Yong-zhong Shi

THE INHIBITION OF LPS-INDUCED PRODUCTION OF INFLAMMATORY CYTOKINES BY HSP70 INVOLVES INACTIVATION OF THE NF-&kgr;B PATHWAY BUT NOT THE MAPK PATHWAYS

D. Tang, Xianzhong Xiao, S. Calderwood, 2006, Shock.

Translocation and Release Peroxide )-Induced HMGB 1 Cytoplasmic Regulator of Oxidative Stress ( Hydrogen Nuclear Heat Shock Protein 72 as a Negative

Haichao Wang, D. Tang, R. Kang, 2007 .

Nuclear Heat Shock Protein 72 as a Negative Regulator of Oxidative Stress (Hydrogen Peroxide)-Induced HMGB1 Cytoplasmic Translocation and Release1

Haichao Wang, D. Tang, R. Kang, 2007, The Journal of Immunology.

Heat shock pretreatment inhibited the release of Smac/DIABLO from mitochondria and apoptosis induced by hydrogen peroxide in cardiomyocytes and C2C12 myogenic cells

Xianzhong Xiao, Mei-dong Liu, Yong-zhong Shi, 2005, Cell stress & chaperones.

Extracellular heat shock protein 70 inhibits tumour necrosis factor-α induced proinflammatory mediator production in fibroblast-like synoviocytes

Xianzhong Xiao, Kangkai Wang, Mei-dong Liu, 2008, Arthritis research & therapy.

HEAT SHOCK RESPONSE INHIBITS RELEASE OF HIGH MOBILITY GROUP BOX 1 PROTEIN INDUCED BY ENDOTOXIN IN MURINE MACROPHAGES

D. Tang, Xianzhong Xiao, Kangkai Wang, 2005, Shock.

Role of Smac/DIABLO in hydrogen peroxide-induced apoptosis in C2C12 myogenic cells.

Xianzhong Xiao, Mei-dong Liu, Yong-zhong Shi, 2005, Free radical biology & medicine.

Hydrogen peroxide stimulates macrophages and monocytes to actively release HMGB1

Haichao Wang, D. Tang, R. Kang, 2007, Journal of leukocyte biology.

[Screen of inflammatory genes regulated by heat shock factor 1 and corroboration with SOCS3 gene].

Y. Liu, Guang-wen Chen, Yong-zhong Shi, 2006, Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences.

[Effect of heat shock response on the cleavage of nucleolin induced by oxidative stress].

D. Tang, Yu-xin Yi, Yong-zhong Shi, 2004, Zhong nan da xue xue bao. Yi xue ban = Journal of Central South University. Medical sciences.