A. Minn

发表

Insulinomas and expression of an insulin splice variant

S. Libutti, D. Harlan, A. Shalev, 2004, The Lancet.

Thioredoxin-interacting protein is stimulated by glucose through a carbohydrate response element and induces beta-cell apoptosis.

A. Shalev, A. Minn, Christian Hafele, 2005, Endocrinology.

Thioredoxin-Interacting Protein Is Stimulated by Glucose through a Carbohydrate Response Element and Induces (cid:1) -Cell Apoptosis

A. Shalev, A. Minn, Christian Hafele, 2005 .

Resistin is expressed in pancreatic islets.

D. Harlan, O. Gavrilova, C. Vinson, 2003, Biochemical and biophysical research communications.

Gene expression profiling in INS-1 cells overexpressing thioredoxin-interacting protein.

C. Kendziorski, J. Brady, C. Pise-Masison, 2005, Biochemical and biophysical research communications.

Molecular basis for negative regulation of the glucagon receptor

I. Hötzel, A. Estevez, C. Koth, 2012, Proceedings of the National Academy of Sciences.

Exenatide inhibits beta-cell apoptosis by decreasing thioredoxin-interacting protein.

A. Shalev, Francesca M Couto, A. Minn, 2006, Biochemical and biophysical research communications.

Phylogenetics of paniceae (poaceae).

M. Duvall, A. Minn, J. D. Noll, 2001, American journal of botany.

Metabolism-independent sugar effects on gene transcription: the role of 3-O-methylglucose.

A. Shalev, Francesca M Couto, A. Minn, 2006, Biochemistry.

Increased insulin translation from an insulin splice-variant overexpressed in diabetes, obesity, and insulin resistance.

D. Harlan, H. Lan, A. Attie, 2005, Molecular endocrinology.

Exenatide blocks JAK1-STAT1 in pancreatic beta cells.

Christina Kendziorski, Matthew Hanson, Ping Wang, 2007, Metabolism: clinical and experimental.

Molecular, immune and histopathological characterization of NSCLC based on PDL1 expression on tumor and immune cells and association with response to the anti-PDL1 antibody MPDL3280A.

P. Hegde, I. Wistuba, F. Hirsch, 2015 .