J. Sipkens

发表

PX-18 Protects Human Saphenous Vein Endothelial Cells under Arterial Blood Pressure.

P. Quax, H. Niessen, V. V. van Hinsbergh, 2017, Annals of vascular surgery.

S-Adenosylhomocysteine induces apoptosis and phosphatidylserine exposure in endothelial cells independent of homocysteine.

H. Blom, C. Stehouwer, H. Niessen, 2012, Atherosclerosis.

Homocysteine-induced cardiomyocyte apoptosis and plasma membrane flip-flop are independent of S-adenosylhomocysteine: a crucial role for nuclear p47phox

C. Stehouwer, H. Niessen, V. V. van Hinsbergh, 2011, Molecular and Cellular Biochemistry.

Original Contribution

C. Schalkwijk, V. V. van Hinsbergh, Nynke E. Hahn, 1988 .

The Dynamics of Compound, Transcript, and Protein Effects After Treatment With 2OMePS Antisense Oligonucleotides in mdx Mice

J. Ridings, Annemieke Aartsma-Rus, S. Hood, 2014, Molecular therapy. Nucleic acids.

Ischemia induces nuclear NOX2 expression in cardiomyocytes and subsequently activates apoptosis

C. Visser, M. Okrój, C. Hack, 2006, Apoptosis.

Inhibition of Rho-ROCK signaling induces apoptotic and non-apoptotic PS exposure in cardiomyocytes via inhibition of flippase.

W. Paulus, C. Stehouwer, V. V. van Hinsbergh, 2010, Journal of molecular and cellular cardiology.

Homocysteine Induces Phosphatidylserine Exposure in Cardiomyocytes through Inhibition of Rho Kinase and Flippase Activity

C. Stehouwer, H. Niessen, V. V. van Hinsbergh, 2011, Cellular Physiology and Biochemistry.

The Pharmacokinetics of 2'-O-Methyl Phosphorothioate Antisense Oligonucleotides: Experiences from Developing Exon Skipping Therapies for Duchenne Muscular Dystrophy.

N. Datson, J. V. van Deutekom, S. Bosgra, 2019, Nucleic acid therapeutics.

P1.6 The incidence of revertant and trace dystrophin expression in muscle biopsies of Duchenne Muscular Dystrophy patients with different exon deletions

C. Beekman, G. Campion, A. Janson, 2011, Neuromuscular Disorders.

Systemic administration of PRO051 in Duchenne's muscular dystrophy.

G. van Ommen, Annemieke Aartsma-Rus, J. Verschuuren, 2011, The New England journal of medicine.

Natural Killer Cells and CD4+ T-Cells Modulate Collateral Artery Development

P. Eilers, P. Quax, J. H. van Bockel, 2007, Arteriosclerosis, thrombosis, and vascular biology.

A Sensitive, Reproducible and Objective Immunofluorescence Analysis Method of Dystrophin in Individual Fibers in Samples from Patients with Duchenne Muscular Dystrophy

C. Beekman, J. V. van Deutekom, G. Campion, 2014, PloS one.

Homocysteine-Induced Apoptosis in Endothelial Cells Coincides With Nuclear NOX2 and Peri-nuclear NOX4 Activity

H. Blom, C. Stehouwer, V. V. van Hinsbergh, 2011, Cell Biochemistry and Biophysics.

Homocysteine affects cardiomyocyte viability: concentration-dependent effects on reversible flip-flop, apoptosis and necrosis

C. Stehouwer, V. V. van Hinsbergh, R. Musters, 2007, Apoptosis.

Chapter 7 Homocysteine-induced apoptosis in endothelial cells coincides with nuclear NOX 2 and peri-nuclear NOX 4 activity

J. Sipkens, 2014 .

Abstract 8670: Homocysteine Induces Reactive Oxygen Species-dependent Apoptosis of Arterial Smooth Muscle Cells Coinciding with (peri)nuclear Nox4 Translocation

C. Stehouwer, H. Niessen, Nynke E. Hahn, 2011 .

Systemic administration of PRO051 in Duchenne's muscular dystrophy.

G. van Ommen, Annemieke Aartsma-Rus, J. Verschuuren, 2011, The New England journal of medicine.

Loss of DPP4 activity is related to a prothrombogenic status of endothelial cells: implications for the coronary microvasculature of myocardial infarction patients

C. Schalkwijk, V. V. van Hinsbergh, Nynke E. Hahn, 2011, Basic Research in Cardiology.