R. Moss

Absence of the β subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the α1 subunit and eliminates excitation-contraction coupling

P. Powers, A. Messing, R. Coronado, 1996 .

Profound functional and signaling changes in viable inflammatory neutrophils homing to cystic fibrosis airways

L. Herzenberg, L. Herzenberg, I. Schrijver, 2008, Proceedings of the National Academy of Sciences.

Absence of the beta subunit (cchb1) of the skeletal muscle dihydropyridine receptor alters expression of the alpha 1 subunit and eliminates excitation-contraction coupling.

P. Powers, A. Messing, R. Coronado, 1996, Proceedings of the National Academy of Sciences of the United States of America.

Activation of critical, host-induced, metabolic and stress pathways marks neutrophil entry into cystic fibrosis lungs

L. Herzenberg, L. Herzenberg, Carol K. Conrad, 2009, Proceedings of the National Academy of Sciences.

ACELL October 46/4

J. Dong, Lei Zhang, N. Lewis, 1999 .

Reduced IL‐10 secretion by CD4+ T lymphocytes expressing mutant cystic fibrosis transmembrane conductance regulator (CFTR)

M. Kemna, M. Kemna, P. Gardner, 1996, Clinical and experimental immunology.

Activation of CFTR chloride current by nitric oxide in human T lymphocytes.

R. Moss, A. Chao, P. Gardner, 1995, The EMBO journal.