C. Aylsworth

发表

Further characterization of the in vitro assay for inhibitors of metabolic cooperation in the Chinese hamster V79 cell line.

J. Trosko, C. Chang, C. Jone, 1985, Carcinogenesis.

Promotion of 7,12-dimethylbenz[a]anthracene-induced mammary tumorigenesis by high dietary fat in the rat: possible role of intercellular communication.

J. Trosko, J. Meites, C. Jone, 1984, Journal of the National Cancer Institute.

Intestinal protozoa are hypothesized to stimulate immunosurveillance against colon cancer.

C. Aylsworth, D. Juckett, J. Quensen, 2008, Medical hypotheses.

Protein from intestinal Eimeria protozoan stimulates IL‐12 release from dendritic cells, exhibits antitumor properties in vivo and is correlated with low intestinal tumorigenicity

N. Dimitrov, S. Kessel, C. Aylsworth, 2005, International journal of cancer.

Enhancement of murine mammary tumorigenesis by feeding high levels of dietary fat: a hormonal mechanism?

C. Welsch, C. Aylsworth, 1983, Journal of the National Cancer Institute.

Effects of fatty acids on gap junctional communication: Possible role in tumor promotion by dietary fat

J. Trosko, C. Welsch, C. Aylsworth, 1987, Lipids.

Possible Involvement of Arachidonate Products in Tumor Promoter Inhibition of Cell-Cell Communication

J. Trosko, C. Jone, C. Aylsworth, 1985 .

Influence of underfeeding during the "critical period" or thereafter on carcinogen-induced mammary tumors in rats.

P. Sylvester, J. Meites, D. V. Van Vugt, 1982, Cancer research.

Normal but not carcinomatous primary rat mammary epithelium: readily transplanted to and maintained in the athymic nude mouse.

C. Welsch, L. Sheffield, C. Aylsworth, 1987, Journal of the National Cancer Institute.

Influence of lipids on gap-junction-mediated intercellular communication between Chinese hamster cells in vitro.

J. Trosko, C. Welsch, C. Aylsworth, 1986, Cancer research.

Effects of lipids on gap junctionally-mediated intercellular communication: possible role in the promotion of tumorigenesis by dietary fat.

C. Aylsworth, 1986, Progress in clinical and biological research.

Immunoprophylaxis of Punta Toro virus (Phlebovirus, Bunyaviridae) infection in hamsters with recombinant Eimeria profilin-like antigen.

P. Melby, B. Gowen, M. Wong, 2008, International immunopharmacology.

Role of adrenals and estrogen in regression of mammary tumors during postpartum lactation in the rat.

J. Meites, G. Kledzik, C. Aylsworth, 1979, Cancer research.

Influence of dietary retinyl acetate on normal rat mammary gland development and on the enhancement of 7,12-dimethylbenz[a]anthracene-induced rat mammary tumorigenesis by high levels of dietary fat.

M. Zile, C. Welsch, C. Aylsworth, 1986, Journal of the National Cancer Institute.

Autoimmune Disease-Associated Variants of Extracellular Endoplasmic Reticulum Aminopeptidase 1 Induce Altered Innate Immune Responses by Human Immune Cells

D. Georgiadis, A. Amalfitano, E. Stratikos, 2015, Journal of Innate Immunity.

Elimination of metabolic cooperation is associated with the tumor promoters, oleic acid and anthralin.

J. Trosko, C. Jone, C. Aylsworth, 1982, Carcinogenesis.

Phase I Study and Preliminary Pharmacology of the Novel Innate Immune Modulator rBBX-01 in Gynecologic Cancers

D. Mutch, J. Rader, N. Dimitrov, 2008, Clinical Cancer Research.

Effects of alterations in early hormonal environment on development and hormone dependency of carcinogen-induced mammary tumors in rats.

P. Sylvester, J. Meites, D. V. Van Vugt, 1983, Cancer research.

Relation of the Neuroendocrine System to the Development of Mammary Tumors in Rats during Aging

C. Welsch, C. Aylsworth, 1983 .

Role of estrogen and prolactin in stimulation of carcinogen-induced mammary tumor development by a high-fat diet.

P. Sylvester, J. Meites, D. V. Van Vugt, 1984, Cancer research.

Inhibition of mammary tumor growth by dexamethasone in rats in the presence of high serum prolactin levels.

P. Sylvester, J. Meites, C. Aylsworth, 1980, Cancer research.

Endoplasmic reticulum aminopeptidase-1 alleles associated with increased risk of ankylosing spondylitis reduce HLA-B27 mediated presentation of multiple antigens

A. Amalfitano, Irini Evnouchidou, I. York, 2013, Autoimmunity.

Endoplasmic Reticulum Aminopeptidase-1 Functions Regulate Key Aspects of the Innate Immune Response

A. Amalfitano, S. Seregin, Sungjin Kim, 2013, PloS one.

Manipulation of EAT-2 expression promotes induction of multiple beneficial regulatory and effector functions of the human innate immune system as a novel immunomodulatory strategy.

A. Amalfitano, S. Seregin, S. Godbehere, 2014, International immunology.

Retinoid feeding, hormone inhibition, and/or immune stimulation and the progression of N-methyl-N-nitrosourea-induced rat mammary carcinoma: suppression by retinoids of peptide hormone-induced tumor cell proliferation in vivo and in vitro.

C. Welsch, C. Aylsworth, J. Dehoog, 1984, Cancer research.

Comparison of acute effects of dynorphin and beta-endorphin on prolactin release in the rat.

P. Sylvester, J. Meites, D. V. Van Vugt, 1981, Endocrinology.

Relationship of hormones to inhibition of mammary tumor development by underfeeding during the "critical period" after carcinogen administration.

P. Sylvester, J. Meites, C. Aylsworth, 1981, Cancer research.

Evidence for hypothalamic noradrenergic involvement in naloxone-induced stimulation of luteinizing hormone release.

P. Sylvester, J. Meites, D. V. Van Vugt, 1981, Neuroendocrinology.

Effects of a prolactin-secreting pituitary tumor on hypothalamic, gonadotropic and testicular function in male rats.

J. Simpkins, R. Steger, C. Aylsworth, 1980, Neuroendocrinology.

Counteraction of gonadal steroid inhibition of luteinizing hormone release by naloxone.

P. Sylvester, J. Meites, D. V. Van Vugt, 1982, Neuroendocrinology.

Retinola Feeding , Hormone Inhibition , and / or Immune Stimulation and the Progression of / V-Methyl-N-nitrosourea-induced Rat Mammary Carcinoma : Suppression by Retinoids of Peptide Hormone-induced Tumor Cell Proliferation in Vivo and in Vitro 1

C. Welsch, C. Aylsworth, J. Dehoog, 2006 .

TRIF Is a Critical Negative Regulator of TLR Agonist Mediated Activation of Dendritic Cells In Vivo

A. Amalfitano, D. Appledorn, S. Seregin, 2011, PloS one.

Opiate Antagonists Can Inhibit Mammary Tumor Growth in Rats 1

J. Meites, C. Aylsworth, C. Hodson, 1979, Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine.

Inhibitionof MammaryTumor Growthby Dexamethasonein Rats in the Presenceof HighSerum ProlactinLevels 1

P. Sylvester, J. Meites, C. Aylsworth, 2006 .

Synergistic inhibition of metabolic cooperation by oleic acid or 12-0-tetradecanoylphorbol-13-acetate and dichlorodiphenyltrichlorethane (DDT) in Chinese hamster V79 cells: Implication of a role for protein kinase C in the regulation of gap junctional intercellular communication

J. Trosko, Chia‐cheng Chang, C. Aylsworth, 2004, Cell Biology and Toxicology.

Activation of human natural killer cells by the novel innate immune modulator recombinant Eimeria antigen.

A. Amalfitano, S. Seregin, S. Godbehere, 2013, Human immunology.