Hong Liu

发表

Mitochondria Oxidative Stress, Connexin43 Remodeling, and Sudden Arrhythmic Death

M. Bonini, S. Dudley, E. Dolmatova, 2013, Circulation. Arrhythmia and electrophysiology.

Inhibition of c-Src tyrosine kinase prevents angiotensin II-mediated connexin-43 remodeling and sudden cardiac death.

M. Bonini, S. Iravanian, K. Bernstein, 2011, Journal of the American College of Cardiology.

Elevation of B-type natriuretic peptide is a sensitive marker of left ventricular diastolic dysfunction in patients with maintenance haemodialysis

Bicheng Liu, Ying-Zi Zhang, Hong Liu, 2010, Biomarkers : biochemical indicators of exposure, response, and susceptibility to chemicals.

Reactive Oxygen Species Originating from Mitochondria Regulate the Cardiac Sodium Channel

S. Dudley, Hong Liu, Man Liu, 2010 .

c-Src kinase inhibition reduces arrhythmia inducibility and connexin43 dysregulation after myocardial infarction.

I. Efimov, J. Gemel, F. Ng, 2014, Journal of the American College of Cardiology.

Caveolin-1 Modulates Cardiac Gap Junction Homeostasis and Arrhythmogenecity by Regulating cSrc Tyrosine Kinase

M. Bonini, Kai-Chien Yang, R. Minshall, 2014, Circulation. Arrhythmia and electrophysiology.

RNA Binding Protein, HuR, Regulates SCN5A Expression Through Stabilizing MEF2C transcription factor mRNA

S. Dudley, G. Kang, E. Jeong, 2018, Journal of the American Heart Association.

Tetrahydrobiopterin improves diastolic dysfunction by reversing changes in myofilament properties.

R. Solaro, S. Dudley, Bindiya G. Patel, 2013, Journal of molecular and cellular cardiology.

Magnesium Deficiency Causes a Reversible, Metabolic, Diastolic Cardiomyopathy

Xiaoxu Zhou, S. Dudley, G. Kang, 2021, Journal of the American Heart Association.

Circulating S‐Glutathionylated cMyBP‐C as a Biomarker for Cardiac Diastolic Dysfunction

Xiaoxu Zhou, S. Dudley, K. Kavanagh, 2022, Journal of the American Heart Association.

Mitochondrial Ca2+ flux modulates spontaneous electrical activity in ventricular cardiomyocytes

K. Boheler, S. Dudley, Hong Liu, 2018, PloS one.

Interleukin-1β, Oxidative Stress, and Abnormal Calcium Handling Mediate Diabetic Arrhythmic Risk

D. Terentyev, Radmila Terentyeva, S. Dudley, 2021, JACC. Basic to translational science.

Reduced sarcoplasmic reticulum Ca2+ pump activity is anti-arrhythmic in ischemic cardiomyopathy.

S. Dudley, G. Kang, Hong Liu, 2022, Heart rhythm.

Mitochondrial Ca2+ Influx Contributes to Arrhythmic Risk in Nonischemic Cardiomyopathy

Z. Qu, Lai-Hua Xie, Z. Song, 2018, Journal of the American Heart Association.

HuR-mediated SCN5A messenger RNA stability reduces arrhythmic risk in heart failure.

Tae Yun Kim, S. Dudley, Bum-Rak Choi, 2018, Heart rhythm.

HuR-mediated SCN5A mRNA stability reduces arrhythmic risk in heart failure

Tae Yun Kim, S. Dudley, G. Kang, 2018 .

Inflammatory Macrophage Interleukin-1β Mediates High-Fat Diet-Induced Heart Failure With Preserved Ejection Fraction

D. Bernlohr, S. Dudley, G. Kang, 2022, JACC. Basic to translational science.

NF-kappaB-dependent transcriptional regulation of the cardiac scn5a sodium channel by angiotensin II.

L. Shang, A. Pfahnl, S. Sanyal, 2008, American journal of physiology. Cell physiology.

Role of Mitochondrial Oxidative Stress in Glucose Tolerance, Insulin Resistance, and Cardiac Diastolic Dysfunction

E. Lewandowski, A. Farzaneh-Far, S. Gladstein, 2016, Journal of the American Heart Association.

Diastolic dysfunction is associated with cardiac fibrosis in the senescence-accelerated mouse.

D. Sorescu, S. Dudley, R. Sutliff, 2011, American journal of physiology. Heart and circulatory physiology.

MIR448 antagomir reduces arrhythmic risk after myocardial infarction by upregulating the cardiac sodium channel

S. Dudley, G. Kang, Hong Liu, 2020, JCI insight.

Reactive Oxygen Species Originating From Mitochondria Regulate the Cardiac Sodium Channel

S. Dudley, Hong Liu, Man Liu, 2010, Circulation research.

the cardiac scn5a sodium channel by angiotensin II NF-{kappa}B-dependent transcriptional regulation of

L. Shang, A. Pfahnl, Zhe Jiao, 2008 .

Inhibition of renin-angiotensin system (RAS) reduces ventricular tachycardia risk by altering connexin43

S. Iravanian, K. Bernstein, Zhe Jiao, 2011, Journal of Molecular Medicine.

Inhibition of the unfolded protein response reduces arrhythmic risk after myocardial infarction.

S. Dudley, E. Tolkacheva, G. Kang, 2021, The Journal of clinical investigation.

Magnesium supplementation improves diabetic mitochondrial and cardiac diastolic function.

E. So, S. Dudley, E. Jeong, 2019, JCI insight.

Ranolazine Improves Cardiac Diastolic Dysfunction Through Modulation of Myofilament Calcium Sensitivity

D. Sorescu, L. Belardinelli, R. Solaro, 2010, Circulation research.

Uncoupled Cardiac Nitric Oxide Synthase Mediates Diastolic Dysfunction

D. Harrison, B. Wolska, K. Bernstein, 2010, Circulation.

Self-management practices of Chinese Americans with type 2 diabetes.

W. Pan, Hong Liu, Yin Xu, 2010, Nursing & health sciences.

Mitochondrial dysfunction causing cardiac sodium channel downregulation in cardiomyopathy.

I. Efimov, S. Dudley, M. Sulkin, 2013, Journal of molecular and cellular cardiology.

TRPM7 kinase mediates hypomagnesemia-induced seizure-related death

Esther Krook-Magnuson, S. Dudley, Hong Liu, 2023, Scientific reports.

Magnesium supplementation improves diabetic mitochondrial and cardiac diastolic function.

E. So, S. Dudley, E. Jeong, 2019, JCI insight.

Uncoupled Cardiac Nitric Oxide Synthase Mediates Diastolic Dysfunction

S. Dikalov, J. Lovelock, Hong Liu, 2010, Circulation.

Mitochondrial dysfunction causing cardiac sodium channel downregulation in cardiomyopathy.

I. Efimov, S. Dudley, M. Sulkin, 2013, Journal of molecular and cellular cardiology.