H. Yoshida

发表

Alpha1-adrenoceptor-mediated breakdown of phosphatidylinositol 4,5-bisphosphate inhibits pinacidil-activated ATP-sensitive K+ currents in rat ventricular myocytes.

M. Horie, Lai-Hua Xie, M. Takano, 2002, Circulation research.

High risk for bradyarrhythmic complications in patients with Brugada syndrome caused by SCN5A gene mutations.

A. Kobori, M. Horie, T. Kita, 2005, Journal of the American College of Cardiology.

N- and C-terminal KCNE1 mutations cause distinct phenotypes of long QT syndrome.

J. Hancox, M. Horie, T. Kita, 2007, Heart rhythm.

AMP-Activated Protein Kinase Connects Cellular Energy Metabolism to K ATP Channel Function

W. Coetzee, M. Kaneko, H. Yoshida, 2012 .

AMP-activated protein kinase connects cellular energy metabolism to KATP channel function.

W. Coetzee, Eirini Kefaloyianni, M. Kaneko, 2012, Journal of molecular and cellular cardiology.

K ATP channels of primary human coronary artery endothelial cells consist of a heteromultimeric complex of Kir6.1, Kir6.2, and SUR2B subunits.

I. Ghiu, M. Artman, W. Coetzee, 2004, Journal of molecular and cellular cardiology.

Immunolocalization of KATP channel subunits in mouse and rat cardiac myocytes and the coronary vasculature

M. Artman, W. Coetzee, A. Tinker, 2005, BMC Physiology.

Evidence for a Single Nucleotide Polymorphism in the KCNQ1 Potassium Channel that Underlies Susceptibility to Life‐Threatening Arrhythmias

M. Horie, S. Sasayama, M. Takano, 2001, Journal of cardiovascular electrophysiology.

Expression of ATP-Sensitive K+ Channel Subunits during Perinatal Maturation in the Mouse Heart

M. Artman, W. Coetzee, Xiaoyong Tong, 2005, Pediatric Research.

A novel mutation associated with Jervell and Lange-Nielsen syndrome in a Japanese family.

M. Horie, T. Kita, S. Ohno, 2008, Circulation journal : official journal of the Japanese Circulation Society.

Mutation site dependent variability of cardiac events in Japanese LQT2 form of congenital long-QT syndrome.

W. Shimizu, M. Horie, S. Ohno, 2008, Circulation journal : official journal of the Japanese Circulation Society.

Characterization and subcellular localization of KCNQ1 with a heterozygous mutation in the C terminus.

A. Kobori, M. Horie, S. Sasayama, 2001, Journal of molecular and cellular cardiology.

Bradycardia-induced long QT syndrome caused by a de novo missense mutation in the S2-S3 inner loop of HERG.

M. Horie, S. Sasayama, H. Otani, 2001, American journal of medical genetics.

Role of KCNQ1 in the cell swelling-induced enhancement of the slowly activating delayed rectifier K(+) current.

M. Horie, S. Sasayama, M. Takano, 2002, The Japanese journal of physiology.

Consequences of cardiac myocyte-specific ablation of KATP channels in transgenic mice expressing dominant negative Kir6 subunits.

G. Fishman, G. Morley, L. Porter, 2006, American journal of physiology. Heart and circulatory physiology.

Genotype‐phenotype correlations of KCNJ2 mutations in Japanese patients with Andersen‐Tawil syndrome

W. Shimizu, A. Kobori, M. Horie, 2007, Human mutation.

(cid:1) 1 -Adrenoceptor–Mediated Breakdown of Phosphatidylinositol 4,5-Bisphosphate Inhibits Pinacidil-Activated ATP-Sensitive K (cid:2) Currents in Rat Ventricular Myocytes

M. Horie, Lai-Hua Xie, M. Takano, 2002 .

Digitalis-induced abolishment of ischemic preconditioning—Functional correlation between cardiac ATP-sensitive K channels and membrane transporter molecules—

M. Horie, S. Sasayama, T. Murakami, 2000 .

Altered expression of ATP-sensitive K channel subunit after myocardial infarction and its physiological implication

M. Horie, S. Sasayama, M. Takano, 1999 .

The Glycolytic Enzymes, Glyceraldehyde-3-phosphate Dehydrogenase, Triose-phosphate Isomerase, and Pyruvate Kinase Are Components of the KATP Channel Macromolecular Complex and Regulate Its Function*

W. Coetzee, Weixia Liu, S. Srivastava, 2005, Journal of Biological Chemistry.

Mechanistic basis for the pathogenesis of long QT syndrome associated with a common splicing mutation in KCNQ1 gene.

T. Ishii, M. Horie, T. Kita, 2007, Journal of molecular and cellular cardiology.

Characterization of a Novel Missense Mutation in the Pore of HERG in a Patient with Long QT Syndrome

M. Horie, S. Sasayama, M. Takano, 1999, Journal of cardiovascular electrophysiology.

Long QT Syndrome as a Cause of Cardiac Sudden Death

M. Horie, S. Sasayama, H. Kono, 1998 .

Alteration of the membrane lipid environment by L-palmitoylcarnitine modulates KATP channels in guinea-pig ventricular myocytes

M. Horie, S. Sasayama, M. Takano, 2000, Pflügers Archiv.

Novel KCNE3 mutation reduces repolarizing potassium current and associated with long QT syndrome

W. Shimizu, M. Horie, F. Toyoda, 2009, Human mutation.

Adrenergic regulation of the rapid component of delayed rectifier K+ current: implications for arrhythmogenesis in LQT2 patients.

M. Horie, F. Toyoda, K. Tsuji, 2009, Heart rhythm.