Jun Wu

Important role of p38 MAP kinase/NF-kappaB signaling pathway in the sepsis-induced conversion of cardiac myocytes to a proinflammatory phenotype.

Claudio M. Martin, P. Kvietys, T. Rui, 2008, American journal of physiology. Heart and circulatory physiology.

Myocardial protection of MCI-186 in rabbit ischemia-reperfusion.

K. Fung, Jun Wu, Tai-Wing Wu, 2002, Life sciences.

Cell death inhibitors protect against brain damage caused by cardiac ischemia/reperfusion injury

N. Chattipakorn, S. Chattipakorn, N. Apaijai, 2021, Cell death discovery.

The Alarmin Cytokine, High Mobility Group Box 1, Is Produced by Viable Cardiomyocytes and Mediates the Lipopolysaccharide-Induced Myocardial Dysfunction via a TLR4/Phosphatidylinositol 3-Kinase γ Pathway

J. Penninger, Claudio M. Martin, Z. Su, 2009, The Journal of Immunology.

NADPH oxidase contributes to conversion of cardiac myocytes to a proinflammatory phenotype in sepsis.

Claudio M. Martin, P. Kvietys, T. Rui, 2009, Free radical biology & medicine.

Trolox protects rat hepatocytes against oxyradical damage and the ischemic rat liver from reperfusion injury

T. W. Wu, D. Mickle, Jun Wu, 1991, Hepatology.

Suramin protects hepatocytes from LPS-induced apoptosis by regulating mitochondrial stress and inactivating the JNK-Mst1 signaling pathway

Aizhong Wang, Jiali Wang, Jun Wu, 2019, The Journal of Physiological Sciences.

MCI-186: further histochemical and biochemical evidence of neuroprotection.

K. Fung, T. W. Wu, Jun Wu, 2000, Life sciences.

NADPH oxidase contributes to conversion of cardiac myocytes to a proinflammatory phenotype in sepsis.

Claudio M. Martin, P. Kvietys, T. Rui, 2009, Free radical biology & medicine.