D. Boeldt

发表

Pregnancy Enhances Sustained Ca2+ Bursts and Endothelial Nitric Oxide Synthase Activation in Ovine Uterine Artery Endothelial Cells Through Increased Connexin 43 Function1

R. Magness, J. Sullivan, M. Grummer, 2010, Biology of reproduction.

Maternal disease and gasotransmitters.

D. Boeldt, Aishwarya Rengarajan, Amanda K. Mauro, 2020, Nitric oxide : biology and chemistry.

Conjugated linoleic acid improves endothelial Ca2+ signaling by blocking growth factor and cytokine-mediated Cx43 phosphorylation

I. Bird, D. Shah, Amanda C. Ampey, 2020, Molecular and Cellular Endocrinology.

eNOS activation and NO function: pregnancy adaptive programming of capacitative entry responses alters nitric oxide (NO) output in vascular endothelium--new insights into eNOS regulation through adaptive cell signaling.

F. Yi, I. Bird, D. Boeldt, 2011, The Journal of endocrinology.

The loss of sustained Ca(2+) signaling underlies suppressed endothelial nitric oxide production in preeclamptic pregnancies: implications for new therapy.

M. Grummer, F. Yi, I. Bird, 2013, American journal of physiology. Heart and circulatory physiology.

Vascular adaptation in pregnancy and endothelial dysfunction in preeclampsia.

I. Bird, D. Boeldt, D. S. Boeldt, 2017, The Journal of endocrinology.

Fatty acids in normal and pathological pregnancies

D. Boeldt, Aishwarya Rengarajan, Amanda K. Mauro, 2021, Molecular and Cellular Endocrinology.

Altered VEGF-stimulated Ca2+ signaling in part underlies pregnancy-adapted eNOS activity in UAEC.

R. Magness, M. Grummer, I. Bird, 2014, The Journal of endocrinology.

Artery Endothelium: Relative roles in pregnancy adaptation and reversal by

R. Magness, F. Yi, I. Bird, 2011 .

[Ca2+]i signaling vs. eNOS expression as determinants of NO output in uterine artery endothelium: relative roles in pregnancy adaptation and reversal by VEGF165.

R. Magness, F. Yi, I. Bird, 2011, American journal of physiology. Heart and circulatory physiology.

Pregnancy programming and preeclampsia: identifying a human endothelial model to study pregnancy-adapted endothelial function and endothelial adaptive failure in preeclamptic subjects.

M. Grummer, F. Yi, I. Bird, 2014, Advances in experimental medicine and biology.

Positive versus negative effects of VEGF165 on Ca2+ signaling and NO production in human endothelial cells.

F. Yi, I. Bird, D. Shah, 2017, American journal of physiology. Heart and circulatory physiology.

Regulating Specific Growth Factor Signaling Using Immobilized Branched Ligands

W. Murphy, I. Bird, M. W. Toepke, 2012, Advanced healthcare materials.

Endothelial Nitric Oxide Production in Preeclamptic Pregnancies

A. Heather, M. Grummer, F. Yi, 2013 .

TNF-alpha inhibits pregnancy-adapted Ca2+ signaling in uterine artery endothelial cells

R. Magness, M. Grummer, F. Yi, 2019, Molecular and Cellular Endocrinology.

Pregnancy Induced Reprogramming of Endothelial Function in Response to ATP: Evidence for Post Receptor Ca2+ Signaling Plasticity

F. Yi, I. Bird, D. Boeldt, 2010 .

Cytokine concentrations direct endothelial function in pregnancy and preeclampsia.

D. Adu, D. Shah, Amanda C. Ampey, 2020, The Journal of endocrinology.

Differential Distribution of Tryptophan-Metabolites in Fetal and Maternal Circulations During Normotensive and Preeclamptic Pregnancies

Kai Wang, Jing Zheng, W. Lei, 2021, Reproductive Sciences.

Pregnancy, programming and preeclampsia: gap junctions at the nexus of pregnancy-induced adaptation of endothelial function and endothelial adaptive failure in PE.

R. Magness, M. Grummer, F. Yi, 2013, Current vascular pharmacology.

Adenoviral transduction of EGFR into pregnancy-adapted uterine artery endothelial cells remaps growth factor induction of endothelial dysfunction

P. Bertics, M. Grummer, I. Bird, 2020, Molecular and Cellular Endocrinology.