Y. Lee

发表

Correction: potential therapeutic application of gold nanoparticles in B-chronic lymphocytic leukemia (BCLL): enhancing apoptosis

D. Mukhopadhyay, Lichun Lu, M. Yaszemski, 2013, Journal of Nanobiotechnology.

The Nitric Acid Method for Protein Estimation in Biological Samples

S. Kaufmann, K. Bible, S. Boerner, 2002 .

A recombinant IL-4-Pseudomonas exotoxin inhibits protein synthesis and overcomes apoptosis resistance in human CLL B cells.

R. Puri, D. Frank, N. Kay, 2005, Leukemia research.

Phase 1 Trial of Flavopiridol Combined with Cisplatin or Carboplatin in Patients with Advanced Malignancies with the Assessment of Pharmacokinetic and Pharmacodynamic End Points

J. Sloan, C. Erlichman, S. Kaufmann, 2005, Clinical Cancer Research.

Inhibition of GSK-3 Induces Apoptosis of CLL Cells by Abrogating NFkB Nuclear Activity.

N. Kay, A. Ougolkov, D. Billadeau, 2006 .

Adaphostin-induced apoptosis in CLL B cells is associated with induction of oxidative stress and exhibits synergy with fludarabine.

E. Sausville, T. Shanafelt, S. Kaufmann, 2004, Blood.

The Green Tea Extract Epigallocatechin Induces In Vitro Cell Death in Primary Human Lymphoma Cells through an ROS Dependent Mechanism.

T. Shanafelt, S. Ansell, S. Geyer, 2006 .

Potential therapeutic application of gold nanoparticles in B-chronic lymphocytic leukemia (BCLL): enhancing apoptosis

Michael J Yaszemski, Chitta Ranjan Patra, Lichun Lu, 2007, Journal of nanobiotechnology.

Prevalence of Pathogenic Mutations in Cancer Predisposition Genes among Pancreatic Cancer Patients

Raymond M. Moore, F. Couch, W. Bamlet, 2015, Cancer Epidemiology, Biomarkers & Prevention.

Aberrant regulation of pVHL levels by microRNA promotes the HIF/VEGF axis in CLL B cells.

D. Mukhopadhyay, C. Croce, Chang-gong Liu, 2009, Blood.

Serine protease HtrA1 modulates chemotherapy-induced cytotoxicity.

Alfonso Baldi, Jeremy Chien, L. Hartmann, 2006, The Journal of clinical investigation.

Flavopiridol disrupts STAT3/DNA interactions, attenuates STAT3-directed transcription, and combines with the Jak kinase inhibitor AG490 to achieve cytotoxic synergy

K. Bible, C. Isham, Y. Lee, 2006, Molecular Cancer Therapeutics.

Lymphocytic Leukemia Green Tea Component, Epigallocatechin-3-gallate (EGCG) in B cell Chronic VEGF Receptor Phosphorylation Status and Apoptosis is Modulated by a

N. Kay, D. Jelinek, N. Bone, 2013 .

chronic lymphocytic leukemia green tea component, epigallocatechin-3-gallate (EGCG), in B-cell VEGF receptor phosphorylation status and apoptosis is modulated by a

T. Shanafelt, N. Kay, D. Jelinek, 2011 .

VEGF receptor phosphorylation status and apoptosis is modulated by a green tea component, epigallocatechin-3-gallate (EGCG), in B-cell chronic lymphocytic leukemia.

T. Shanafelt, N. Kay, D. Jelinek, 2004, Blood.

Identification of gene mutations in autosomal dominant polycystic kidney disease through targeted resequencing.

B. Eckloff, V. Torres, P. Harris, 2012, Journal of the American Society of Nephrology : JASN.

Bone biopsy derived marrow stromal elements rescue chronic lymphocytic leukemia B-cells from spontaneous and drug induced cell death and facilitates an "angiogenic switch".

T. Shanafelt, N. Kay, A. Raza, 2007, Leukemia research.

Proteomic Analysis of Chronic Lymphocytic Leukemia Cells Identifies Vimentin as a Novel Prognostic Factor for Aggressive Disease.

L. Rassenti, T. Kipps, T. Shanafelt, 2005 .