R. Keeton

发表

Immune control of Mycobacterium tuberculosis is dependent on both soluble TNFRp55 and soluble TNFRp75

M. Jacobs, F. Dube, J. du Toit, 2021, Immunology.

Soluble TNFRp 75 regulates host protective immunity against Mycobacterium tuberculosis

B. Ryffel, M. Jacobs, V. Quesniaux, 2014 .

Soluble TNFRp75 regulates host protective immunity against Mycobacterium tuberculosis.

B. Ryffel, M. Jacobs, V. Quesniaux, 2014, The Journal of clinical investigation.

Reactivation of M. tuberculosis Infection in Trans-Membrane Tumour Necrosis Factor Mice

M. Jacobs, L. Fick, V. Quesniaux, 2011, PloS one.

Neurons Are Host Cells for Mycobacterium tuberculosis

B. Ryffel, L. Kellaway, M. Jacobs, 2014, Infection and Immunity.

BCG‐mediated protection against M. tuberculosis is sustained post‐malaria infection independent of parasite virulence

S. Magez, M. Jacobs, R. Keeton, 2021, Immunology.

TNF-dependent regulation and activation of innate immune cells are essential for host protection against cerebral tuberculosis

B. Ryffel, D. Govender, L. Kellaway, 2015, Journal of Neuroinflammation.

Persistent p55TNFR expression impairs T cell responses during chronic tuberculosis and promotes reactivation

B. Ryffel, M. Jacobs, V. Quesniaux, 2016, Scientific Reports.

Neurons Are Host Cells for Mycobacterium tuberculosis

B. Ryffel, L. Kellaway, M. Jacobs, 2014, Journal of Neuroimmunology.

Breakthrough infections with SARS-CoV-2 omicron despite mRNA vaccine booster dose

C. Riou, W. Preiser, W. Burgers, 2022, The Lancet.

Prior infection with SARS-CoV-2 boosts and broadens Ad26.COV2.S immunogenicity in a variant-dependent manner

S. Richardson, P. Moore, N. Garrett, 2021, Cell Host & Microbe.

T cell responses to SARS-CoV-2 spike cross-recognize Omicron

A. Sette, A. Sigal, T. de Oliveira, 2022, Nature.

Myeloid and T Cell-Derived TNF Protects against Central Nervous System Tuberculosis

B. Ryffel, M. Jacobs, V. Quesniaux, 2017, Front. Immunol..

Limited Role for Lymphotoxin α in the Host Immune Response to Mycobacterium tuberculosis

B. Ryffel, M. Drutskaya, S. Nedospasov, 2010, The Journal of Immunology.

SARS-CoV-2 spike T cell responses induced upon vaccination or infection remain robust against Omicron

A. Sigal, L. Bekker, C. Riou, 2021, medRxiv.

Prior infection with SARS-CoV-2 boosts and broadens Ad26.COV2.S immunogenicity in a variant-dependent manner

A. Sette, S. Richardson, P. Moore, 2021, Cell Host & Microbe.

IL-4 Receptor-Alpha Signalling of Intestinal Epithelial Cells, Smooth Muscle Cells, and Macrophages Plays a Redundant Role in Oxazolone Colitis

F. Brombacher, J. C. Hoving, R. Keeton, 2020, Mediators of inflammation.

SARS-CoV-2 antigens expressed in plants detect antibody responses in 1 COVID-19 patients 2

L. Scott, I. Sanne, W. Stevens, 2020 .

TNFRp75‐dependent immune regulation of alveolar macrophages and neutrophils during early Mycobacterium tuberculosis and Mycobacterium bovis BCG infection

M. Jacobs, R. Keeton, N. Hsu, 2020, Immunology.

Impact of SARS-CoV-2 exposure history on the T cell and IgG response

A. Sette, L. Bekker, C. Riou, 2022, Cell Reports Medicine.

SARS-CoV-2 Antigens Expressed in Plants Detect Antibody Responses in COVID-19 Patients

J. Blackburn, L. Scott, I. Sanne, 2020, Frontiers in Plant Science.

NK-, NKT- and CD8-Derived IFNγ Drives Myeloid Cell Activation and Erythrophagocytosis, Resulting in Trypanosomosis-Associated Acute Anemia

L. Boon, F. Brombacher, S. Magez, 2015, PLoS pathogens.

Escape from recognition of SARS-CoV-2 Beta variant spike epitopes but overall preservation of T cell immunity

T. de Oliveira, R. Wilkinson, P. Moore, 2021, Science Translational Medicine.

Loss of recognition of SARS-CoV-2 B.1.351 variant spike epitopes but overall preservation of T cell immunity

T. de Oliveira, R. Wilkinson, P. Moore, 2021, medRxiv.

IL-4-Responsive B Cells Are Detrimental During Chronic Tuberculosis Infection in Mice

S. P. Parihar, F. Brombacher, R. Guler, 2021, Frontiers in Immunology.

Author Correction: T cell responses to SARS-CoV-2 spike cross-recognize Omicron

A. Sette, A. Sigal, T. de Oliveira, 2022, Nature.

Homologous Ad26.COV2.S vaccination results in reduced boosting of humoral responses in hybrid immunity, but elicits antibodies of similar magnitude regardless of prior infection

S. Richardson, P. Moore, N. Garrett, 2023, medRxiv.

Prominent role for T cell-derived Tumour Necrosis Factor for sustained control of Mycobacterium tuberculosis infection

Sergei A. Nedospasov, B. Ryffel, S. Nedospasov, 2013, Scientific Reports.

Th22 Cells Are a Major Contributor to the Mycobacterial CD4+ T Cell Response and Are Depleted During HIV Infection

R. Wilkinson, C. Riou, W. Burgers, 2019, The Journal of Immunology.

TNF-dependent regulation and activation of innate immune cells are essential for host protection against cerebral tuberculosis

B. Ryffel, D. Govender, L. Kellaway, 2015, Journal of Neuroinflammation.

Author Correction: T cell responses to SARS-CoV-2 spike cross-recognize Omicron

A. Sette, A. Sigal, R. Wilkinson, 2022 .

The C-Type Lectin Receptor CLECSF8/CLEC4D Is a Key Component of Anti-Mycobacterial Immunity

M. Netea, G. Besra, B. Alisjahbana, 2015, Cell host & microbe.

Infection pre-Ad26.COV2.S-vaccination primes greater class switching and reduced CXCR5 expression by SARS-CoV-2-specific memory B cells

A. Sigal, A. Leslie, S. Richardson, 2023, NPJ vaccines.

Distinct T cell functional profiles in SARS-CoV-2 seropositive and seronegative children associated with endemic human coronavirus cross-reactivity

H. Zar, L. Zühlke, P. Moore, 2023, medRxiv.

Mycobacterium Immune Response to in the Host a Limited Role for Lymphotoxin

M. Drutskaya, V. Vasseur, R. Guler, 2010 .

Neurons Are Host Cells for Mycobacterium tuberculosis

B. Ryffel, L. Kellaway, M. Jacobs, 2014, Journal of Neuroimmunology.