S. Tucker

Monoclonal antibodies selective for α‐synuclein oligomers/protofibrils recognize brain pathology in Lewy body disorders and α‐synuclein transgenic mice with the disease‐causing A30P mutation

L. Lannfelt, M. Ingelsson, H. Kalimo, 2013, Journal of neurochemistry.

Immunotherapy targeting α-synuclein protofibrils reduced pathology in (Thy-1)-h[A30P] α-synuclein mice

L. Lannfelt, M. Ingelsson, C. Möller, 2014, Neurobiology of Disease.

The murine version of BAN2401 (mAb158) selectively reduces amyloid-β protofibrils in brain and cerebrospinal fluid of tg-ArcSwe mice.

L. Lannfelt, A. Satlin, C. Möller, 2014, Journal of Alzheimer's disease : JAD.

Amyloid‐β protofibril levels correlate with spatial learning in Arctic Alzheimer’s disease transgenic mice

L. Lannfelt, L. Hillered, D. Morgan, 2009, The FEBS journal.

Nuclear-targeting of mutant huntingtin fragments produces Huntington's disease-like phenotypes in transgenic mice.

D. Borchelt, A. Savonenko, C. Ross, 2004, Human molecular genetics.

Amyloid precursor protein increases cortical neuron size in transgenic mice

D. Borchelt, A. Savonenko, J. Troncoso, 2009, Neurobiology of Aging.

Amyloid precursor protein increases cortical neuron size in transgenic mice

D. Borchelt, A. Savonenko, J. Troncoso, 2009, Neurobiology of Aging.