G. Faulkner

发表

Nucleophosmin Interacts with and Inhibits the Catalytic Function of Eukaryotic Initiation Factor 2 Kinase PKR*

L. David, G. Bagby, Q. Pang, 2003, Journal of Biological Chemistry.

, evidence of multifunctionality The Fanconi anemia complementation group C gene product : structural

S. Olson, G. Bagby, Q. Pang, 2001 .

The Fanconi Anemia Protein FANCC Binds to and Facilitates the Activation of STAT1 by Gamma Interferon and Hematopoietic Growth Factors

S. Fagerlie, G. Bagby, Q. Pang, 2000, Molecular and Cellular Biology.

Role of double-stranded RNA-dependent protein kinase in mediating hypersensitivity of Fanconi anemia complementation group C cells to interferon gamma, tumor necrosis factor-alpha, and double-stranded RNA.

S. Fagerlie, M. O'Dwyer, J. Diaz, 2001, Blood.

FANCC interacts with Hsp70 to protect hematopoietic cells from IFN-gamma/TNF-alpha-mediated cytotoxicity.

G. Bagby, Q. Pang, T. Christianson, 2001, The EMBO journal.

of caspase 3 family members dependent activation - hematopoietic progenitor cells involve caspase 8 induced apoptotic responses of Fanconi anemia group C - γ Interferon-

M. O'Dwyer, G. Bagby, T. Christianson, 2013 .

Expression of Apoptosis‐related Genes in Chronic Cyclosporine Nephrotoxicity in Mice

H. Abboud, W. Bennett, Chul-woo Yang, 2002, American journal of transplantation : official journal of the American Society of Transplantation and the American Society of Transplant Surgeons.

Interferon-gamma-induced apoptotic responses of Fanconi anemia group C hematopoietic progenitor cells involve caspase 8-dependent activation of caspase 3 family members.

M. O'Dwyer, Michael O'Dwyer, Winifred Keeble, 2000, Blood.

The Fanconi anemia group C gene product modulates apoptotic responses to tumor necrosis factor-alpha and Fas ligand but does not suppress expression of receptors of the tumor necrosis factor receptor superfamily.

G. Hughes, G. Bagby, W. Keeble, 1999, Experimental hematology.

Inactivation of the Fanconi anemia group C gene augments interferon-gamma-induced apoptotic responses in hematopoietic cells.

Gordon Royle, M. Heinrich, M. Grompe, 1997, Blood.

The Fanconi anemia complementation group C gene product: structural evidence of multifunctionality.

S. Olson, C Reifsteck, S Olson, 2001, Blood.

Functional correction of FAC cells with FANCC suppresses the expression of interferon g – inducible genes

S. Fagerlie, G. Bagby, T. Christianson, 2001 .

Inactivation of the Fanconi anemia group C gene augments interferon-gamma-induced apoptotic responses in hematopoietic cells.

M. Grompe, R. Maziarz, R K Rathbun, 1997, Blood.

Functional correction of FA-C cells with FANCC suppresses the expression of interferon gamma-inducible genes.

S. Fagerlie, J. Diaz, G. Bagby, 2001, Blood.

FANCC interacts with Hsp70 to protect hematopoietic cells from IFN‐γ/TNF‐α‐mediated cytotoxicity

G. Bagby, Q. Pang, T. Christianson, 2001 .

Expression of the Fanconi anemia group C gene in hematopoietic cells is not influenced by oxidative stress, cross-linking agents, radiation, heat, or mitotic inhibitory factors.

M. Heinrich, A. J. Zigler, S. T. Peters, 1998, Experimental hematology.

Assessment of hepatitis C virus quasispecies heterogeneity by gel shift analysis: correlation with response to interferon therapy.

L. Corey, S. Polyak, D. Gretch, 1997, The Journal of infectious diseases.

evidence of multifunctionality The Fanconi anemia complementation group C gene product: structural

S. Olson, G. Bagby, Q. Pang, 2013 .