L. Umannová

Benzo[a]pyrene and tumor necrosis factor-α coordinately increase genotoxic damage and the production of proinflammatory mediators in alveolar epithelial type II cells.

A. Kozubík, J. Vondráček, M. Machala, 2011, Toxicology letters.

The interplay of the aryl hydrocarbon receptor and β-catenin alters both AhR-dependent transcription and Wnt/β-catenin signaling in liver progenitors.

A. Kozubík, J. Vondráček, M. Machala, 2011, Toxicological sciences : an official journal of the Society of Toxicology.

Aryl hydrocarbon receptor-mediated disruption of contact inhibition is associated with connexin43 downregulation and inhibition of gap junctional intercellular communication

A. Kozubík, Z. Andrysik, J. Vondráček, 2012, Archives of Toxicology.

Non-dioxin-like polychlorinated biphenyls induce a release of arachidonic acid in liver epithelial cells: a partial role of cytosolic phospholipase A(2) and extracellular signal-regulated kinases 1/2 signalling.

J. Trosko, A. Kozubík, Z. Andrysik, 2008, Toxicology.

Aryl hydrocarbon receptor-mediated disruption of contact inhibition is associated with connexin43 downregulation and inhibition of gap junctional intercellular communication

A. Kozubík, Z. Andrysik, J. Vondráček, 2013, Archives of Toxicology.

Interactions of the aryl hydrocarbon receptor with inflammatory mediators: beyond CYP1A regulation.

J. Vondráček, M. Machala, L. Umannová, 2011, Current drug metabolism.

Tumor necrosis factor-alpha potentiates genotoxic effects of benzo[a]pyrene in rat liver epithelial cells through upregulation of cytochrome P450 1B1 expression.

A. Kozubík, J. Vondráček, M. Machala, 2008, Mutation research.

Tumor Necrosis Factor-α Modulates Effects of Aryl Hydrocarbon Receptor Ligands on Cell Proliferation and Expression of Cytochrome P450 Enzymes in Rat Liver “Stem-Like” Cells

A. Kozubík, B. Hennig, J. Vondráček, 2007 .