J. Sonnemann

发表

Tribbles 2 mediates cisplatin sensitivity and DNA damage response in epithelial ovarian cancer

I. Runnebaum, D. Steinbach, Margarita Melnikova, 2017, International journal of cancer.

Molecular and Translational Classifications of DAMPs in Immunogenic Cell Death

Michael R Hamblin, P. Vandenabeele, L. Zitvogel, 2015, Front. Immunol..

The histone deacetylase inhibitor vorinostat induces calreticulin exposure in childhood brain tumour cells in vitro

J. Beck, M. Schmudde, S. Wittig, 2010, Cancer Chemotherapy and Pharmacology.

Cooperative treatment effectiveness of ATR and HSP90 inhibition in Ewing’s sarcoma cells

Zhao-Qi Wang, René Thierbach, Yibo Geng, 2021, Cell & bioscience.

The bisphosphonate pamidronate is a potent inhibitor of Ewing's sarcoma cell growth in vitro

W. Winkelmann, F. van Valen, J. Boos, 2001, Anti-cancer drugs.

Cooperative treatment effectiveness of ATR and HSP90 inhibition in Ewing’s sarcoma cells

Zhao-Qi Wang, René Thierbach, Yibo Geng, 2020, Cell & bioscience.

Histone deacetylase inhibitors and aspirin interact synergistically to induce cell death in ovarian cancer cells.

U. Völker, H. Kroemer, J. Beck, 2008, Oncology reports.

Comparative evaluation of the treatment efficacy of suberoylanilide hydroxamic acid (SAHA) and paclitaxel in ovarian cancer cell lines and primary ovarian cancer cells from patients

R. Ohlinger, G. Lorenz, J. Beck, 2006, BMC Cancer.

Down-regulation of protein kinase Ceta potentiates the cytotoxic effects of exogenous tumor necrosis factor-related apoptosis-inducing ligand in PC-3 prostate cancer cells.

V. Gekeler, J. Beck, H. Hofmann, 2004, Molecular cancer therapeutics.

Down-regulation of protein kinase Ceta by antisense oligonucleotides sensitises A549 lung cancer cells to vincristine and paclitaxel.

P. Bader, V. Gekeler, K. Ahlbrecht, 2004, Cancer letters.

Reduced expression of Rho guanine nucleotide dissociation inhibitor-&agr; modulates the cytotoxic effect of busulfan in HEK293 cells

T. Wieland, H. Kroemer, J. Beck, 2007, Anti-cancer drugs.

Global metabolic alterations in colorectal cancer cells during irinotecan-induced DNA replication stress

René Thierbach, M. Linnebacher, T. Heinzel, 2022, Cancer & metabolism.

Histone deacetylase inhibitors, but not vincristine, cooperate with radiotherapy to induce cell death in medulloblastoma.

U. Völker, J. Beck, J. Sonnemann, 2007, Anticancer research.

Elongation in a Dictyostelium in vitro translation system is affected by calmodulin antagonists

A. Haenni, R. Mutzel, J. Sonnemann, 1993, FEBS letters.

Class I histone deacetylases regulate p53/NF-κB crosstalk in cancer cells.

S. Dietrich, M. Tomicic, G. Schneider, 2017, Cellular signalling.

Protease-antiprotease imbalances differ between Cystic Fibrosis patients' upper and lower airway secretions.

J. Mainz, J. Hentschel, U. Markert, 2015, Journal of cystic fibrosis : official journal of the European Cystic Fibrosis Society.

Reverse chemomodulatory effects of the SIRT1 activators resveratrol and SRT1720 in Ewing’s sarcoma cells: resveratrol suppresses and SRT1720 enhances etoposide- and vincristine-induced anticancer activity

O. Krämer, J. Beck, M. Kahl, 2015, Journal of Cancer Research and Clinical Oncology.

Histone deacetylase inhibitors induce cell death in supratentorial primitive neuroectodermal tumor cells.

J. Beck, J. Sonnemann, K. S. Kumar, 2006, Oncology reports.

Cytosolic [Ca2+] Transients in Dictyostelium discoideum Depend on the Filling State of Internal Stores and on an Active Sarco/Endoplasmic Reticulum Calcium ATPase (SERCA) Ca2+ Pump*

Daniel F Lusche, C. Schlatterer, J. Sonnemann, 2004, Journal of Biological Chemistry.

Nuclear factor-kappaB mediates up-regulation of cathepsin B by doxorubicin in tumor cells.

H. Kroemer, B. Sperker, J. Beck, 2004, Molecular pharmacology.

Peroxiredoxin III protects pancreatic ß cells from apoptosis.

R. Walther, N. Aumann, P. Newsholme, 2010, The Journal of endocrinology.

Apoptotic responsiveness of the Ewing's sarcoma family of tumours to tumour necrosis factor–related apoptosis‐inducing ligand (TRAIL)

W. Winkelmann, S. Fulda, F. van Valen, 2000, International journal of cancer.

LSD1 (KDM1A)‐independent effects of the LSD1 inhibitor SP2509 in cancer cells

F. Ebert, Miriam Zimmermann, J. Beck, 2018, British journal of haematology.

Synergistic activity of the histone deacetylase inhibitor suberoylanilide hydroxamic acid and the bisphosphonate zoledronic acid against prostate cancer cells in vitro

J. Beck, J. Sonnemann, Beata Bumbul, 2007, Molecular Cancer Therapeutics.

Histone deacetylase inhibitors interact synergistically with tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) to induce apoptosis in carcinoma cell lines

P. Bader, J. Beck, J. Sonnemann, 2005, Investigational New Drugs.

Tumor-Intrinsic PD-L1 Exerts an Oncogenic Function through the Activation of the Wnt/β-Catenin Pathway in Human Non-Small Cell Lung Cancer

N. Gassler, O. Huber, Yunxia Ma, 2022, International journal of molecular sciences.

Tight association of autophagy and cell cycle in leukemia cells

J. Beck, L. Marx-Blümel, Martin Förster, 2021, Cellular & molecular biology letters.

Changes of Proteases, Antiproteases, and Pathogens in Cystic Fibrosis Patients' Upper and Lower Airways after IV-Antibiotic Therapy

J. Mainz, J. Hentschel, U. Hipler, 2015, Mediators of inflammation.

Histone deacetylase inhibitors sensitize tumour cells for cytotoxic effects of natural killer cells.

L. Moretta, B. Bröker, J. Beck, 2008, Cancer letters.

Inhibition of novel protein kinase Cɛ augments TRAIL-induced cell death in A549 lung cancer cells

J. Beck, J. Sonnemann, Matthias Felber, 2008, Pathology & Oncology Research.

Establishment of a one-sided ex vivo human placenta perfusion model to assess adhesion and invasion behavior of T cell leukemia cell lines

U. Markert, J. Sonnemann, S. Schamberger, 2013, Leukemia & lymphoma.

The bisphosphonate pamidronate is a potent inhibitor of human osteosarcoma cell growth in vitro

W. Winkelmann, F. van Valen, J. Boos, 2001, Anti-cancer drugs.

Histone deacetylase inhibitor-mediated sensitization to TRAIL-induced apoptosis in childhood malignancies is not associated with upregulation of TRAIL receptor expression, but with potentiated caspase-8 activation

J. Beck, S. Wittig, J. Sonnemann, 2012, Cancer biology & therapy.

The sirtuin 1/2 inhibitor tenovin-1 induces a nonlinear apoptosis-inducing factor-dependent cell death in a p53 null Ewing’s sarcoma cell line

Zhao-Qi Wang, René Thierbach, H. Slevogt, 2018, Investigational New Drugs.

Histone deacetylase inhibitors induce cell death and enhance the apoptosis-inducing activity of TRAIL in Ewing’s sarcoma cells

U. Völker, M. Hartwig, B. Bröker, 2007, Journal of Cancer Research and Clinical Oncology.

Histone deacetylase inhibitors induce cell death and enhance the susceptibility to ionizing radiation, etoposide, and TRAIL in medulloblastoma cells.

P. Bader, S. Heesch, J. Beck, 2006, International journal of oncology.

Histone deacetylase inhibitors require caspase activity to induce apoptosis in lung and prostate carcinoma cells.

M. Hartwig, J. Beck, A. Plath, 2006, Cancer letters.

KRAS-mutated non-small cell lung cancer cells are responsive to either co-treatment with erlotinib or gefitinib and histone deacetylase inhibitors or single treatment with lapatinib.

J. Beck, J. Sonnemann, Ina Kurtze, 2011, Oncology reports.

The expression of histone deacetylase 4 is associated with prednisone poor-response in childhood acute lymphoblastic leukemia.

M. Walther, B. Gruhn, J. Beck, 2013, Leukemia research.

Histone deacetylase inhibitors enhance the anticancer activity of nutlin-3 and induce p53 hyperacetylation and downregulation of MDM2 and MDM4 gene expression

J. Beck, J. Sonnemann, C. Palani, 2012, Investigational New Drugs.

Nanostructured delivery system for Suberoylanilide hydroxamic acid against lung cancer cells.

R. Sankar, Venkateshwaran Krishnaswami, N. Subramanian, 2015, Materials science & engineering. C, Materials for biological applications.

Histone deacetylase inhibitors prevent activation of tumour-reactive NK cells and T cells but do not interfere with their cytolytic effector functions.

B. Bröker, J. Beck, M. Schmudde, 2010, Cancer letters.

Dissection of the cAMP induced cytosolic calcium response in Dictyostelium discoideum: the role of cAMP receptor subtypes and G protein subunits

A. Aichem, C. Schlatterer, J. Sonnemann, 1998, FEBS letters.

cAMP-induced changes in the cytosolic free Ca2+ concentration in Dictyostelium discoideum are light sensitive.

C. Schlatterer, G. Knoll, J. Sonnemann, 1997, Cell calcium.

Fatty acids induce release of Ca2+ from acidosomal stores and activate capacitative Ca2+ entry in Dictyostelium discoideum.

D. Malchow, R. Schaloske, C. Schlatterer, 1998, The Biochemical journal.

Differential responsiveness of human hepatoma cells versus normal hepatocytes to TRAIL in combination with either histone deacetylase inhibitors or conventional cytostatics

J. Beck, J. Sonnemann, J. Dzieran, 2008, Cancer science.

p53-dependent and p53-independent anticancer effects of different histone deacetylase inhibitors

O. Krämer, J. Beck, S. Wittig, 2013, British Journal of Cancer.

Mechanistic insights into p53‐regulated cytotoxicity of combined entinostat and irinotecan against colorectal cancer cells

Zhao-Qi Wang, René Thierbach, M. Linnebacher, 2021, Molecular oncology.

Anticancer effects of the p53 activator nutlin-3 in Ewing's sarcoma cells.

J. Beck, S. Wittig, A. Voigt, 2011, European journal of cancer.

Increased activity of histone deacetylases in childhood acute lymphoblastic leukaemia and acute myeloid leukaemia: support for histone deacetylase inhibitors as antileukaemic agents

B. Gruhn, J. Beck, S. Wittig, 2012, British journal of haematology.

Influence of high glucose in the expression of miRNAs and IGF1R signaling pathway in human myometrial explants

E. Schleußner, U. Markert, T. M. Zorn, 2021, Archives of Gynecology and Obstetrics.

RETRA exerts anticancer activity in Ewing's sarcoma cells independent of their TP53 status.

J. Hentschel, O. Krämer, J. Beck, 2015, European journal of cancer.

Epithelial Membrane Protein 2 Suppresses Non-Small Cell Lung Cancer Cell Growth by Inhibition of MAPK Pathway

N. Gassler, M. Westermann, Yuan-Hua Chen, 2021, International journal of molecular sciences.

Influences of nasal lavage collection-, processing- and storage methods on inflammatory markers--evaluation of a method for non-invasive sampling of epithelial lining fluid in cystic fibrosis and other respiratory diseases.

J. Mainz, J. Hentschel, U. Markert, 2014, JIM - Journal of Immunological Methods.

Assessment of Mitochondrial Dysfunctions After Sirtuin Inhibition.

Zhao-Qi Wang, L. Marx-Blümel, J. Sonnemann, 2023, Methods in molecular biology.

Synergistic anticancer activity of combined ATR and ribonucleotide reductase inhibition in Ewing's sarcoma cells

J. Beck, H. Proquitté, Julián Henao-Restrepo, 2023, Journal of Cancer Research and Clinical Oncology.

Molecular characterization of hematopoietic stem cells after in vitro amplification on biomimetic 3D PDMS cell culture scaffolds

Zhao-Qi Wang, M. Groth, Emilio Cirri, 2021, Scientific Reports.

Cytosolic nucleoside diphosphate kinase associated with the translation apparatus may provide GTP for protein synthesis.

R. Mutzel, J. Sonnemann, 1995, Biochemical and biophysical research communications.

Comparative evaluation of the treatment efficacy of suberoylanilide hydroxamic acid (SAHA) and paclitaxel in ovarian cancer cell lines and primary ovarian cancer cells from patients

R. Ohlinger, G. Lorenz, J. Beck, 2006, BMC Cancer.

Apoptotic responsiveness of PC-3 prostate cancer cells to tumor necrosis factor-related apoptosis-inducing ligand: evidence for differential effects of Bcl-xL and Bcl-2 down-regulation.

V. Gekeler, J. Beck, H. Hofmann, 2004, International journal of oncology.

Assessment of HDACi-Induced Cytotoxicity.

L. Marx-Blümel, J. Sonnemann, C. Marx, 2017, Methods in molecular biology.

Anticancer effects of the PLK4 inhibitors CFI-400945 and centrinone in Ewing’s sarcoma cells

J. Beck, J. Sonnemann, S. Becker, 2020, Journal of Cancer Research and Clinical Oncology.