C. Murciano

发表

Candidalysin is a fungal peptide toxin critical for mucosal infection

E. Cota, O. Kniemeyer, O. Kurzai, 2016, Nature.

Candida albicans Cell Wall Glycosylation May Be Indirectly Required for Activation of Epithelial Cell Proinflammatory Responses

N. Gow, D. Moyes, Manohursingh Runglall, 2011, Infection and Immunity.

Oral and Vaginal Epithelial Cell Lines Bind and Transfer Cell-Free Infectious HIV-1 to Permissive Cells but Are Not Productively Infected

S. Challacombe, D. Moyes, J. Naglik, 2014, PloS one.

Enhanced proinflammatory response to the Candida albicans gpi7 null mutant by murine cells.

C. Gaillardin, D. Gozalbo, Mathias L Richard, 2008, Microbes and infection.

Host-pathogen interactions in Vibrio vulnificus: responses of monocytes and vascular endothelial cells to live bacteria.

L. Hor, C. Amaro, C. Murciano, 2015, Future microbiology.

Killed Candida albicans Yeasts and Hyphae Inhibit Gamma Interferon Release by Murine Natural Killer Cells

J. O'connor, E. Villamón, D. Gozalbo, 2006, Infection and Immunity.

Signalling through TLR2/MyD88 induces differentiation of murine bone marrow stem and progenitor cells to functional phagocytes in response to Candida albicans

J. O'connor, D. Gozalbo, A. Yáñez, 2010, Cellular microbiology.

Dectin-1 mediates in vitro phagocytosis of Candida albicans yeast cells by retinal microglia.

D. Gozalbo, A. Yáñez, C. Murciano, 2011, FEMS immunology and medical microbiology.

Toll-like receptor 4 defective mice carrying point or null mutations do not show increased susceptibility to Candida albicans in a model of hematogenously disseminated infection.

J. O'connor, E. Villamón, D. Gozalbo, 2006, Medical mycology.

Effect of the Oral Administration of Fungal Ligands in a Murine Model of DSS-Induced Colitis

D. Gozalbo, A. Yáñez, C. Murciano, 2012 .

Evaluation of the Role of Candida albicans Agglutinin-Like Sequence (Als) Proteins in Human Oral Epithelial Cell Interactions

D. Moyes, Manohursingh Runglall, J. Naglik, 2012, PloS one.

Candida albicans Yeast and Hyphae are Discriminated by MAPK Signaling in Vaginal Epithelial Cells

D. Moyes, Manohursingh Runglall, J. Naglik, 2011, PloS one.

Activation of MAPK/c-Fos induced responses in oral epithelial cells is specific to Candida albicans and Candida dubliniensis hyphae

D. Moyes, Manohursingh Runglall, J. Naglik, 2011, Medical Microbiology and Immunology.

In Vivo and In Vitro Studies on Virulence and Host Responses to Saccharomyces cerevisiae Clinical and Non-Clinical Isolates

D. Gozalbo, A. Yáñez, S. Llopis, 2009 .

In vitro response to Candida albicans in cultures of whole human blood from young and aged donors.

J. O'connor, E. Villamón, D. Gozalbo, 2007, FEMS immunology and medical microbiology.

Invitro response toCandidaalbicans in culturesofwhole human blood fromyoungandageddonors

J. O'connor, E. Villamón, D. Gozalbo, 2007 .

Impaired immune response to Candida albicans in aged mice.

J. O'connor, E. Villamón, D. Gozalbo, 2006, Journal of medical microbiology.

A Biphasic Innate Immune MAPK Response Discriminates between the Yeast and Hyphal Forms of Candida albicans in Epithelial Cells

S. Challacombe, H. Mora-Montes, D. Moyes, 2010, Cell host & microbe.

Characterization of a new murine retinal cell line (MU-PH1) with glial, progenitor and photoreceptor characteristics.

V. Gómez-Vicente, N. Cuenca, D. Gozalbo, 2013, Experimental eye research.

Immune Response to Candida albicans Infection

D. Gozalbo, A. Yáñez, C. Murciano, 2016 .

Both viable and killed Candida albicans cells induce in vitro production of TNF-α and IFN-γ in murine cells through a TLR2-dependent signalling

D. Gozalbo, A. Yáñez, C. Murciano, 2007 .

Both viable and killed Candida albicans cells induce in vitro production of TNF-alpha and IFN-gamma in murine cells through a TLR2-dependent signalling.

D. Gozalbo, A. Yáñez, C. Murciano, 2007, European cytokine network.

Influence of aging on murine neutrophil and macrophage function against Candida albicans.

J. O'connor, D. Gozalbo, A. Yáñez, 2008, FEMS immunology and medical microbiology.

In £ uenceofagingonmurineneutrophil andmacrophage function againstCandidaalbicans

J. O'connor, D. Gozalbo, A. Yáñez, 2008 .

Candidalysin activates innate epithelial immune responses via epidermal growth factor receptor

S. Gaffen, R. Gratacap, B. Hube, 2019, Nature Communications.

Role of Toll-like receptors in systemic Candida albicans infections.

D. Gozalbo, A. Yáñez, C. Murciano, 2016, Frontiers in bioscience.

Candida albicans triggers proliferation and differentiation of hematopoietic stem and progenitor cells by a MyD88-dependent signaling.

J. O'connor, D. Gozalbo, A. Yáñez, 2009, Microbes and infection.

MARTX Toxin in the Zoonotic Serovar of Vibrio vulnificus Triggers an Early Cytokine Storm in Mice

L. Hor, Chung-te Lee, B. Fouz, 2017, Front. Cell. Infect. Microbiol..

Candidalysin activates innate epithelial immune responses via epidermal growth factor receptor

S. Gaffen, R. Gratacap, B. Hube, 2019, Nature Communications.

Protection Against Epithelial Damage During Candida albicans Infection Is Mediated by PI3K/Akt and Mammalian Target of Rapamycin Signaling

M. Arno, Estibaliz Aldecoa-Otalora, Cheng-guo Shen, 2013, The Journal of infectious diseases.

Myeloid differentiation factor 88 (MyD88) is required for murine resistance to Candida albicans and is critically involved in Candida -induced production of cytokines.

J. O'connor, E. Villamón, D. Fradelizi, 2004, European cytokine network.

Activation of MAPK/c-Fos induced responses in oral epithelial cells is specific to Candida albicans and Candida dubliniensis hyphae

D. Moyes, Manohursingh Runglall, J. Naglik, 2011, Medical Microbiology and Immunology.

Killed Candida albicans Yeasts and Hyphae Inhibit Gamma Interferon Release by Murine Natural Killer Cells

J. O'connor, E. Villamón, D. Gozalbo, 2006, Infection and Immunity.