L. Fine

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Mechanisms of tubulo‐interstitiaI injury in progressive renal diseases

L. Fine, A. Ong, J. Norman, 1993, European journal of clinical investigation.

Progressive renal disease: the chronic hypoxia hypothesis.

C. Orphanides, L. Fine, L. Fine, 1998, Kidney international. Supplement.

Stimulation of Na+/H+ antiport is an early event in hypertrophy of renal proximal tubular cells.

J. Wells, A. G. Lowe, L. Fine, 1985, Proceedings of the National Academy of Sciences of the United States of America.

Hypoxia-Induced Changes in Extracellular Matrix Metabolism in Renal Cells

J. Norman, C. Orphanides, L. Fine, 1999, Nephron Experimental Nephrology.

Tubular-derived growth factors and cytokines in the pathogenesis of tubulointerstitial fibrosis: implications for human renal disease progression.

L. Fine, A. Ong, Albert C.M. Ong, 1994, American journal of kidney diseases : the official journal of the National Kidney Foundation.

Physiology and cell biology update: polypeptide growth factors and their relation to renal disease.

L. Fine, D. Kujubu, L. Fine, 1989, American journal of kidney diseases : the official journal of the National Kidney Foundation.

Pexicrine effects of basement membrane components on paracrine signaling by renal tubular cells.

J. Norman, L. Fine, L. Fine, 1996, Kidney international.

BSC-1 growth inhibitor transforms a mitogenic stimulus into a hypertrophic stimulus for renal proximal tubular cells: relationship to Na+/H+ antiport activity.

R. W. Holley, H. Nasri, L. Fine, 1985, Proceedings of the National Academy of Sciences of the United States of America.

Patterns of mRNA expression during early cell growth differ in kidney epithelial cells destined to undergo compensatory hypertrophy versus regenerative hyperplasia.

D. Slamon, J. Bowen, A. McDonough, 1988, Proceedings of the National Academy of Sciences of the United States of America.

Tubular-derived cytokines and human renal disease progression.

L. Fine, A. Ong, 1994, Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association.

Cell-cell cross-talk in the pathogenesis of renal interstitial fibrosis.

J. Norman, L. Fine, A. Ong, 1995, Kidney international. Supplement.

Molecular events in the organization of renal tubular epithelium: from nephrogenesis to regeneration.

L. Fine, R. Bacallao, Leon G. Fine, 1989, The American journal of physiology.

Hypoxia stimulates proximal tubular cell matrix production via a TGF-beta1-independent mechanism.

J. Norman, C. Orphanides, L. Fine, 1997, Kidney international.

Unravelling of the molecular mechanisms of kidney stones

R. Thakker, R. Unwin, L. Fine, 1996, The Lancet.

Mechanism of active chloride secretion by shark rectal gland: role of Na-K-ATPase in chloride transport.

J. Forrest, F. Epstein, L. Fine, 1977, The American journal of physiology.

Fibroblasts of rabbit kidney in culture. II. Paracrine stimulation of papillary fibroblasts by PDGF.

H. Rodemann, G. Müller, A. Knecht, 1991, The American journal of physiology.

Fibroblasts of rabbit kidney in culture. I. Characterization and identification of cell-specific markers.

H. Rodemann, G. Müller, A. Knecht, 1991, The American journal of physiology.

Epidermal growth factor accelerates functional recovery from ischaemic acute tubular necrosis in the rat: role of the epidermal growth factor receptor.

Y. Tsau, L. Fine, J. Norman, 1990, Clinical science.

Delayed branching of endothelial capillary-like cords in glycated collagen I is mediated by early induction of PAI-1.

M. Goligorsky, T. Miyata, T. Weinstein, 2001, American journal of physiology. Renal physiology.

Loss of glomerular function and tubulointerstitial fibrosis: cause or effect?

L. Fine, L. Fine, A. Ong, 1994, Kidney international.

Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxia.

L. Fine, J. Norman, D. Bandyopadhay, 2000, Kidney international. Supplement.

[3H]Ethylpropylamiloride, a ligand to analyze the properties of the Na+/H+ exchange system in the membranes of normal and hypertrophied kidneys.

M. Lazdunski, P. Barbry, P. Vigne, 1985, Journal of Biological Chemistry.

An endothelin-1 mediated autocrine growth loop involved in human renal tubular regeneration.

J. Firth, L. Fine, A. Ong, 1995, Kidney international.

Human tubular-derived endothelin in the paracrine regulation of renal interstitial fibroblast function.

J. Firth, M. Kitamura, L. Fine, 1994, Experimental nephrology.

Hypertrophy of renal mitochondria.

L. Fine, J. Norman, T. Bradley, 1990, Journal of the American Society of Nephrology : JASN.