J. Shaw

发表

Amendment history : Corrigendum ( September 2017 ) The U 2 AF 1 S 34 F mutation induces lineage-specific splicing alterations in myelodysplastic syndromes

M. Cazzola, P. Vyas, C. Smith, 2018 .

The U2AF1S34F mutation induces lineage-specific splicing alterations in myelodysplastic syndromes

M. Cazzola, P. Vyas, C. Smith, 2017, The Journal of clinical investigation.

The U2AF1S34F mutation induces lineage-specific splicing alterations in myelodysplastic syndromes.

M. Cazzola, P. Vyas, C. Smith, 2017, Journal of Clinical Investigation.

Reversion mutations in patients with leukocyte adhesion deficiency type-1 (LAD-1).

S. Holland, T. Fleisher, S. Rosenzweig, 2008, Blood.

Basic science232. Certolizumab pegol prevents pro-inflammatory alterations in endothelial cell function

H. L. Wright, R. Moots, T. Spector, 2012 .

HLA-B27-Homodimer-Specific Antibody Modulates the Expansion of Pro-Inflammatory T-Cells in HLA-B27 Transgenic Rats

M. van den Broek, M. Hausmann, F. Stenner, 2015, PloS one.

No evidence for a direct role of HLA-B27 in pathological bone formation in axial SpA

R. Lories, J. Shaw, S. Kollnberger, 2017, RMD Open.

Inhibiting HLA-B27 homodimer-driven immune cell inflammation in spondylarthritis.

P. Bowness, K. Maenaka, K. Kuroki, 2012, Arthritis and rheumatism.

Are Stronger Ligands for Leukocyte Ig-like HLA-B27 Homodimers and Free H Chains

P. Bowness, K. Maenaka, K. Kuroki, 2012 .

Receptor B 2 than Classical HLA Class I Are Stronger Ligands for Leukocyte Ig-like HLA-B 27 Homodimers and Free H Chains

P. Bowness, K. Maenaka, K. Kuroki, 2012 .

HLA-B27 Homodimers and Free H Chains Are Stronger Ligands for Leukocyte Ig-like Receptor B2 than Classical HLA Class I

P. Bowness, K. Maenaka, K. Kuroki, 2012, The Journal of Immunology.

New perspectives on the ligands and function of the killer cell immunoglobulin-like receptor KIR3DL2 in health and disease

J. Shaw, S. Kollnberger, 2012, Front. Immun..

Th17 Cells Expressing KIR3DL2+ and Responsive to HLA-B27 Homodimers Are Increased in Ankylosing Spondylitis

Fraser Cummings, A. McMichael, A. McMichael, 2011, The Journal of Immunology.

Defining the repeating elements in the cysteine‐rich region (CRR) of the CD18 integrin β2 subunit

M. Robinson, S. Law, J. Shaw, 2001, FEBS letters.

Expression , and Function ( CD 11 b / CD 18 ) Assembly , Surface LFA-1 ( CD 11 a / CD 18 ) and Mac-1 Subunit Truncations on 2 β Effect of Integrin Law

R. Hyland, W. Douglass, J. Shaw, 2000 .

The biochemistry and immunology of non-canonical forms of HLA-B27.

J. Shaw, S. Kollnberger, H. Hatano, 2014, Molecular immunology.

The Leukocyte Immunoglobulin-Like Receptor Family Member LILRB5 Binds to HLA-Class I Heavy Chains

Demin Li, Marloes J. M. Olde Nordkamp, G. Jiang, 2015, PloS one.

The arthritis-associated HLA-B*27:05 allele forms more cell surface B27 dimer and free heavy chain ligands for KIR3DL2 than HLA-B*27:09

P. Bowness, C. López-Larrea, A. Mathieu, 2013, Rheumatology.

KIR3DL2 Binds to HLA-B27 Dimers and Free H Chains More Strongly than Other HLA Class I and Promotes the Expansion of T Cells in Ankylosing Spondylitis

P. Bowness, I. Wong-Baeza, J. Shaw, 2013, The Journal of Immunology.

This information is current as T Cells in Ankylosing Spondylitis HLA Class I and Promotes the Expansion of Free H Chains More Strongly than Other KIR3DL2 Binds to HLA-B27 Dimers and

P. Bowness, Ricardo A. Fernandes, I. Wong-Baeza, 2013 .

A new macrophage differentiation antigen which is a member of the scavenger receptor superfamily

D. Mason, A. Willis, Yin Dong, 1993, European journal of immunology.

Effect of Integrin β2 Subunit Truncations on LFA-1 (CD11a/CD18) and Mac-1 (CD11b/CD18) Assembly, Surface Expression, and Function1

R. Hyland, A. Al-Shamkhani, S. Law, 2000, The Journal of Immunology.

CSNK1A1 mutations and gene expression analysis in myelodysplastic syndromes with del(5q)

M. Calasanz, C. Mecucci, P. Fenaux, 2015, British journal of haematology.

ASXL1 mutation correction by CRISPR/Cas9 restores gene function in leukemia cells and increases survival in mouse xenografts

A. Verma, A. Schuh, T. Fulga, 2015, Oncotarget.

A novel leukocyte adhesion deficiency caused by expressed but nonfunctional beta2 integrins Mac-1 and LFA-1.

N. Hogg, N. Klein, R. Newton, 1999, The Journal of clinical investigation.

The N-terminal Region and the Mid-region Complex of the Integrin β2 Subunit*

M. Robinson, Y. van Kooyk, J. Shaw, 2001, The Journal of Biological Chemistry.

The role of the cysteine‐rich region of the β2 integrin subunit in the leukocyte function‐associated antigen‐1 (LFA‐1, αLβ2, CD11a/CD18) heterodimer formation and ligand binding

C. Buckley, D. Simmons, R. Hyland, 1998 .

Th17 Cells Expressing KIR3DL2+ and Responsive to HLA-B27 Homodimers Are Increased in Ankylosing Spondylitis

Fraser Cummings, A. McMichael, P. Bowness, 2011, The Journal of Immunology.

The D0 Ig-like Domain Plays a Central Role in the Stronger Binding of KIR3DL2 to B27 Free H Chain Dimers

L. Gauthier, B. Rossi, Demin Li, 2015, The Journal of Immunology.

Activation‐Induced Killer Cell Immunoglobulin‐like Receptor 3DL2 Binding to HLA–B27 Licenses Pathogenic T Cell Differentiation in Spondyloarthritis

Katherine K. Matthews, K. Ladell, P. Bowness, 2015, Arthritis & rheumatology.

Effect of integrin beta 2 subunit truncations on LFA-1 (CD11a/CD18) and Mac-1 (CD11b/CD18) assembly, surface expression, and function.

R. Hyland, A. Al-Shamkhani, S. Law, 2000, Journal of immunology.

Expression of aberrant HLA-B27 molecules is dependent on B27 dosage and peptide supply

P. Bowness, C. Renner, Simon Milling, 2013, Annals of the rheumatic diseases.

Spondyloarthropathies (Including Psoriatic Arthritis) [40–69] 40. Eagle's Syndrome: An Unusual Association with Sero-Negative Arthropathy

L. Coates, D. Gladman, C. May, 2010 .

The D0 Ig-like Domain Plays a Central Role in the Stronger Binding of KIR3DL2 to B27 Free H Chain Dimers

S. Mitchell, L. Gauthier, B. Rossi, 2015, The Journal of Immunology.