifter, B. Stallmeyer, S Frank, 1999, The Journal of investigative dermatology.

Regulation of Vascular Endothelial Growth Factor Expression in Cultured Keratinocytes.

M. Longaker, D. Greenhalgh, G. Breier, 1995, The Journal of Biological Chemistry.

Expressional Regulation of Angiopoietin-1 and -2 and the Tie-1 and -2 Receptor Tyrosine Kinases during Cutaneous Wound Healing: A Comparative Study of Normal and Impaired Repair

J. Pfeilschifter, H. Kämpfer, S. Frank, 2001, Laboratory Investigation.

Induction of inducible nitric oxide synthase and its corresponding tetrahydrobiopterin-cofactor-synthesizing enzyme GTP-cyclohydrolase I during cutaneous wound repair.

S. Werner, J. Pfeilschifter, S Frank, 1998, The Journal of investigative dermatology.

Leptin and wound inflammation in diabetic ob/ob mice: differential regulation of neutrophil and macrophage influx and a potential role for the scab as a sink for inflammatory cells and mediators.

J. Pfeilschifter, I. Goren, M. Podda, 2003, Diabetes.

Deregulated unfolded protein response in chronic wounds of diabetic ob/ob mice: a potential connection to inflammatory and angiogenic disorders in diabetes-impaired wound healing.

J. Pfeilschifter, I. Goren, C. Schürmann, 2014, Biochemical and biophysical research communications.

The Dipeptidyl Peptidase-4 Inhibitor Linagliptin Attenuates Inflammation and Accelerates Epithelialization in Wounds of Diabetic ob/ob Mice

J. Pfeilschifter, C. Steinmetz, C. Schürmann, 2012, Journal of Pharmacology and Experimental Therapeutics.

Wound Healing in Mice with High-Fat Diet- or ob Gene-Induced Diabetes-Obesity Syndromes: A Comparative Study

J. Pfeilschifter, O. Seitz, I. Goren, 2011, Experimental diabetes research.

Tight Spatial and Temporal Control in Dynamic Basal to Distal Migration of Epithelial Inflammatory Responses and Infiltration of Cytoprotective Macrophages Determine Healing Skin Flap Transplants in Mice

R. Sader, H. Mühl, J. Pfeilschifter, 2009, Annals of surgery.

Identification of the Fra-1 transcription factor in healing skin flaps transplants: a potential role as a negative regulator of VEGF release from keratinocytes.

R. Sader, J. Pfeilschifter, O. Seitz, 2012, Journal of cranio-maxillo-facial surgery : official publication of the European Association for Cranio-Maxillo-Facial Surgery.

Transforming Growth Factors 1, 2, and 3 and Their Receptors Are Differentially Regulated during Normal and Impaired Wound Healing (*)

S. Werner, S. Frank, M. Madlener, 1996, The Journal of Biological Chemistry.

Epithelial overexpression of SOCS-3 in transgenic mice exacerbates wound inflammation in the presence of elevated TGF-beta1.

J. Pfeilschifter, M. Bösl, I. Goren, 2010, The Journal of investigative dermatology.

p68 DEAD Box RNA Helicase Expression in Keratinocytes

J. Pfeilschifter, I. Goren, S. Frank, 2004, Journal of Biological Chemistry.

Wound inflammation in diabetic ob/ob mice: functional coupling of prostaglandin biosynthesis to cyclooxygenase-1 activity in diabetes-impaired wound healing.

J. Pfeilschifter, G. Geisslinger, R. Schmidt, 2005, Diabetes.

Systemically and topically supplemented leptin fails to reconstitute a normal angiogenic response during skin repair in diabetic ob/ob mice

J. Pfeilschifter, B. Stallmeyer, S. Frank, 2001, Diabetologia.

Akt1 controls insulin-driven VEGF biosynthesis from keratinocytes: implications for normal and diabetes-impaired skin repair in mice.

J. Pfeilschifter, P. Gutwein, O. Seitz, 2009, The Journal of investigative dermatology.

Keratinocyte-Derived Vascular Endothelial Growth Factor Biosynthesis Represents a Pleiotropic Side Effect of Peroxisome Proliferator-Activated Receptor-γ Agonist Troglitazone but Not Rosiglitazone and Involves Activation of p38 Mitogen-Activated Protein Kinase: Implications for Diabetes-Impaired Ski

R. Sader, J. Pfeilschifter, O. Seitz, 2008, Molecular Pharmacology.

Biphasic Regulation of HMG-CoA Reductase Expression and Activity during Wound Healing and Its Functional Role in the Control of Keratinocyte Angiogenic and Proliferative Responses*

J. Pfeilschifter, B. Fisslthaler, G. Geisslinger, 2008, Journal of Biological Chemistry.

Oncostatin M expression is functionally connected to neutrophils in the early inflammatory phase of skin repair: implications for normal and diabetes-impaired wounds.

J. Pfeilschifter, I. Goren, H. Kämpfer, 2006, The Journal of investigative dermatology.

The suppressor of cytokine signaling (SOCS)-3 determines keratinocyte proliferative and migratory potential during skin repair.

J. Pfeilschifter, M. Bösl, I. Goren, 2010, The Journal of investigative dermatology.

Uptake of neutrophil-derived Ym1 protein distinguishes wound macrophages in the absence of interleukin-4 signaling in murine wound healing.

J. Pfeilschifter, I. Goren, S. Frank, 2014, The American journal of pathology.

Regulation of eNOS in normal and diabetes-impaired skin repair: implications for tissue regeneration.

J. Pfeilschifter, B. Stallmeyer, Birgit Stallmeyer, 2002, Nitric oxide : biology and chemistry.

Inducible NO synthase: role in cellular signalling.

H. Mühl, J. Pfeilschifter, A. Huwiler, 1999, The Journal of experimental biology.

Limiting burn extension by transient inhibition of Connexin43 expression at the site of injury.

D. Becker, C. Green, S. Frank, 2005, British journal of plastic surgery.

Dynamic changes in connexin expression correlate with key events in the wound healing process

D. Becker, S. Frank, C. Qiu, 2003, Cell biology international.

Inhibition of cyclooxygenase‐1 and ‐2 activity in keratinocytes inhibits PGE2 formation and impairs vascular endothelial growth factor release and neovascularisation in skin wounds

J. Pfeilschifter, T. Schlich, R. Nüsing, 2017, International wound journal.

Coordinated induction of inducible nitric oxide synthase and GTP-cyclohydrolase I is dependent on inflammatory cytokines and interferon-gamma in HaCaT keratinocytes: implications for the model of cutaneous wound repair.

E. Werner, J. Pfeilschifter, S. Frank, 1998, The Journal of investigative dermatology.

Expression of PKM2 in wound keratinocytes is coupled to angiogenesis during skin repair in vivo and in HaCaT keratinocytes in vitro

J. Pfeilschifter, I. Wittig, I. Goren, 2023, Journal of Molecular Medicine.

Role of wound macrophages in skin flap loss or survival in an experimental diabetes model

J. Pfeilschifter, O. Seitz, I. Goren, 2010, The British journal of surgery.

Reciprocal abrogation of PKM isoforms: contradictory outcomes and differing impact of splicing signal on CRISPR/Cas9 mediates gene editing in keratinocytes

J. Pfeilschifter, G. Imre, I. Goren, 2022, The FEBS journal.

Angiogenic response pattern during normal and impaired skin flap re-integration in mice: a comparative study.

J. Pfeilschifter, O. Seitz, Nadine Schmidt, 2014, Journal of cranio-maxillo-facial surgery : official publication of the European Association for Cranio-Maxillo-Facial Surgery.

Bers-ERK Schwann Cells Coordinate Nerve Regeneration

S. Frank, 2012 .

Excisional Wound Healing

H. Kämpfer, S. Frank, 2003 .

Excisional wound healing. An experimental approach.

H. Kämpfer, S. Frank, 2003, Methods in molecular medicine.

Inhibition of Cytokine-induced Matrix Metalloproteinase 9 Expression by Peroxisome Proliferator-activated Receptor α Agonists Is Indirect and Due to a NO-mediated Reduction of mRNA Stability*

F. Hamada, J. Pfeilschifter, W. Eberhardt, 2002, The Journal of Biological Chemistry.

Regulation of interleukin-18 (IL-18) expression in keratinocytes (HaCaT): implications for early wound healing.

H. Mühl, J. Pfeilschifter, U. Kalina, 2000, European cytokine network.

Protein in Response to IFN-Expression and Release of IL-18 Binding

J. Pfeilschifter, S. Zeuzem, M. Nold, 2001 .

Thr308 determines Akt1 nuclear localization in insulin-stimulated keratinocytes.

J. Pfeilschifter, I. Goren, S. Frank, 2008, Biochemical and biophysical research communications.

Expression and release of chemokines associated with apoptotic cell death in human promonocytic U937 cells and peripheral blood mononuclear cells

H. Mühl, J. Pfeilschifter, W. Eberhardt, 1999, European journal of immunology.