R. Sartori

发表

Mechano‐signalling pathways in an experimental intensive critical illness myopathy model

J. Gromada, L. Larsson, S. Gastaldello, 2016, The Journal of physiology.

Mechanisms of muscle atrophy and hypertrophy: implications in health and disease

M. Sandri, R. Sartori, V. Romanello, 2021, Nature communications.

Smad2 and 3 transcription factors control muscle mass in adulthood.

C. Mammucari, M. Sandri, B. Blaauw, 2009, American journal of physiology. Cell physiology.

Propeptide-mediated inhibition of myostatin increases muscle mass through inhibiting proteolytic pathways in aged mice.

K. Patel, M. Sandri, A. Matsakas, 2014, The journals of gerontology. Series A, Biological sciences and medical sciences.

TGFβ and BMP signaling in skeletal muscle: potential significance for muscle-related disease

M. Sandri, P. Gregorevic, R. Sartori, 2014, Trends in Endocrinology & Metabolism.

Activation of Akt–mTORC1 signalling reverts cancer‐dependent muscle wasting

B. Blaauw, H. Nolte, S. Ciciliot, 2021, Journal of cachexia, sarcopenia and muscle.

Enhanced exercise and regenerative capacity in a mouse model that violates size constraints of oxidative muscle fibres

K. Patel, O. Kretz, M. Sandri, 2016, eLife.

Epigenetic targeting of bromodomain protein BRD4 counteracts cancer cachexia and prolongs survival

S. Hatakeyama, M. Sandri, S. Merigliano, 2017, Nature Communications.

Bone and morphogenetic protein signalling and muscle mass

M. Sandri, R. Sartori, 2015, Current opinion in clinical nutrition and metabolic care.

Food Restriction Reverses the Hyper-Muscular Phenotype and Force Generation Capacity Deficit of the Myostatin Null Mouse

K. Patel, M. Sandri, E. Masiero, 2012, International Journal of Sports Medicine.

S6K1 Is Required for Increasing Skeletal Muscle Force during Hypertrophy.

M. Pende, S. Pyronnet, B. Blaauw, 2016, Cell reports.

S 6 K 1 Is Required for Incre asing Skeletal Muscle Force during Hypertrophy Graphical Abstract Highlights

M. Pende, S. Pyronnet, B. Blaauw, 2022 .

Exercise training attenuates the hypermuscular phenotype and restores skeletal muscle function in the myostatin null mouse

K. Patel, M. Sandri, A. Matsakas, 2012, Experimental physiology.

Iron supplementation is sufficient to rescue skeletal muscle mass and function in cancer cachexia

E. Hirsch, M. Sandri, M. Mazzone, 2022, EMBO reports.

NAD+ repletion with niacin counteracts cancer cachexia

M. Sandri, P. Porporato, S. Zampieri, 2022, bioRxiv.

Program and Book of the 15th Interuniversity Institute of Myology Meeting - Assisi (Italy), 2018

W. Świȩszkowski, G. Forte, A. Musarò, 2018, European journal of translational myology.

Exercise‐dependent increases in protein synthesis are accompanied by chromatin modifications and increased MRTF‐SRF signalling

K. Vissing, M. Krüger, A. A. Mir, 2020, Acta physiologica.

BMP signaling controls muscle mass

Stefano Piccolo, Marco Sandri, Luana Toniolo, 2013, Nature Genetics.

BMPs and the muscle-bone connection.

M. Sandri, R. Sartori, 2015, Bone.

Perturbed BMP signaling and denervation promote muscle wasting in cancer cachexia

L. Larsson, M. Sandri, S. Merigliano, 2021, Science Translational Medicine.

C‐terminal agrin fragment as a biomarker of muscle wasting and weakness: a narrative review

H. Kern, M. Narici, S. Löfler, 2023, Journal of cachexia, sarcopenia and muscle.

The Prognostic Value of Low Muscle Mass in Pancreatic Cancer Patients: A Systematic Review and Meta-Analysis

A. Brazzale, S. Merigliano, C. Sperti, 2021, Journal of clinical medicine.

MYTHO is a novel regulator of skeletal muscle autophagy and integrity

Anais Franco-Romero, M. Sandri, S. Hussain, 2023, Nature Communications.

Plasma proteome profiling of healthy subjects undergoing bed rest reveals unloading‐dependent changes linked to muscle atrophy

M. Narici, G. Biolo, M. Murgia, 2022, Journal of cachexia, sarcopenia and muscle.

Epigenetic targeting of bromodomain protein BRD4 counteracts cancer cachexia and prolongs survival

S. Hatakeyama, M. Sandri, S. Merigliano, 2017, Nature Communications.

Correction: Symmorphosis through Dietary Regulation: A Combinatorial Role for Proteolysis, Autophagy and Protein Synthesis in Normalising Muscle Metabolism and Function of Hypertrophic Mice after Acute Starvation

K. Patel, M. Sandri, A. Matsakas, 2015, PloS one.

Symmorphosis through Dietary Regulation: A Combinatorial Role for Proteolysis, Autophagy and Protein Synthesis in Normalising Muscle Metabolism and Function of Hypertrophic Mice after Acute Starvation

K. Patel, M. Sandri, A. Matsakas, 2015, PloS one.

The ERG1A K+ Channel Is More Abundant in Rectus abdominis Muscle from Cancer Patients Than that from Healthy Humans

H. Kern, J. Davie, M. Sandri, 2021, Diagnostics.

CIR-Myo News : Proceedings of the 2014 Spring Padua Muscle Days

M. Sandri, S. Zampieri, S. Mosole, 2014 .

BMPs and the muscle-bone connection.

M. Sandri, R. Sartori, 2015, Bone.

The ERG1 Potassium Channel is More Abundant in Skeletal Muscle from Cachectic than Healthy Humans

H. Kern, J. Davie, M. Sandri, 2020 .

Mechano‐signalling pathways in an experimental intensive critical illness myopathy model

J. Gromada, L. Larsson, J. Ruas, 2016, The Journal of physiology.

Targeting senescence induced by age or chemotherapy with a polyphenol-rich natural extract improves longevity and healthspan in mice.

M. Montopoli, E. Pasquini, R. Luisetto, 2024, Nature aging.